Eva Polaková

674 total citations
28 papers, 491 citations indexed

About

Eva Polaková is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Eva Polaková has authored 28 papers receiving a total of 491 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Cardiology and Cardiovascular Medicine, 16 papers in Molecular Biology and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in Eva Polaková's work include Cardiomyopathy and Myosin Studies (12 papers), Muscle Physiology and Disorders (7 papers) and Ion channel regulation and function (7 papers). Eva Polaková is often cited by papers focused on Cardiomyopathy and Myosin Studies (12 papers), Muscle Physiology and Disorders (7 papers) and Ion channel regulation and function (7 papers). Eva Polaková collaborates with scholars based in Czechia, Slovakia and United States. Eva Polaková's co-authors include Natalia Shirokova, Ernst Niggli, Nina D. Ullrich, Sergii Kyrychenko, Alexandra Zahradnı́ková, Ivan Zahradnı́k, Eric A. Sobie, Jakob Ogrodnik, Carole Jung and Zdena Sulová and has published in prestigious journals such as The Journal of Physiology, International Journal of Molecular Sciences and Biophysical Journal.

In The Last Decade

Eva Polaková

26 papers receiving 487 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eva Polaková Czechia 14 365 285 96 53 44 28 491
Julia H. Steinbrecher Germany 5 353 1.0× 265 0.9× 77 0.8× 74 1.4× 24 0.5× 7 493
Domenico M. Taglieri United States 12 377 1.0× 394 1.4× 57 0.6× 49 0.9× 39 0.9× 14 617
Dimitar P. Zankov Japan 16 465 1.3× 455 1.6× 109 1.1× 18 0.3× 56 1.3× 30 657
Elyshia McNamara Australia 11 352 1.0× 266 0.9× 62 0.6× 34 0.6× 31 0.7× 21 507
Martin N. Andersen Denmark 14 476 1.3× 357 1.3× 93 1.0× 32 0.6× 28 0.6× 19 665
Stephen L. Belmonte United States 8 260 0.7× 171 0.6× 72 0.8× 35 0.7× 33 0.8× 8 361
Julia O. Reynolds United States 18 509 1.4× 519 1.8× 61 0.6× 80 1.5× 16 0.4× 24 832
Qiujing Song China 13 425 1.2× 302 1.1× 55 0.6× 40 0.8× 60 1.4× 30 615
Mari Merentie Finland 13 322 0.9× 193 0.7× 36 0.4× 52 1.0× 52 1.2× 17 547
Katherina M. Alsina United States 14 454 1.2× 440 1.5× 55 0.6× 15 0.3× 33 0.8× 23 648

Countries citing papers authored by Eva Polaková

Since Specialization
Citations

This map shows the geographic impact of Eva Polaková's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Polaková with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Polaková more than expected).

Fields of papers citing papers by Eva Polaková

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Polaková. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Polaková. The network helps show where Eva Polaková may publish in the future.

Co-authorship network of co-authors of Eva Polaková

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Polaková. A scholar is included among the top collaborators of Eva Polaková based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Polaková. Eva Polaková is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rowin, Ethan J., Raymond H. Chan, Michael T. Chin, et al.. (2024). Relationship Between Genotype Status and Clinical Outcome in Hypertrophic Cardiomyopathy. Journal of the American Heart Association. 13(10). e033565–e033565. 10 indexed citations
2.
Veselka, Josef, Max Liebregts, Robert Cooper, et al.. (2022). Outcomes of Patients With Hypertrophic Obstructive Cardiomyopathy and Pacemaker Implanted After Alcohol Septal Ablation. JACC: Cardiovascular Interventions. 15(19). 1910–1917. 12 indexed citations
3.
Hájek, Petr, et al.. (2022). Mid-term outcomes of patients with Lotus and Evolut transcatheter valves. Advances in Interventional Cardiology. 18(2). 146–153.
4.
Veselka, Josef, Max Liebregts, Robert Cooper, et al.. (2022). Prediction of Sudden Cardiac Arrest After Alcohol Septal Ablation for Hypertrophic Obstructive Cardiomyopathy: ASA-SCARRE Risk Score. The American Journal of Cardiology. 184. 120–126. 9 indexed citations
5.
Veselka, Josef, Lothar Faber, Max Liebregts, et al.. (2021). Alcohol dose in septal ablation for hypertrophic obstructive cardiomyopathy. International Journal of Cardiology. 333. 127–132. 10 indexed citations
6.
Polaková, Eva, et al.. (2021). Genetic Testing in Patients with Hypertrophic Cardiomyopathy. International Journal of Molecular Sciences. 22(19). 10401–10401. 21 indexed citations
7.
Polaková, Eva, et al.. (2020). Effectiveness of alcohol septal ablation for hypertrophic obstructive cardiomyopathy in patients with late gadolinium enhancement on cardiac magnetic resonance. International Journal of Cardiology. 319. 101–105. 2 indexed citations
8.
Polaková, Eva, et al.. (2015). Mitochondrial dysfunctions during progression of dystrophic cardiomyopathy. Cell Calcium. 58(2). 186–195. 46 indexed citations
9.
Kyrychenko, Sergii, Eva Polaková, Nina D. Ullrich, Ernst Niggli, & Natalia Shirokova. (2012). Insights into RyRs Dysfunctions via Studies of Intracellular Calcium Signals. Biophysical Journal. 102(3). 213a–213a. 1 indexed citations
10.
Niggli, Ernst, et al.. (2012). Posttranslational modifications of cardiac ryanodine receptors: Ca2+ signaling and EC-coupling. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1833(4). 866–875. 74 indexed citations
11.
Kyrychenko, Sergii, Eva Polaková, Chifei Kang, et al.. (2012). Hierarchical accumulation of RyR post-translational modifications drives disease progression in dystrophic cardiomyopathy. Cardiovascular Research. 97(4). 666–675. 40 indexed citations
12.
Polaková, Eva & Natalia Shirokova. (2011). Abnormal Sodium Handling and Mitochondrial Metabolism in Cardiac Dystrophy. Biophysical Journal. 100(3). 81a–81a. 3 indexed citations
13.
Polaková, Eva, et al.. (2009). Pathways of abnormal stress-induced Ca2+ influx into dystrophic mdx cardiomyocytes. Cell Calcium. 46(2). 114–121. 60 indexed citations
14.
Polaková, Eva, et al.. (2009). Pathways of Abnormal Stress-Induced Calcium Influx into Dystrophic mdx Cardiomyocytes. Biophysical Journal. 96(3). 274a–274a. 1 indexed citations
15.
Ullrich, Nina D., et al.. (2009). Changes of EC-coupling and RyR Calcium Sensitivity in Dystrophic mdx Mouse Cardiomyocytes. Biophysical Journal. 96(3). 10a–11a. 2 indexed citations
16.
Križanová, Oľga, Dana Jurkovičová, Eva Polaková, et al.. (2008). Type 1 and 2 IP3 Receptors Respond Differently to Catecholamines and Stress. Annals of the New York Academy of Sciences. 1148(1). 331–337. 9 indexed citations
17.
Polaková, Eva, et al.. (2008). Local calcium release activation by DHPR calcium channel openings in rat cardiac myocytes. The Journal of Physiology. 586(16). 3839–3854. 33 indexed citations
18.
Sulová, Zdena, Danica Mislovičová, Lenka Gibalová, et al.. (2008). Vincristine-Induced Overexpression of P-Glycoprotein in L1210 Cells Is Associated with Remodeling of Cell Surface Saccharides. Journal of Proteome Research. 8(2). 513–520. 22 indexed citations
19.
Zahradnı́ková, Alexandra, Eva Polaková, & Ivan Zahradnı́k. (2006). Kinetics of calcium spikes in rat cardiac myocytes. The Journal of Physiology. 578(3). 677–691. 22 indexed citations
20.
Sulová, Zdena, Ima Dovinová, Eva Polaková, et al.. (2006). L1210 cells cultivated under the selection pressure of doxorubicin or vincristine express common mechanisms of multidrug resistance based on the overexpression of P-glycoprotein. Toxicology in Vitro. 20(8). 1560–1568. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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