Eva Tomás

3.9k total citations · 2 hit papers
27 papers, 3.2k citations indexed

About

Eva Tomás is a scholar working on Molecular Biology, Surgery and Physiology. According to data from OpenAlex, Eva Tomás has authored 27 papers receiving a total of 3.2k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 10 papers in Surgery and 10 papers in Physiology. Recurrent topics in Eva Tomás's work include Metabolism, Diabetes, and Cancer (20 papers), Pancreatic function and diabetes (10 papers) and Adipose Tissue and Metabolism (10 papers). Eva Tomás is often cited by papers focused on Metabolism, Diabetes, and Cancer (20 papers), Pancreatic function and diabetes (10 papers) and Adipose Tissue and Metabolism (10 papers). Eva Tomás collaborates with scholars based in United States, Spain and Germany. Eva Tomás's co-authors include Neil B. Ruderman, Asish K. Saha, Tsu‐Shuen Tsao, Harvey F. Lodish, Heather E. Murrey, Joel F. Habener, Samar I. Itani, Cheng Zhang, V Stanojević and Christopher Hug and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Diabetes.

In The Last Decade

Eva Tomás

27 papers receiving 3.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Enhanced muscle fat oxidation and glucose transport by ACRP30 globular domain: Acetyl–CoA carboxylase inhibition and AMP-activated protein kinase activation

2002 788 citations Eva Tomás, Tsu‐Shuen Tsao et al. Proceedings of the National Academy of Sciences profile →
Mice Lacking Adiponectin Show Decreased Hepatic Insulin Sensitivity and Reduced Responsiveness to Peroxisome Proliferator-activated Receptor γ Agonists

2005 522 citations Andrea R. Nawrocki, Michael W. Rajala et al. Journal of Biological Chemistry profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Eva Tomás United States	20	1.4k	1.4k	1.4k	622	621	27	3.2k
Haihong Zong United States	22	1.1k 0.7×	2.5k 1.7×	2.1k 1.4×	663 1.1×	464 0.7×	38	4.4k
Hye Lim Noh United States	26	839 0.6×	1.2k 0.8×	1.1k 0.8×	422 0.7×	445 0.7×	40	2.8k
Qichen Fang China	24	1.2k 0.8×	1.4k 1.0×	818 0.6×	362 0.6×	357 0.6×	67	2.8k
Peter A. Meléndez United States	6	1.2k 0.9×	1.5k 1.1×	1.2k 0.8×	287 0.5×	467 0.8×	7	3.2k
Yi Zhu United States	32	815 0.6×	1.7k 1.2×	1.3k 0.9×	496 0.8×	271 0.4×	60	3.2k
Yongsheng Chang China	31	772 0.5×	1.9k 1.3×	1.0k 0.7×	395 0.6×	288 0.5×	90	3.6k
Yongzhong Wei United States	30	1.1k 0.8×	1.4k 1.0×	1.4k 1.0×	479 0.8×	1.0k 1.6×	57	4.1k
Yan-Ting Zhou United States	18	1.3k 0.9×	1.9k 1.3×	2.2k 1.6×	1.3k 2.2×	825 1.3×	18	5.1k
Kathleen R. Markan United States	16	915 0.6×	1.3k 0.9×	1.6k 1.1×	280 0.5×	165 0.3×	19	2.9k
Caroline Tao United States	14	1.4k 1.0×	680 0.5×	1.6k 1.1×	301 0.5×	153 0.2×	16	2.6k

Countries citing papers authored by Eva Tomás

Since Specialization

Citations

This map shows the geographic impact of Eva Tomás's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Tomás with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Tomás more than expected).

Fields of papers citing papers by Eva Tomás

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Tomás. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Tomás. The network helps show where Eva Tomás may publish in the future.

Co-authorship network of co-authors of Eva Tomás

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Tomás. A scholar is included among the top collaborators of Eva Tomás based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Tomás. Eva Tomás is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bernardo, Barbara, Min Lü, Gautam Bandyopadhyay, et al.. (2015). FGF21 does not require interscapular brown adipose tissue and improves liver metabolic profile in animal models of obesity and insulin-resistance. Scientific Reports. 5(1). 11382–11382. 40 indexed citations

Elahi, Dariush, Franca S. Angeli, Olga D. Carlson, et al.. (2014). GLP-1(32–36)amide, a novel pentapeptide cleavage product of GLP-1, modulates whole body glucose metabolism in dogs. Peptides. 59. 20–24. 12 indexed citations

Tomás, Eva, V Stanojević, & Joel F. Habener. (2011). GLP-1-derived nonapeptide GLP-1(28–36)amide targets to mitochondria and suppresses glucose production and oxidative stress in isolated mouse hepatocytes. Regulatory Peptides. 167(2-3). 177–184. 87 indexed citations

Tomás, Eva, Jenna Wood, V Stanojević, & Joel F. Habener. (2011). GLP-1-derived nonapeptide GLP-1(28–36)amide inhibits weight gain and attenuates diabetes and hepatic steatosis in diet-induced obese mice. Regulatory Peptides. 169(1-3). 43–48. 40 indexed citations

Tomás, Eva, V Stanojević, & J F Habener. (2010). GLP-1 (9–36) Amide Metabolite Suppression of Glucose Production in Isolated Mouse Hepatocytes. Hormone and Metabolic Research. 42(9). 657–662. 44 indexed citations

Tomás, Eva, Jenna Wood, V Stanojević, & Joel F. Habener. (2010). Glucagon‐like peptide‐1(9‐36)amide metabolite inhibits weight gain and attenuates diabetes and hepatic steatosis in diet‐induced obese mice. Diabetes Obesity and Metabolism. 13(1). 26–33. 39 indexed citations

Tomás, Eva & Joel F. Habener. (2009). Insulin-like actions of glucagon-like peptide-1: a dual receptor hypothesis. Trends in Endocrinology and Metabolism. 21(2). 59–67. 107 indexed citations

LeBrasseur, Nathan K., Meghan Kelly, Tsu‐Shuen Tsao, et al.. (2006). Thiazolidinediones can rapidly activate AMP-activated protein kinase in mammalian tissues. American Journal of Physiology-Endocrinology and Metabolism. 291(1). E175–E181. 245 indexed citations

Kelly, Meghan, Neil B. Ruderman, & Eva Tomás. (2006). AMP-activated protein kinase and its regulation by adiponectin and interleukin-6. 50(sup2). 85–91. 8 indexed citations

10.

Nawrocki, Andrea R., Michael W. Rajala, Eva Tomás, et al.. (2005). Mice Lacking Adiponectin Show Decreased Hepatic Insulin Sensitivity and Reduced Responsiveness to Peroxisome Proliferator-activated Receptor γ Agonists. Journal of Biological Chemistry. 281(5). 2654–2660. 522 indexed citations breakdown →

11.

Tomás, Eva, Meghan Kelly, Xiaoqin Xiang, et al.. (2004). Metabolic and hormonal interactions between muscle and adipose tissue. Proceedings of The Nutrition Society. 63(2). 381–385. 42 indexed citations

12.

Tsao, Tsu‐Shuen, Eva Tomás, Heather E. Murrey, et al.. (2003). Role of Disulfide Bonds in Acrp30/Adiponectin Structure and Signaling Specificity. Journal of Biological Chemistry. 278(50). 50810–50817. 399 indexed citations

13.

Iglesias, Miguel A., Ji‐Ming Ye, Georgia Frangioudakis, et al.. (2002). AICAR Administration Causes an Apparent Enhancement of Muscle and Liver Insulin Action in Insulin-Resistant High-Fat-Fed Rats. Diabetes. 51(10). 2886–2894. 245 indexed citations

14.

Tomás, Eva, Yenshou Lin, Zeina Dagher, et al.. (2002). Hyperglycemia and Insulin Resistance: Possible Mechanisms. Annals of the New York Academy of Sciences. 967(1). 43–51. 127 indexed citations

15.

Tomás, Eva, Tsu‐Shuen Tsao, Asish K. Saha, et al.. (2002). Enhanced muscle fat oxidation and glucose transport by ACRP30 globular domain: Acetyl–CoA carboxylase inhibition and AMP-activated protein kinase activation. Proceedings of the National Academy of Sciences. 99(25). 16309–16313. 788 indexed citations breakdown →

16.

Tomás, Eva, Lídia Sevilla, Manuel Palacı́n, & António Zorzano. (2001). The Insulin-Sensitive GLUT4 Storage Compartment Is a Postendocytic and Heterogeneous Population Recruited by Acute Exercise. Biochemical and Biophysical Research Communications. 284(2). 490–495. 16 indexed citations

17.

Becker, Christoph, Lídia Sevilla, Eva Tomás, et al.. (2001). The Endosomal Compartment Is an Insulin-Sensitive Recruitment Site for GLUT4 and GLUT1 Glucose Transporters in Cardiac Myocytes. Endocrinology. 142(12). 5267–5276. 50 indexed citations

18.

Kessler, Alexandra, Eva Tomás, Dorian Immler, et al.. (2000). Rab11 is associated with GLUT4-containing vesicles and redistributes in response to insulin. Diabetologia. 43(12). 1518–1527. 91 indexed citations

19.

Sevilla, Lídia, Eva Tomás, Purificación Muñoz, et al.. (1997). Characterization of Two Distinct Intracellular GLUT4 Membrane Populations in Muscle Fiber. Differential Protein Composition and Sensitivity to Insulin*. Endocrinology. 138(7). 3006–3015. 38 indexed citations

20.

Muñoz, Purificación, Sílvia Mora, Lídia Sevilla, et al.. (1996). Expression and Insulin-regulated Distribution of Caveolin in Skeletal Muscle. Journal of Biological Chemistry. 271(14). 8133–8139. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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