Emily M. Rocha

2.8k total citations · 2 hit papers
23 papers, 2.0k citations indexed

About

Emily M. Rocha is a scholar working on Neurology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Emily M. Rocha has authored 23 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Neurology, 10 papers in Physiology and 9 papers in Cellular and Molecular Neuroscience. Recurrent topics in Emily M. Rocha's work include Parkinson's Disease Mechanisms and Treatments (16 papers), Lysosomal Storage Disorders Research (7 papers) and Cellular transport and secretion (6 papers). Emily M. Rocha is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (16 papers), Lysosomal Storage Disorders Research (7 papers) and Cellular transport and secretion (6 papers). Emily M. Rocha collaborates with scholars based in United States, United Kingdom and Italy. Emily M. Rocha's co-authors include Laurie H. Sanders, Penelope J. Hallett, Ole Isacson, Gaynor A. Smith, J. Timothy Greenamyre, Briana R. De Miranda, Jesse R. McLean, Eric Park, Hongmei Cao and Melissa A. Hayes and has published in prestigious journals such as PLoS ONE, Human Molecular Genetics and Science Translational Medicine.

In The Last Decade

Emily M. Rocha

22 papers receiving 2.0k citations

Hit Papers

Alpha-synuclein: Pathology, mitochondrial dysfunction and... 2017 2026 2020 2023 2017 2018 100 200 300 400 500

Peers

Emily M. Rocha
Lena F. Burbulla United States
Elpida Tsika United States
Adam L. Knight United States
Enrico Zampese United States
Anand Rane United States
Lena F. Burbulla United States
Emily M. Rocha
Citations per year, relative to Emily M. Rocha Emily M. Rocha (= 1×) peers Lena F. Burbulla

Countries citing papers authored by Emily M. Rocha

Since Specialization
Citations

This map shows the geographic impact of Emily M. Rocha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emily M. Rocha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emily M. Rocha more than expected).

Fields of papers citing papers by Emily M. Rocha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emily M. Rocha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emily M. Rocha. The network helps show where Emily M. Rocha may publish in the future.

Co-authorship network of co-authors of Emily M. Rocha

This figure shows the co-authorship network connecting the top 25 collaborators of Emily M. Rocha. A scholar is included among the top collaborators of Emily M. Rocha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emily M. Rocha. Emily M. Rocha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Thomas, Andrew P., et al.. (2024). A role for lysosomal calcium channels in mitigating mitochondrial damage and oxidative stress. Cell Calcium. 125. 102986–102986. 1 indexed citations
3.
Keeney, Matthew T., Emily M. Rocha, Eric K. Hoffman, et al.. (2024). LRRK2 regulates production of reactive oxygen species in cell and animal models of Parkinson’s disease. Science Translational Medicine. 16(767). eadl3438–eadl3438. 13 indexed citations
4.
Bellantuono, Ilaria, Heather Mortiboys, Emily M. Rocha, et al.. (2023). Aging, Parkinson’s Disease, and Models: What Are the Challenges?. PubMed. 1(1). 20230010–20230010. 3 indexed citations
5.
Mamais, Adamantios, Rebecca L. Wallings, & Emily M. Rocha. (2023). Disease mechanisms as subtypes: Lysosomal dysfunction in the endolysosomal Parkinson's disease subtype. Handbook of clinical neurology. 193. 33–51. 2 indexed citations
6.
Laar, Amber D. Van, Matthew T. Keeney, Victor S. Van Laar, et al.. (2023). Transient exposure to rotenone causes degeneration and progressive parkinsonian motor deficits, neuroinflammation, and synucleinopathy. npj Parkinson s Disease. 9(1). 121–121. 37 indexed citations
7.
Miranda, Briana R. De, et al.. (2021). The industrial solvent trichloroethylene induces LRRK2 kinase activity and dopaminergic neurodegeneration in a rat model of Parkinson's disease. Neurobiology of Disease. 153. 105312–105312. 40 indexed citations
8.
Miranda, Briana R. De, Emily M. Rocha, Sandra L. Castro, & J. Timothy Greenamyre. (2020). Protection from α-Synuclein induced dopaminergic neurodegeneration by overexpression of the mitochondrial import receptor TOM20. npj Parkinson s Disease. 6(1). 38–38. 25 indexed citations
9.
Miranda, Briana R. De, Marco Fazzari, Emily M. Rocha, Sandra L. Castro, & J. Timothy Greenamyre. (2019). Sex Differences in Rotenone Sensitivity Reflect the Male-to-Female Ratio in Human Parkinson’s Disease Incidence. Toxicological Sciences. 170(1). 133–143. 41 indexed citations
10.
Rocha, Emily M., Briana R. De Miranda, Sandra L. Castro, et al.. (2019). LRRK2 inhibition prevents endolysosomal deficits seen in human Parkinson's disease. Neurobiology of Disease. 134. 104626–104626. 61 indexed citations
11.
Maio, Roberto Di, Eric K. Hoffman, Emily M. Rocha, et al.. (2018). LRRK2 activation in idiopathic Parkinson’s disease. Science Translational Medicine. 10(451). 385 indexed citations breakdown →
12.
Miranda, Briana R. De, Emily M. Rocha, Qing Bai, et al.. (2018). Astrocyte-specific DJ-1 overexpression protects against rotenone-induced neurotoxicity in a rat model of Parkinson's disease. Neurobiology of Disease. 115. 101–114. 76 indexed citations
13.
Hallett, Penelope J., Mylene Huebecker, Oeystein Roed Brekk, et al.. (2018). Glycosphingolipid levels and glucocerebrosidase activity are altered in normal aging of the mouse brain. Neurobiology of Aging. 67. 189–200. 66 indexed citations
14.
Rocha, Emily M., et al.. (2017). Alpha-synuclein: Pathology, mitochondrial dysfunction and neuroinflammation in Parkinson’s disease. Neurobiology of Disease. 109(Pt B). 249–257. 555 indexed citations breakdown →
15.
Rocha, Emily M., Gaynor A. Smith, Eric Park, et al.. (2015). Sustained Systemic Glucocerebrosidase Inhibition Induces Brain α-Synuclein Aggregation, Microglia and Complement C1q Activation in Mice. Antioxidants and Redox Signaling. 23(6). 550–564. 117 indexed citations
16.
Rocha, Emily M., Gaynor A. Smith, Eric Park, et al.. (2015). Glucocerebrosidase gene therapy prevents α-synucleinopathy of midbrain dopamine neurons. Neurobiology of Disease. 82. 495–503. 129 indexed citations
17.
Smith, Gaynor A., Johan Jansson, Emily M. Rocha, et al.. (2015). Fibroblast Biomarkers of Sporadic Parkinson’s Disease and LRRK2 Kinase Inhibition. Molecular Neurobiology. 53(8). 5161–5177. 53 indexed citations
18.
McLean, Jesse R., Gaynor A. Smith, Emily M. Rocha, et al.. (2014). Widespread neuron-specific transgene expression in brain and spinal cord following synapsin promoter-driven AAV9 neonatal intracerebroventricular injection. Neuroscience Letters. 576. 73–78. 75 indexed citations
19.
Smith, Gaynor A., Emily M. Rocha, Jesse R. McLean, et al.. (2014). Progressive axonal transport and synaptic protein changes correlate with behavioral and neuropathological abnormalities in the heterozygous Q175 KI mouse model of Huntington's disease. Human Molecular Genetics. 23(17). 4510–4527. 77 indexed citations
20.
McLean, Jesse R., Gaynor A. Smith, Emily M. Rocha, et al.. (2014). ALS-associated peripherin spliced transcripts form distinct protein inclusions that are neuroprotective against oxidative stress. Experimental Neurology. 261. 217–229. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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