Elizabeth Mazzio
- Molecular Biology top 10%
- Plant Science top 10%
- Cellular and Molecular Neuroscience top 10%
- Cancer Research top 10%
- Pharmacology top 5%
- Co-authors
- Karam F. A. SolimanRenee ReamsDavid F. BauerNatholyn D. HarrisRamesh BadisaCarl B. GoodmanEquar TakaHernan Flores‐Rozas
- Topics
- Neuroinflammation and Neurodegeneration Mechanisms (12 papers)Mitochondrial Function and Pathology (10 papers)Parkinson's Disease Mechanisms and Treatments (10 papers)
- Partner nations
- United StatesAustraliaPuerto Rico
In The Last Decade
Elizabeth Mazzio
88 papers receiving 2.1k citations
Peers
Comparison fields: 5 of 131
- Molecular Biology 917
- Plant Science 266
- Cellular and Molecular Neuroscience 257
- Cancer Research 227
- Pharmacology 221
Countries citing papers authored by Elizabeth Mazzio
This map shows the geographic impact of Elizabeth Mazzio's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elizabeth Mazzio with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elizabeth Mazzio more than expected).
Fields of papers citing papers by Elizabeth Mazzio
This network shows the impact of papers produced by Elizabeth Mazzio. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elizabeth Mazzio. The network helps show where Elizabeth Mazzio may publish in the future.
Co-authorship network of co-authors of Elizabeth Mazzio
This figure shows the co-authorship network connecting the top 25 collaborators of Elizabeth Mazzio. A scholar is included among the top collaborators of Elizabeth Mazzio based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elizabeth Mazzio. Elizabeth Mazzio is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 1 | |
| 3 | 1 | |
| 4 | 11 | |
| 5 | 16 | |
| 6 | 23 | |
| 7 | 37 | |
| 8 | 7 | |
| 9 | 33 | |
| 10 | 15 | |
| 11 | 8 | |
| 12 | 22 | |
| 13 | 74 | |
| 14 | 19 | |
| 15 | 55 | |
| 16 | 16 | |
| 17 | 40 | |
| 18 | 38 | |
| 19 | 24 | |
| 20 | 17 |
About Elizabeth Mazzio
Elizabeth Mazzio is a scholar working on Neurology, Applied Microbiology and Biotechnology and Toxicology, having authored 90 papers that have together received 2.2k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (12 papers), Mitochondrial Function and Pathology (10 papers) and Parkinson's Disease Mechanisms and Treatments (10 papers). The work is most often cited by research in Biological Psychiatry (79 citations), Complementary and alternative medicine (205 citations) and Biochemistry (142 citations). Elizabeth Mazzio has collaborated with scholars based in United States, Australia and Puerto Rico. Frequent co-authors include Karam F. A. Soliman, Renee Reams, David F. Bauer, Natholyn D. Harris, Ramesh Badisa, Carl B. Goodman, Equar Taka, Hernan Flores‐Rozas, Selina Darling‐Reed and Suresh Eyunni. Their work appears in journals such as PLoS ONE, The Journal of Physical Chemistry B and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.