Eva Rother

1.5k total citations · 1 hit paper
19 papers, 1.2k citations indexed

About

Eva Rother is a scholar working on Endocrine and Autonomic Systems, Surgery and Physiology. According to data from OpenAlex, Eva Rother has authored 19 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Endocrine and Autonomic Systems, 8 papers in Surgery and 6 papers in Physiology. Recurrent topics in Eva Rother's work include Regulation of Appetite and Obesity (11 papers), Pancreatic function and diabetes (6 papers) and Birth, Development, and Health (5 papers). Eva Rother is often cited by papers focused on Regulation of Appetite and Obesity (11 papers), Pancreatic function and diabetes (6 papers) and Birth, Development, and Health (5 papers). Eva Rother collaborates with scholars based in Germany, United States and United Kingdom. Eva Rother's co-authors include Jens C. Brüning, Brigitte Hampel, A. Christine Könner, Gregory S. Barsh, Ruth Janoschek, Leona Plum, F. Thomas Wunderlich, Frances M. Ashcroft, Sabine D. Jordan and Xiaosong Ma and has published in prestigious journals such as Proceedings of the National Academy of Sciences, SHILAP Revista de lepidopterología and Cell Metabolism.

In The Last Decade

Eva Rother

19 papers receiving 1.2k citations

Hit Papers

Insulin Action in AgRP-Expressing Neurons Is Required for... 2007 2026 2013 2019 2007 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eva Rother Germany 13 679 530 321 278 248 19 1.2k
Jesús P. Camiña Spain 22 737 1.1× 554 1.0× 155 0.5× 451 1.6× 401 1.6× 43 1.4k
Zhenyan He China 18 458 0.7× 374 0.7× 130 0.4× 444 1.6× 232 0.9× 29 1.3k
Fang Zou China 14 429 0.6× 338 0.6× 99 0.3× 196 0.7× 108 0.4× 25 1.0k
Beatrix Sármán Hungary 11 454 0.7× 310 0.6× 77 0.2× 92 0.3× 182 0.7× 18 762
Shigeru Okuya Japan 14 377 0.6× 163 0.3× 153 0.5× 290 1.0× 127 0.5× 22 937
Grace W.C. Cheung Canada 11 289 0.4× 358 0.7× 322 1.0× 346 1.2× 103 0.4× 16 938
Yujiro Hayashi Japan 12 950 1.4× 595 1.1× 127 0.4× 243 0.9× 682 2.8× 17 1.4k
Melissa A. Burmeister United States 18 273 0.4× 294 0.6× 314 1.0× 361 1.3× 76 0.3× 34 1.2k
K. Alexander Iwen Germany 17 172 0.3× 416 0.8× 183 0.6× 303 1.1× 92 0.4× 30 1.1k
Stephanie M. Krasnow United States 21 340 0.5× 394 0.7× 90 0.3× 317 1.1× 108 0.4× 27 1.1k

Countries citing papers authored by Eva Rother

Since Specialization
Citations

This map shows the geographic impact of Eva Rother's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Rother with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Rother more than expected).

Fields of papers citing papers by Eva Rother

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Rother. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Rother. The network helps show where Eva Rother may publish in the future.

Co-authorship network of co-authors of Eva Rother

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Rother. A scholar is included among the top collaborators of Eva Rother based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Rother. Eva Rother is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Alcázar, Miguel A. Alejandre, Katharina Dinger, Eva Rother, et al.. (2014). Prevention of Early Postnatal Hyperalimentation Protects against Activation of Transforming Growth Factor-β/Bone Morphogenetic Protein and Interleukin-6 Signaling in Rat Lungs after Intrauterine Growth Restriction. Journal of Nutrition. 144(12). 1943–1951. 15 indexed citations
2.
Appel, Sarah, Eva Rother, Ruth Janoschek, et al.. (2014). Hypoxia-Mediated Soluble Fms-Like Tyrosine Kinase 1 Increase Is Not Attenuated in Interleukin 6-Deficient Mice. Reproductive Sciences. 22(6). 735–742. 4 indexed citations
3.
Appel, Sarah, Miguel A. Alejandre Alcázar, Eva Rother, et al.. (2014). Leptin does not Induce an Inflammatory Response in the Murine Placenta. Hormone and Metabolic Research. 46(6). 384–389. 5 indexed citations
4.
Rother, Eva, Bengt‐Frederik Belgardt, Eva Tsaousidou, et al.. (2012). Acute selective ablation of rat insulin promoter-expressing (RIP HER ) neurons defines their orexigenic nature. Proceedings of the National Academy of Sciences. 109(44). 18132–18137. 26 indexed citations
5.
Alcázar, Miguel A. Alejandre, Sarah Appel, Eva Rother, et al.. (2012). Developmental regulation of inflammatory cytokine-mediated Stat3 signaling: the missing link between intrauterine growth restriction and pulmonary dysfunction?. Journal of Molecular Medicine. 90(8). 945–957. 21 indexed citations
6.
Alcázar, Miguel A. Alejandre, Eva Rother, Kerstin Amann, et al.. (2012). Early Postnatal Hyperalimentation Impairs Renal Function via SOCS-3 Mediated Renal Postreceptor Leptin Resistance. Endocrinology. 153(3). 1397–1410. 19 indexed citations
7.
Rother, Eva, Miguel A. Alejandre Alcázar, André Oberthuer, et al.. (2011). Hypothalamic JNK1 and IKKβ Activation and Impaired Early Postnatal Glucose Metabolism after Maternal Perinatal High-Fat Feeding. Endocrinology. 153(2). 770–781. 59 indexed citations
8.
Remedi, Marı́a S., Harley T. Kurata, F. Thomas Wunderlich, et al.. (2009). Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of KATP-Induced Neonatal Diabetes Mellitus. Cell Metabolism. 9(2). 140–151. 80 indexed citations
9.
Rother, Eva, Sue Jordan, & Jens C. Brüning. (2009). Die Bedeutung des Gehirns für die Regulation von Energie- und Glukosestoffwechsel. DMW - Deutsche Medizinische Wochenschrift. 134(20). 1057–1059. 1 indexed citations
10.
Mesaros, Andrea, Sergei B. Koralov, Eva Rother, et al.. (2008). Activation of Stat3 Signaling in AgRP Neurons Promotes Locomotor Activity. Cell Metabolism. 7(3). 236–248. 104 indexed citations
11.
Rother, Eva, A. Christine Könner, & Jens C. Brüning. (2008). Neurocircuits integrating hormone and nutrient signaling in control of glucose metabolism. American Journal of Physiology-Endocrinology and Metabolism. 294(5). E810–E816. 25 indexed citations
12.
Belgardt, Bengt‐Frederik, Andreas Husch, Eva Rother, et al.. (2008). PDK1 Deficiency in POMC-Expressing Cells Reveals FOXO1-Dependent and -Independent Pathways in Control of Energy Homeostasis and Stress Response. Cell Metabolism. 7(4). 291–301. 128 indexed citations
13.
Lankiewicz, Silke, Eva Rother, Silke Zimmermann, et al.. (2008). Tumour-associated transcripts and EGFR deletion variants in colorectal cancer in primary tumour, metastases and circulating tumour cells.. SHILAP Revista de lepidopterología. 30(6). 463–71. 28 indexed citations
14.
Lankiewicz, Silke, Eva Rother, Silke Zimmermann, et al.. (2008). Tumour-Associated Transcripts and EGFR Deletion Variants in Colorectal Cancer in Primary Tumour, Metastases and Circulating Tumour Cells. Analytical Cellular Pathology. 30(6). 463–471. 13 indexed citations
15.
Plum, Leona, Eva Rother, Heike Münzberg, et al.. (2007). Enhanced Leptin-Stimulated Pi3k Activation in the CNS Promotes White Adipose Tissue Transdifferentiation. Cell Metabolism. 6(6). 431–445. 119 indexed citations
16.
Könner, A. Christine, Ruth Janoschek, Leona Plum, et al.. (2007). Insulin Action in AgRP-Expressing Neurons Is Required for Suppression of Hepatic Glucose Production. Cell Metabolism. 5(6). 438–449. 538 indexed citations breakdown →
17.
Plum, Lori A., Xianyong Ma, Brigitte Hampel, et al.. (2006). Enhanced PIP3 signaling in POMC neurons causes diet-sensitive obesity as the consequence of neuronal silencing via KATP channel activation. Diabetologie und Stoffwechsel. 1(S 1). 1 indexed citations
18.
Rother, Eva, et al.. (2006). Selective ablation of hypothalamic POMC-expressing neurons leads to hyperphagia and weight gain. Diabetologie und Stoffwechsel. 1(S 1). 2 indexed citations
19.
Wiek, Jutta, et al.. (1993). [Unilateral blindness in neurosyphilis].. PubMed. 105(15). 437–9. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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