Elsa J. Brochmann Murray
- Co-authors
- Samuel S. MurrayHarold H. MesserCarlotta A. GlackinMario GrisantiArnold KahnMarshall R. UristKatherine A. WintersDan Gazit
- Topics
- Bone Metabolism and Diseases (14 papers)Protease and Inhibitor Mechanisms (5 papers)Calpain Protease Function and Regulation (4 papers)
- Partner nations
- United StatesBrazilUnited Kingdom
In The Last Decade
Elsa J. Brochmann Murray
30 papers receiving 571 citations
Peers
Comparison fields: 5 of 81
- Molecular Biology 352
- Oncology 111
- Cell Biology 92
- Genetics 74
- Nutrition and Dietetics 70
Countries citing papers authored by Elsa J. Brochmann Murray
This map shows the geographic impact of Elsa J. Brochmann Murray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elsa J. Brochmann Murray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elsa J. Brochmann Murray more than expected).
Fields of papers citing papers by Elsa J. Brochmann Murray
This network shows the impact of papers produced by Elsa J. Brochmann Murray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elsa J. Brochmann Murray. The network helps show where Elsa J. Brochmann Murray may publish in the future.
Co-authorship network of co-authors of Elsa J. Brochmann Murray
This figure shows the co-authorship network connecting the top 25 collaborators of Elsa J. Brochmann Murray. A scholar is included among the top collaborators of Elsa J. Brochmann Murray based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elsa J. Brochmann Murray. Elsa J. Brochmann Murray is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 8 | |
| 2 | 10 | |
| 3 | 5 | |
| 4 | 15 | |
| 5 | 22 | |
| 6 | 20 | |
| 7 | 8 | |
| 8 | 36 | |
| 9 | 44 | |
| 10 | 6 | |
| 11 | 33 | |
| 12 | 5 | |
| 13 | 1 | |
| 14 | 14 | |
| 15 | 34 | |
| 16 | Doxorubicin inhibits differentiation and enhances expression of the helix-loop-helix genes Id and mTwi in mouse osteoblastic cells. | 22 |
| 17 | 6 | |
| 18 | 35 | |
| 19 | 64 | |
| 20 | 6 |
About Elsa J. Brochmann Murray
Elsa J. Brochmann Murray is a scholar working on Anatomy, Cancer Research and Orthopedics and Sports Medicine, having authored 30 papers that have together received 582 indexed citations. Recurring topics across this work include Bone Metabolism and Diseases (14 papers), Protease and Inhibitor Mechanisms (5 papers) and Calpain Protease Function and Regulation (4 papers). The work is most often cited by research in Aging (10 citations), Cell Biology (92 citations) and Orthopedics and Sports Medicine (48 citations). Elsa J. Brochmann Murray has collaborated with scholars based in United States, Brazil and United Kingdom. Frequent co-authors include Samuel S. Murray, Harold H. Messer, Carlotta A. Glackin, Mario Grisanti, Arnold Kahn, Marshall R. Urist, Katherine A. Winters, Dan Gazit, Kewei Zhao and S C Manolagas. Their work appears in journals such as Proceedings of the National Academy of Sciences, Endocrinology and Kidney International.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.