Ewa Świderska

469 total citations
17 papers, 264 citations indexed

About

Ewa Świderska is a scholar working on Epidemiology, Molecular Biology and Cancer Research. According to data from OpenAlex, Ewa Świderska has authored 17 papers receiving a total of 264 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Epidemiology, 7 papers in Molecular Biology and 5 papers in Cancer Research. Recurrent topics in Ewa Świderska's work include Adipokines, Inflammation, and Metabolic Diseases (7 papers), MicroRNA in disease regulation (5 papers) and Histone Deacetylase Inhibitors Research (3 papers). Ewa Świderska is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (7 papers), MicroRNA in disease regulation (5 papers) and Histone Deacetylase Inhibitors Research (3 papers). Ewa Świderska collaborates with scholars based in Poland, Australia and Taiwan. Ewa Świderska's co-authors include Agnieszka Śliwińska, Justyna Strycharz, Janusz Szemraj, Józef Drzewoski, Leszek Szmigiero, Piotr Czarny, Jacek Kasznicki, Aneta Balcerczyk, Monika Talarowska and Lech Pomorski and has published in prestigious journals such as International Journal of Molecular Sciences, Nutrients and Current Medicinal Chemistry.

In The Last Decade

Ewa Świderska

15 papers receiving 262 citations

Peers

Ewa Świderska

PHYSI

EPIDE

EDM

Fengwei Nan China

Justyna Strycharz Poland

Daniel Siuda Germany

Francisca M. Santandreu Spain

Pingping Yu China

Hang Jin China

Hye-Na Cha South Korea

Katrin Pfuhlmann Germany

Lan Luo China

Fengwei Nan China

Ewa Świderska

156 ×1.3

92 ×1.5

PHYSI

53 ×1.1

EPIDE

32 ×0.7

33 ×0.8

EDM

Citations per year, relative to Ewa Świderska Ewa Świderska (= 1×) peers Fengwei Nan

Countries citing papers authored by Ewa Świderska

Since Specialization

Citations

This map shows the geographic impact of Ewa Świderska's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ewa Świderska with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ewa Świderska more than expected).

Fields of papers citing papers by Ewa Świderska

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ewa Świderska. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ewa Świderska. The network helps show where Ewa Świderska may publish in the future.

Co-authorship network of co-authors of Ewa Świderska

This figure shows the co-authorship network connecting the top 25 collaborators of Ewa Świderska. A scholar is included among the top collaborators of Ewa Świderska based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ewa Świderska. Ewa Świderska is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Strycharz, Justyna, Katarzyna Oszajca, Piotr Czarny, et al.. (2024). Errate: Dysregulation of Inflammation, Oxidative Stress, and Glucose Metabolism-Related Genes and miRNAs in Visceral Adipose Tissue of Women with Type 2 Diabetes Mellitus. Medical Science Monitor. 30. e944015–e944015. 1 indexed citations

Szymczak-Pajor, Izabela, Józef Drzewoski, Ewa Świderska, et al.. (2023). Metformin Induces Apoptosis in Human Pancreatic Cancer (PC) Cells Accompanied by Changes in the Levels of Histone Acetyltransferases (Particularly, p300/CBP-Associated Factor (PCAF) Protein Levels). Pharmaceuticals. 16(1). 115–115. 15 indexed citations

Pieczonka, Adam M., Ewa Świderska, Michał Rachwalski, et al.. (2023). Autophagy Inhibition with Chloroquine Increased Pro-Apoptotic Potential of New Aziridine-Hydrazide Hydrazone Derivatives against Glioblastoma Cells. Cells. 12(14). 1906–1906. 7 indexed citations

Strycharz, Justyna, Katarzyna Oszajca, Piotr Czarny, et al.. (2023). Expression of NFKB1, TIGAR, and a Set of miRNAs Is Upregulated in Visceral Adipose Tissue of Women with T2DM but Not with IFG. Medical Science Monitor. 29. e939299–e939299. 5 indexed citations

Strycharz, Justyna, Ewa Świderska, Aneta Balcerczyk, et al.. (2021). Chronic and Transient Hyperglycemia Induces Changes in the Expression Patterns of IL6 and ADIPOQ Genes and Their Associated Epigenetic Modifications in Differentiating Human Visceral Adipocytes. International Journal of Molecular Sciences. 22(13). 6964–6964. 12 indexed citations

Strycharz, Justyna, Andrzej Zieleniak, Ewa Świderska, et al.. (2021). Visceral Adipose Tissue of Prediabetic and Diabetic Females Shares a Set of Similarly Upregulated microRNAs Functionally Annotated to Inflammation, Oxidative Stress and Insulin Signaling. Antioxidants. 10(1). 101–101. 26 indexed citations

Świderska, Ewa, Justyna Strycharz, Piotr Czarny, et al.. (2021). Chronic and Intermittent Hyperglycemia Modulates Expression of Key Molecules of PI3K/AKT Pathway in Differentiating Human Visceral Adipocytes. International Journal of Molecular Sciences. 22(14). 7712–7712. 11 indexed citations

Czarny, Piotr, Paulina Wigner, Justyna Strycharz, et al.. (2019). Mitochondrial DNA copy number, damage, repair and degradation in depressive disorder. The World Journal of Biological Psychiatry. 21(2). 91–101. 28 indexed citations

Wojtala, Martyna, Dorota Rybaczek, Agnieszka Śliwińska, et al.. (2019). Silencing Lysine-Specific Histone Demethylase 1 (LSD1) Causes Increased HP1-Positive Chromatin, Stimulation of DNA Repair Processes, and Dysregulation of Proliferation by Chk1 Phosphorylation in Human Endothelial Cells. Cells. 8(10). 1212–1212. 22 indexed citations

10.

Świderska, Ewa, Justyna Strycharz, Marzena Szwed, et al.. (2019). Hyperglycemia Changes Expression of Key Adipogenesis Markers (C/EBPα and PPARᵞ)and Morphology of Differentiating Human Visceral Adipocytes. Nutrients. 11(8). 1835–1835. 12 indexed citations

11.

Strycharz, Justyna, Ewa Świderska, Józef Drzewoski, et al.. (2019). Molecular Insight into the Interaction between Epigenetics and Leptin in Metabolic Disorders. Nutrients. 11(8). 1872–1872. 33 indexed citations

12.

Czarny, Piotr, Paulina Wigner, Justyna Strycharz, et al.. (2018). Single-nucleotide polymorphisms of uracil-processing genes affect the occurrence and the onset of recurrent depressive disorder. PeerJ. 6. e5116–e5116. 9 indexed citations

13.

Strycharz, Justyna, Ewa Świderska, Józef Drzewoski, et al.. (2018). SIRT1 as a Therapeutic Target in Diabetic Complications. Current Medicinal Chemistry. 25(9). 1002–1035. 71 indexed citations

14.

Strycharz, Justyna, Ewa Świderska, Piotr Czarny, et al.. (2018). Hyperglycemia Affects miRNAs Expression Pattern during Adipogenesis of Human Visceral Adipocytes—Is Memorization Involved?. Nutrients. 10(11). 1774–1774. 8 indexed citations

15.

Kirchner, Hanna, et al.. (2008). Pisma rozproszone, listy : 1913-1939. 3 indexed citations

16.

Zajac, Lauren, et al.. (1991). [Clinical manifestations of extrinsic and intrinsic sinus node dysfunction (functional and organic factors)].. PubMed. 35(7). 3–8.

17.

Trusz‐Gluza, Maria, et al.. (1989). [Effect of selective and non-selective adrenergic beta receptor blockaders on selected electrophysiologic properties of the human heart].. PubMed. 32(6). 342–6. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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