Erik Selsing

3.6k total citations
58 papers, 3.0k citations indexed

About

Erik Selsing is a scholar working on Molecular Biology, Immunology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Erik Selsing has authored 58 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 29 papers in Immunology and 24 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Erik Selsing's work include T-cell and B-cell Immunology (26 papers), Monoclonal and Polyclonal Antibodies Research (24 papers) and DNA and Nucleic Acid Chemistry (12 papers). Erik Selsing is often cited by papers focused on T-cell and B-cell Immunology (26 papers), Monoclonal and Polyclonal Antibodies Research (24 papers) and DNA and Nucleic Acid Chemistry (12 papers). Erik Selsing collaborates with scholars based in United States, France and Japan. Erik Selsing's co-authors include Struther Arnott, Ursula Storb, Robert D. Wells, Mark W. Moore, Jeannine M. Durdik, Ronald D. Klein, Frederick W. Alt, Janet Stavnezer, Carol E. Schrader and Jim Miller and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Erik Selsing

58 papers receiving 2.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Erik Selsing United States 30 1.9k 1.4k 682 373 211 58 3.0k
M L Gefter United States 29 2.4k 1.3× 1.1k 0.8× 792 1.2× 761 2.0× 268 1.3× 44 3.8k
Joanne E. Hesse United States 23 2.1k 1.1× 1.4k 1.0× 329 0.5× 600 1.6× 434 2.1× 29 3.3k
C. Deborah Wilde United Kingdom 13 1.5k 0.8× 548 0.4× 887 1.3× 217 0.6× 176 0.8× 14 2.3k
Janet G. Pumphrey United States 16 1.2k 0.7× 1.1k 0.7× 488 0.7× 417 1.1× 436 2.1× 24 2.4k
Joëlle Wiels France 32 1.6k 0.9× 1.1k 0.8× 292 0.4× 321 0.9× 687 3.3× 91 3.3k
S.G. Nathenson United States 30 1.2k 0.7× 2.1k 1.5× 856 1.3× 310 0.8× 212 1.0× 56 3.1k
Joseph Haimovich Israel 29 1.5k 0.8× 1.2k 0.9× 1.3k 1.9× 131 0.4× 284 1.3× 89 2.8k
Gerald Walter Germany 21 1.6k 0.9× 674 0.5× 1.4k 2.1× 168 0.5× 129 0.6× 27 2.5k
Ira Pastan United States 34 1.9k 1.0× 2.2k 1.5× 1.5k 2.1× 305 0.8× 942 4.5× 71 4.2k
P Kourilsky France 20 764 0.4× 1.0k 0.7× 230 0.3× 328 0.9× 228 1.1× 53 2.0k

Countries citing papers authored by Erik Selsing

Since Specialization
Citations

This map shows the geographic impact of Erik Selsing's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Erik Selsing with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Erik Selsing more than expected).

Fields of papers citing papers by Erik Selsing

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Erik Selsing. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Erik Selsing. The network helps show where Erik Selsing may publish in the future.

Co-authorship network of co-authors of Erik Selsing

This figure shows the co-authorship network connecting the top 25 collaborators of Erik Selsing. A scholar is included among the top collaborators of Erik Selsing based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Erik Selsing. Erik Selsing is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Selsing, Erik, et al.. (2011). Translocation of an antibody transgene requires AID and occurs by interchromosomal switching to all switch regions except the mu switch region. European Journal of Immunology. 41(5). 1456–1464. 2 indexed citations
3.
Selsing, Erik, et al.. (2011). p21 is dispensable for AID-mediated class switch recombination and mutagenesis of immunoglobulin genes during somatic hypermutation. Molecular Immunology. 48(6-7). 973–978. 1 indexed citations
5.
Huang, Feng‐Ting, Kefei Yu, Erik Selsing, et al.. (2007). Sequence Dependence of Chromosomal R-Loops at the Immunoglobulin Heavy-Chain Sμ Class Switch Region. Molecular and Cellular Biology. 27(16). 5921–5932. 72 indexed citations
6.
Schrader, Carol E., Jeroen E. J. Guikema, Erin K. Linehan, Erik Selsing, & Janet Stavnezer. (2007). Activation-Induced Cytidine Deaminase-Dependent DNA Breaks in Class Switch Recombination Occur during G1 Phase of the Cell Cycle and Depend upon Mismatch Repair. The Journal of Immunology. 179(9). 6064–6071. 112 indexed citations
7.
8.
Wuerffel, Robert, Lili Wang, John Manis, et al.. (2007). S-S Synapsis during Class Switch Recombination Is Promoted by Distantly Located Transcriptional Elements and Activation-Induced Deaminase. Immunity. 27(5). 711–722. 162 indexed citations
9.
Truong, David M., et al.. (2005). Sequence Transfers between Variable Regions in a Mouse Antibody Transgene Can Occur by Gene Conversion. The Journal of Immunology. 175(12). 8133–8137. 8 indexed citations
10.
Min, Irene M., et al.. (2005). Shifts in targeting of class switch recombination sites in mice that lack μ switch region tandem repeats or Msh2. The Journal of Experimental Medicine. 201(12). 1885–1890. 25 indexed citations
11.
Min, Irene M., et al.. (2003). The Sμ Tandem Repeat Region Is Critical for Ig Isotype Switching in the Absence of Msh2. Immunity. 19(4). 515–524. 50 indexed citations
12.
Truong, David M., et al.. (2002). Gene Conversion-Like Sequence Transfers Between Transgenic Antibody V Genes Are Independent of RAD54. The Journal of Immunology. 169(6). 3069–3075. 7 indexed citations
13.
Selsing, Erik, et al.. (1996). Gene conversion and homologous recombination in murine B cells. Seminars in Immunology. 8(3). 151–158. 17 indexed citations
14.
Sohn, Jeongwon, et al.. (1993). Somatic hypermutation of an immunoglobulin mu heavy chain transgene.. The Journal of Experimental Medicine. 177(2). 493–504. 62 indexed citations
15.
Miller, Jim, et al.. (1990). Tissue-specific expression of allogeneic class II MHC molecules induces neither tissue rejection nor clonal inactivation of alloreactive T cells.. The Journal of Immunology. 144(1). 334–341. 47 indexed citations
16.
Smith, Graham A., et al.. (1990). Neonatal and adult primary B cells use the same germ-line VH and Vkappa genes in their (T,G)-A-L-specific repertoire.. The Journal of Immunology. 144(2). 583–592. 8 indexed citations
17.
Durdik, Jeannine M., et al.. (1989). Quantitative analysis of idiotypic mimicry and allelic exclusion in mice with a mu Ig transgene.. The Journal of Immunology. 143(6). 2074–2080. 14 indexed citations
18.
Selsing, Erik, et al.. (1989). DNase I sensitivity of immunoglobulin light chain genes in Abelson murine leukemia virus transformed pre-B cell lines. Nucleic Acids Research. 17(13). 5339–5348. 13 indexed citations
19.
Durdik, Jeannine M., et al.. (1987). Active lambda and kappa antibody gene rearrangement in Abelson murine leukemia virus-transformed pre-B cell lines.. The Journal of Experimental Medicine. 165(6). 1655–1674. 39 indexed citations
20.
Selsing, Erik, Andrew G. W. Leslie, Struther Arnott, et al.. (1976). Conformations of satellite DNAs. Nucleic Acids Research. 3(10). 2451–2458. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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