Eric T. Gangl
- Molecular Biology
- Spectroscopy top 5%
- Pharmacology top 5%
- Infectious Diseases top 10%
- Oncology
- Co-authors
- Paul VourosNicolas GerberIlya UtkinNeil SpoonerRobert J. TureskyBarry L. KargerFrantišek ForetHaihong Zhou
- Topics
- Cancer therapeutics and mechanisms (5 papers)Analytical Chemistry and Chromatography (4 papers)Estrogen and related hormone effects (4 papers)
- Cited by
- PharmacologySpectroscopyVirology
- Partner nations
- United StatesUnited KingdomSingapore
In The Last Decade
Eric T. Gangl
30 papers receiving 705 citations
Peers
Comparison fields: 5 of 79
- Molecular Biology 234
- Spectroscopy 209
- Pharmacology 139
- Infectious Diseases 137
- Oncology 124
Countries citing papers authored by Eric T. Gangl
This map shows the geographic impact of Eric T. Gangl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eric T. Gangl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eric T. Gangl more than expected).
Fields of papers citing papers by Eric T. Gangl
This network shows the impact of papers produced by Eric T. Gangl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eric T. Gangl. The network helps show where Eric T. Gangl may publish in the future.
Co-authorship network of co-authors of Eric T. Gangl
This figure shows the co-authorship network connecting the top 25 collaborators of Eric T. Gangl. A scholar is included among the top collaborators of Eric T. Gangl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eric T. Gangl. Eric T. Gangl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 2 | |
| 4 | 2 | |
| 5 | 8 | |
| 6 | 5 | |
| 7 | 3 | |
| 8 | 1 | |
| 9 | 1 | |
| 10 | 4 | |
| 11 | 19 | |
| 12 | 1 | |
| 13 | 19 | |
| 14 | 28 | |
| 15 | 30 | |
| 16 | 0 | |
| 17 | 71 | |
| 18 | 57 | |
| 19 | 43 | |
| 20 | Metabolism of the human immunodeficiency virus protease inhibitors indinavir and ritonavir by human intestinal microsomes and expressed cytochrome P4503A4/3A5: mechanism-based inactivation of cytochrome P4503A by ritonavir. | 194 |
About Eric T. Gangl
Eric T. Gangl is a scholar working on Toxicology, Pharmacology and Physiology, having authored 33 papers that have together received 733 indexed citations. Recurring topics across this work include Cancer therapeutics and mechanisms (5 papers), Analytical Chemistry and Chromatography (4 papers) and Estrogen and related hormone effects (4 papers). The work is most often cited by research in Pharmacology (139 citations), Spectroscopy (209 citations) and Virology (56 citations). Eric T. Gangl has collaborated with scholars based in United States, United Kingdom and Singapore. Frequent co-authors include Paul Vouros, Nicolas Gerber, Ilya Utkin, Neil Spooner, Robert J. Turesky, Barry L. Karger, František Foret, Haihong Zhou, James S. Scott and Petar Pop-Damkov. Their work appears in journals such as Journal of Clinical Oncology, Analytical Chemistry and Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.