Eva Crosas‐Molist

2.0k total citations
22 papers, 1.2k citations indexed

About

Eva Crosas‐Molist is a scholar working on Molecular Biology, Oncology and Hepatology. According to data from OpenAlex, Eva Crosas‐Molist has authored 22 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 9 papers in Oncology and 5 papers in Hepatology. Recurrent topics in Eva Crosas‐Molist's work include Liver physiology and pathology (5 papers), Cancer Cells and Metastasis (5 papers) and Cancer Research and Treatments (4 papers). Eva Crosas‐Molist is often cited by papers focused on Liver physiology and pathology (5 papers), Cancer Cells and Metastasis (5 papers) and Cancer Research and Treatments (4 papers). Eva Crosas‐Molist collaborates with scholars based in Spain, United Kingdom and Austria. Eva Crosas‐Molist's co-authors include Isabel Fabregat, Victoria Sanz‐Moreno, Esther Bertrán, Patricia Sancho, Óscar Maiques, Gaia Cantelli, Judit López‐Luque, Mirella Georgouli, José L. Orgaz and Samantha L. George and has published in prestigious journals such as Nature Communications, Physiological Reviews and PLoS ONE.

In The Last Decade

Eva Crosas‐Molist

21 papers receiving 1.2k citations

Peers

Eva Crosas‐Molist
Haeng Ran Seo South Korea
Jack Leslie United Kingdom
Chris K. Sun Hong Kong
Thomas Amann Germany
Yaojie Fu United States
Eva Crosas‐Molist
Citations per year, relative to Eva Crosas‐Molist Eva Crosas‐Molist (= 1×) peers Miguel Murillo

Countries citing papers authored by Eva Crosas‐Molist

Since Specialization
Citations

This map shows the geographic impact of Eva Crosas‐Molist's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Crosas‐Molist with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Crosas‐Molist more than expected).

Fields of papers citing papers by Eva Crosas‐Molist

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Crosas‐Molist. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Crosas‐Molist. The network helps show where Eva Crosas‐Molist may publish in the future.

Co-authorship network of co-authors of Eva Crosas‐Molist

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Crosas‐Molist. A scholar is included among the top collaborators of Eva Crosas‐Molist based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Crosas‐Molist. Eva Crosas‐Molist is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Graziani, Vittoria, Aurélien Tripp, Óscar Maiques, et al.. (2025). SLC7A11 protects amoeboid-disseminating cancer cells from oxidative stress. Cell Reports. 44(7). 115939–115939.
2.
Graziani, Vittoria, Eva Crosas‐Molist, Samantha L. George, & Victoria Sanz‐Moreno. (2024). Organelle adaptations in response to mechanical forces during tumour dissemination. Current Opinion in Cell Biology. 88. 102345–102345. 5 indexed citations
3.
Crosas‐Molist, Eva, Vittoria Graziani, Óscar Maiques, et al.. (2023). AMPK is a mechano-metabolic sensor linking cell adhesion and mitochondrial dynamics to Myosin-dependent cell migration. Nature Communications. 14(1). 2740–2740. 48 indexed citations
4.
Crosas‐Molist, Eva, Daniel Caballero‐Díaz, Ania Alay, et al.. (2022). The NADPH oxidase NOX4 regulates redox and metabolic homeostasis preventing HCC progression. Hepatology. 78(2). 416–433. 34 indexed citations
5.
Winde, Charlotte M. de, Samantha L. George, Eva Crosas‐Molist, et al.. (2021). Podoplanin drives dedifferentiation and amoeboid invasion of melanoma. iScience. 24(9). 102976–102976. 11 indexed citations
6.
Maiques, Óscar, Bruce Fanshawe, Eva Crosas‐Molist, et al.. (2021). A preclinical pipeline to evaluate migrastatics as therapeutic agents in metastatic melanoma. British Journal of Cancer. 125(5). 699–713. 14 indexed citations
7.
Crosas‐Molist, Eva, Rémi Samain, José L. Orgaz, et al.. (2021). Rho GTPase signaling in cancer progression and dissemination. Physiological Reviews. 102(1). 455–510. 149 indexed citations
8.
López‐Luque, Judit, Ester Gonzalez‐Sanchez, Daniel Caballero‐Díaz, et al.. (2020). NADPH oxidase 4 (Nox4) deletion accelerates liver regeneration in mice. Redox Biology. 40. 101841–101841. 23 indexed citations
9.
Orgaz, José L., Eva Crosas‐Molist, Amine Sadok, et al.. (2020). Myosin II Reactivation and Cytoskeletal Remodeling as a Hallmark and a Vulnerability in Melanoma Therapy Resistance. Cancer Cell. 37(1). 85–103.e9. 96 indexed citations
10.
López‐Luque, Judit, Esther Bertrán, Eva Crosas‐Molist, et al.. (2019). Downregulation of Epidermal Growth Factor Receptor in hepatocellular carcinoma facilitates Transforming Growth Factor-β-induced epithelial to amoeboid transition. Cancer Letters. 464. 15–24. 22 indexed citations
11.
Valcárcel-Jiménez, Lorea, Eva Crosas‐Molist, Laura Camacho, et al.. (2019). PGC1α Suppresses Prostate Cancer Cell Invasion through ERRα Transcriptional Control. Cancer Research. 79(24). 6153–6165. 49 indexed citations
12.
Jiménez‐Altayó, Francesc, Thayna Meirelles, Eva Crosas‐Molist, et al.. (2018). Redox stress in Marfan syndrome: Dissecting the role of the NADPH oxidase NOX4 in aortic aneurysm. Free Radical Biology and Medicine. 118. 44–58. 59 indexed citations
13.
Malfettone, Andrea, Jitka Soukupová, Esther Bertrán, et al.. (2017). Transforming growth factor-β-induced plasticity causes a migratory stemness phenotype in hepatocellular carcinoma. Cancer Letters. 392. 39–50. 75 indexed citations
14.
Cantelli, Gaia, Eva Crosas‐Molist, Mirella Georgouli, & Victoria Sanz‐Moreno. (2016). TGFΒ-induced transcription in cancer. Seminars in Cancer Biology. 42. 60–69. 79 indexed citations
15.
Crosas‐Molist, Eva, Esther Bertrán, Irene Rodríguez‐Hernández, et al.. (2016). The NADPH oxidase NOX4 represses epithelial to amoeboid transition and efficient tumour dissemination. Oncogene. 36(21). 3002–3014. 58 indexed citations
16.
Herráiz, Cecilia, Eva Crosas‐Molist, & Victoria Sanz‐Moreno. (2016). Reactive oxygen species and tumor dissemination: Allies no longer. Molecular & Cellular Oncology. 3(2). e1127313–e1127313. 6 indexed citations
17.
Crosas‐Molist, Eva, Esther Bertrán, & Isabel Fabregat. (2015). Cross-Talk Between TGF-β and NADPH Oxidases During Liver Fibrosis and Hepatocarcinogenesis. Current Pharmaceutical Design. 21(41). 5964–5976. 32 indexed citations
18.
Crosas‐Molist, Eva & Isabel Fabregat. (2015). Role of NADPH oxidases in the redox biology of liver fibrosis. Redox Biology. 6. 106–111. 130 indexed citations
19.
Crosas‐Molist, Eva, Esther Bertrán, Patricia Sancho, et al.. (2014). The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression. Free Radical Biology and Medicine. 69. 338–347. 77 indexed citations
20.
Sancho, Patricia, Jèssica Mainez, Eva Crosas‐Molist, et al.. (2012). NADPH Oxidase NOX4 Mediates Stellate Cell Activation and Hepatocyte Cell Death during Liver Fibrosis Development. PLoS ONE. 7(9). e45285–e45285. 142 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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