Eva Maria Deiner

808 citations
10 papers · 689 indexed · h-index 8
Topics
Erythrocyte Function and Pathophysiology (7 papers)Cytokine Signaling Pathways and Interactions (3 papers)Cell death mechanisms and regulation (2 papers)

In The Last Decade

Eva Maria Deiner

10 papers receiving 672 citations

Peers

Eva Maria Deiner
Comparison fields: 5 of 61
  • Molecular Biology 426
  • Physiology 221
  • Oncology 168
  • Cell Biology 121
  • Genetics 119
Replace Satomi Kuramochi with:
Satomi Kuramochi Japan
Bradley McColl Australia
Basem S. Goueli United States
M Fukumoto Japan
Sarah Ogilvy Australia
I Wadman United Kingdom
Sonja E. Gustin Australia
Karoline J. Briegel United States
François Morlé France
Jeffrey Malik United States
Eva Maria Deiner relative to Satomi Kuramochi Japan Satomi Kuramochi's profile →
Citations per field
00.5×4.5×
Satomi Kuramochi · 1×
Citations per year

Countries citing papers authored by Eva Maria Deiner

Since Specialization
Citations

This map shows the geographic impact of Eva Maria Deiner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Maria Deiner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Maria Deiner more than expected).

Fields of papers citing papers by Eva Maria Deiner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Maria Deiner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Maria Deiner. The network helps show where Eva Maria Deiner may publish in the future.

Co-authorship network of co-authors of Eva Maria Deiner

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Maria Deiner. A scholar is included among the top collaborators of Eva Maria Deiner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Maria Deiner. Eva Maria Deiner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
#WorkIndexed citations
1 44
2 116
3 71
4 93
5 90
6 1
7 31
8
Transformation of erythroid progenitors by viral and cellular tyrosine kinases.
6
9 28
10 209

About Eva Maria Deiner

Eva Maria Deiner is a scholar working on Physiology, Oncology and Immunology and Allergy, having authored 10 papers that have together received 689 indexed citations. Recurring topics across this work include Erythrocyte Function and Pathophysiology (7 papers), Cytokine Signaling Pathways and Interactions (3 papers) and Cell death mechanisms and regulation (2 papers). The work is most often cited by research in Genetics (119 citations), Hematology (110 citations) and Physiology (221 citations). Eva Maria Deiner has collaborated with scholars based in Austria, Netherlands and Germany. Frequent co-authors include Hartmut Beug, Helmut Dolznig, Ernst W. Müllner, Meinrad Busslinger, Jürgen Berger, Heinz Schwarz, Ernst Reichmann, Martin Eilers, Richard Moriggl and Bianca Habermann. Their work appears in journals such as Cell, The Journal of Experimental Medicine and Oncogene.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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