Éva Wisniewski

575 total citations
13 papers, 421 citations indexed

About

Éva Wisniewski is a scholar working on Molecular Biology, Cell Biology and Physiology. According to data from OpenAlex, Éva Wisniewski has authored 13 papers receiving a total of 421 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Cell Biology and 5 papers in Physiology. Recurrent topics in Éva Wisniewski's work include Cellular transport and secretion (4 papers), Erythrocyte Function and Pathophysiology (3 papers) and Pancreatic function and diabetes (3 papers). Éva Wisniewski is often cited by papers focused on Cellular transport and secretion (4 papers), Erythrocyte Function and Pathophysiology (3 papers) and Pancreatic function and diabetes (3 papers). Éva Wisniewski collaborates with scholars based in United States, Hungary and United Kingdom. Éva Wisniewski's co-authors include Tamás Balla, Yeun Ju Kim, María Luisa Guzmán-Hernández, András Spät, Gergő Szanda, Niko Popitsch, Malcolm F. Howard, Evžen Bouřa, Marek Korzeniowski and Dominika Chalupská and has published in prestigious journals such as Blood, The Journal of Immunology and Scientific Reports.

In The Last Decade

Éva Wisniewski

13 papers receiving 420 citations

Peers

Éva Wisniewski
Ganesh Kadamur United States
Daniel Baird United States
Aleck W.E. Jones United Kingdom
Bo-Jhih Guan United States
Shanna L. Bowman United States
Éva Wisniewski
Citations per year, relative to Éva Wisniewski Éva Wisniewski (= 1×) peers María Luisa Guzmán-Hernández

Countries citing papers authored by Éva Wisniewski

Since Specialization
Citations

This map shows the geographic impact of Éva Wisniewski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Éva Wisniewski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Éva Wisniewski more than expected).

Fields of papers citing papers by Éva Wisniewski

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Éva Wisniewski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Éva Wisniewski. The network helps show where Éva Wisniewski may publish in the future.

Co-authorship network of co-authors of Éva Wisniewski

This figure shows the co-authorship network connecting the top 25 collaborators of Éva Wisniewski. A scholar is included among the top collaborators of Éva Wisniewski based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Éva Wisniewski. Éva Wisniewski is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Pettkó‐Szandtner, Aladár, et al.. (2024). Neutrophil-specific interactome of ARHGAP25 reveals novel partners and regulatory insights. Scientific Reports. 14(1). 20106–20106. 1 indexed citations
2.
Szanda, Gergő, Tony Jourdan, Éva Wisniewski, et al.. (2023). Cannabinoid receptor type 1 (CB1R) inhibits hypothalamic leptin signaling via β-arrestin1 in complex with TC-PTP and STAT3. iScience. 26(7). 107207–107207. 3 indexed citations
3.
Wisniewski, Éva, Gergő Szanda, Aladár Pettkó‐Szandtner, et al.. (2022). A novel BRET ‐Based GAP assay reveals phosphorylation‐dependent regulation of the RAC ‐specific GTPase activating protein ARHGAP25. The FASEB Journal. 36(11). e22584–e22584. 1 indexed citations
4.
Wisniewski, Éva, et al.. (2021). Optimization of the Heterologous Expression of the Cannabinoid Type-1 (CB1) Receptor. Frontiers in Endocrinology. 12. 740913–740913. 3 indexed citations
5.
Szanda, Gergő, et al.. (2018). Mitochondrial cAMP exerts positive feedback on mitochondrial Ca2+ uptake via the recruitment of Epac1. Journal of Cell Science. 131(10). 18 indexed citations
6.
Korzeniowski, Marek, Éva Wisniewski, Barbara Baird, David A. Holowka, & Tamás Balla. (2017). Molecular anatomy of the early events in STIM1 activation – oligomerization or conformational change?. Journal of Cell Science. 130(17). 2821–2832. 21 indexed citations
7.
Wang, Leo D., Scott B. Ficarro, John N. Hutchinson, et al.. (2016). Phosphoproteomic profiling of mouse primary HSPCs reveals new regulators of HSPC mobilization. Blood. 128(11). 1465–1474. 13 indexed citations
8.
Kim, Yeun Ju, et al.. (2016). Phosphatidylinositol and phosphatidic acid transport between the ER and plasma membrane during PLC activation requires the Nir2 protein. Biochemical Society Transactions. 44(1). 197–201. 31 indexed citations
9.
Csépányi‐Kömi, Roland, Éva Wisniewski, Tamás Németh, et al.. (2016). Rac GTPase Activating Protein ARHGAP25 Regulates Leukocyte Transendothelial Migration in Mice. The Journal of Immunology. 197(7). 2807–2815. 18 indexed citations
10.
Kim, Yeun Ju, María Luisa Guzmán-Hernández, Éva Wisniewski, & Tamás Balla. (2015). Phosphatidylinositol-Phosphatidic Acid Exchange by Nir2 at ER-PM Contact Sites Maintains Phosphoinositide Signaling Competence. Developmental Cell. 33(5). 549–561. 182 indexed citations
11.
Pagnamenta, Alistair T., Malcolm F. Howard, Éva Wisniewski, et al.. (2015). Germline recessive mutations in PI4KA are associated with perisylvian polymicrogyria, cerebellar hypoplasia and arthrogryposis. Human Molecular Genetics. 24(13). 3732–3741. 51 indexed citations
12.
Chalupská, Dominika, Bartosz Różycki, Marko Jović, et al.. (2014). The crystal structure of the phosphatidylinositol 4‐kinase II α. EMBO Reports. 15(10). 1085–1092. 49 indexed citations
13.
Zhong, Nan, Éva Wisniewski, Jaana M. Hartikainen, et al.. (1998). Two common mutations in the CLN2 gene underlie late infantile neuronal ceroid lipoluscinosis. Clinical Genetics. 54(3). 234–238. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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