Elise Holvey-Bates

705 total citations · 1 hit paper
10 papers, 511 citations indexed

About

Elise Holvey-Bates is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Elise Holvey-Bates has authored 10 papers receiving a total of 511 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Immunology and 7 papers in Oncology. Recurrent topics in Elise Holvey-Bates's work include interferon and immune responses (8 papers), Cytokine Signaling Pathways and Interactions (5 papers) and RNA regulation and disease (3 papers). Elise Holvey-Bates is often cited by papers focused on interferon and immune responses (8 papers), Cytokine Signaling Pathways and Interactions (5 papers) and RNA regulation and disease (3 papers). Elise Holvey-Bates collaborates with scholars based in United States, China and Australia. Elise Holvey-Bates's co-authors include George R. Stark, HyeonJoo Cheon, John W. Schoggins, Paul J. Hertzog, Charles M. Rice, Mark W. Jackson, Naoko Imanaka, Samuel C. Forster, Damian J. Junk and Belinda Willard and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The EMBO Journal and Cancer Research.

In The Last Decade

Elise Holvey-Bates

9 papers receiving 509 citations

Hit Papers

IFNβ-dependent increases in STAT1, STAT2, and IRF9 mediat... 2013 2026 2017 2021 2013 50 100 150 200 250

Peers

Elise Holvey-Bates
Mi Li China
Sarah Van Scoy United States
Emily Hopewell United States
Kaja Kostyrko United States
Céline Robert-Tissot United States
Seoyun Yum United States
Diane Maurice United Kingdom
Mi Li China
Elise Holvey-Bates
Citations per year, relative to Elise Holvey-Bates Elise Holvey-Bates (= 1×) peers Mi Li

Countries citing papers authored by Elise Holvey-Bates

Since Specialization
Citations

This map shows the geographic impact of Elise Holvey-Bates's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elise Holvey-Bates with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elise Holvey-Bates more than expected).

Fields of papers citing papers by Elise Holvey-Bates

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Elise Holvey-Bates. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elise Holvey-Bates. The network helps show where Elise Holvey-Bates may publish in the future.

Co-authorship network of co-authors of Elise Holvey-Bates

This figure shows the co-authorship network connecting the top 25 collaborators of Elise Holvey-Bates. A scholar is included among the top collaborators of Elise Holvey-Bates based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elise Holvey-Bates. Elise Holvey-Bates is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Wang, Chenyao, Nan Jing, Elise Holvey-Bates, et al.. (2023). STAT2 hinders STING intracellular trafficking and reshapes its activation in response to DNA damage. Proceedings of the National Academy of Sciences. 120(16). e2216953120–e2216953120. 12 indexed citations
2.
De, Sarmishtha, et al.. (2021). The ubiquitin E3 ligase FBXO22 degrades PD-L1 and sensitizes cancer cells to DNA damage. Proceedings of the National Academy of Sciences. 118(47). 63 indexed citations
3.
Cheon, HyeonJoo, Elise Holvey-Bates, Daniel J. McGrail, & George R. Stark. (2021). PD-L1 sustains chronic, cancer cell–intrinsic responses to type I interferon, enhancing resistance to DNA damage. Proceedings of the National Academy of Sciences. 118(47). 45 indexed citations
4.
Kondratova, Anna A., HyeonJoo Cheon, Beihua Dong, et al.. (2020). Suppressing PAR ylation by 2′,5′‐oligoadenylate synthetase 1 inhibits DNA damage‐induced cell death. The EMBO Journal. 39(11). e101573–e101573. 20 indexed citations
5.
Wang, Yuxin, Qiaoling Song, Wei Huang, et al.. (2020). A virus-induced conformational switch of STAT1-STAT2 dimers boosts antiviral defenses. Cell Research. 31(2). 206–218. 39 indexed citations
6.
Banerjee, Shuvojit, Elona Gusho, Christina Gaughan, et al.. (2019). OAS-RNase L innate immune pathway mediates the cytotoxicity of a DNA-demethylating drug. Proceedings of the National Academy of Sciences. 116(11). 5071–5076. 50 indexed citations
7.
Cheon, HyeonJoo, Elise Holvey-Bates, & George R. Stark. (2014). Abstract 485: Chronic exposure to a low dose of interferon-β increases levels of DNA damage resistance signature genes through unphosphorylated ISGF3. Cancer Research. 74(19_Supplement). 485–485. 1 indexed citations
8.
Kolosenko, Iryna, Mårten Fryknäs, Per Johnsson, et al.. (2014). Cell crowding induces interferon regulatory factor 9, which confers resistance to chemotherapeutic drugs. International Journal of Cancer. 136(4). E51–61. 27 indexed citations
9.
Cheon, HyeonJoo, Elise Holvey-Bates, John W. Schoggins, et al.. (2013). IFNβ-dependent increases in STAT1, STAT2, and IRF9 mediate resistance to viruses and DNA damage. The EMBO Journal. 32(20). 2751–2763. 254 indexed citations breakdown →
10.
Cheon, HyeonJoo, Elise Holvey-Bates, & George R. Stark. (2013). 44. Cytokine. 63(3). 253–254.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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