Elena M. Ribé

3.5k total citations
24 papers, 2.0k citations indexed

About

Elena M. Ribé is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Elena M. Ribé has authored 24 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 17 papers in Physiology and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in Elena M. Ribé's work include Alzheimer's disease research and treatments (17 papers), Cell death mechanisms and regulation (5 papers) and Nuclear Receptors and Signaling (4 papers). Elena M. Ribé is often cited by papers focused on Alzheimer's disease research and treatments (17 papers), Cell death mechanisms and regulation (5 papers) and Nuclear Receptors and Signaling (4 papers). Elena M. Ribé collaborates with scholars based in United Kingdom, United States and Spain. Elena M. Ribé's co-authors include Carol M. Troy, Simon Lovestone, Isidró Ferrer, Teresa Gómez‐Isla, Esther Dalfó, Berta Puig, Noel J. Buckley, Evangeline M. Foster, Jesús Ávila and Ottavio Arancio and has published in prestigious journals such as Nature Communications, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Elena M. Ribé

24 papers receiving 2.0k citations

Peers

Elena M. Ribé
Michela Guglielmotto Italy
Paige E. Cramer United States
Faraj Terro France
Wendy Sigurdson United States
Jian–Zhi Wang China
Guylène Page France
Lih-Fen Lue United States
Magali Dumont United States
Janaky Coomaraswamy Germany
Michela Guglielmotto Italy
Elena M. Ribé
Citations per year, relative to Elena M. Ribé Elena M. Ribé (= 1×) peers Michela Guglielmotto

Countries citing papers authored by Elena M. Ribé

Since Specialization
Citations

This map shows the geographic impact of Elena M. Ribé's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elena M. Ribé with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elena M. Ribé more than expected).

Fields of papers citing papers by Elena M. Ribé

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Elena M. Ribé. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elena M. Ribé. The network helps show where Elena M. Ribé may publish in the future.

Co-authorship network of co-authors of Elena M. Ribé

This figure shows the co-authorship network connecting the top 25 collaborators of Elena M. Ribé. A scholar is included among the top collaborators of Elena M. Ribé based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elena M. Ribé. Elena M. Ribé is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Elliott, Christina, Joshua Jackson, J. N. Findlay, et al.. (2025). Blocking the Dkk1-LRP6 interaction prevents acute amyloid-β-driven cognitive impairment. Cellular Signalling. 131. 111716–111716. 1 indexed citations
2.
Killick, Richard, Christina Elliott, Elena M. Ribé, et al.. (2023). Neurodegenerative Disease Associated Pathways in the Brains of Triple Transgenic Alzheimer’s Model Mice Are Reversed Following Two Weeks of Peripheral Administration of Fasudil. International Journal of Molecular Sciences. 24(13). 11219–11219. 9 indexed citations
3.
Foster, Evangeline M., Marco Fernandes, Philip Hublitz, et al.. (2022). Glycosylated clusterin species facilitate Aβ toxicity in human neurons. Scientific Reports. 12(1). 18639–18639. 6 indexed citations
4.
Martín‐Sánchez, Ana, Janet Piñero, Lara Nonell, et al.. (2021). Comorbidity between Alzheimer’s disease and major depression: a behavioural and transcriptomic characterization study in mice. Alzheimer s Research & Therapy. 13(1). 73–73. 28 indexed citations
5.
Foster, Evangeline M., et al.. (2019). Clusterin in Alzheimer’s Disease: Mechanisms, Genetics, and Lessons From Other Pathologies. Frontiers in Neuroscience. 13. 164–164. 253 indexed citations
6.
Nevado‐Holgado, Alejo, Elena M. Ribé, Laura Thei, et al.. (2019). Genetic and Real-World Clinical Data, Combined with Empirical Validation, Nominate Jak-Stat Signaling as a Target for Alzheimer’s Disease Therapeutic Development. Cells. 8(5). 425–425. 36 indexed citations
7.
Elliott, Christina, Ana I. Rojo, Elena M. Ribé, et al.. (2018). A role for APP in Wnt signalling links synapse loss with β-amyloid production. Translational Psychiatry. 8(1). 179–179. 80 indexed citations
8.
Robbins, J., Elena M. Ribé, Marcello Maresca, et al.. (2018). Clusterin Is Required for β-Amyloid Toxicity in Human iPSC-Derived Neurons. Frontiers in Neuroscience. 12. 504–504. 41 indexed citations
9.
Rojo, Ana I., Marta Pajares, Patricia Rada, et al.. (2017). NRF2 deficiency replicates transcriptomic changes in Alzheimer's patients and worsens APP and TAU pathology. Redox Biology. 13. 444–451. 181 indexed citations
10.
Pozueta, Julio, Roger Lefort, Elena M. Ribé, et al.. (2013). Caspase-2 is required for dendritic spine and behavioural alterations in J20 APP transgenic mice. Nature Communications. 4(1). 1939–1939. 82 indexed citations
11.
Ribé, Elena M., Ying Y. Jean, Rebecca Goldstein, et al.. (2012). Neuronal caspase 2 activity and function requires RAIDD, but not PIDD. Biochemical Journal. 444(3). 591–599. 33 indexed citations
12.
Ribé, Elena M., et al.. (2011). The ε3 and ε4 Alleles of Human APOE Differentially Affect Tau Phosphorylation in Hyperinsulinemic and Pioglitazone Treated Mice. PLoS ONE. 6(2). e16991–e16991. 36 indexed citations
13.
Tizón, Belén, Elena M. Ribé, Weiqian Mi, Carol M. Troy, & Efrat Levy. (2010). Cystatin C Protects Neuronal Cells from Amyloid-β-induced Toxicity. Journal of Alzheimer s Disease. 19(3). 885–894. 94 indexed citations
14.
Puzzo, Daniela, Lucia Privitera, Mauro Fà, et al.. (2010). Endogenous amyloid‐β is necessary for hippocampal synaptic plasticity and memory. Annals of Neurology. 69(5). 819–830. 245 indexed citations
15.
16.
Nieto, M. Ángela, Francisco J. Gil‐Bea, Esther Dalfó, et al.. (2005). Increased sensitivity to MPTP in human α-synuclein A30P transgenic mice. Neurobiology of Aging. 27(6). 848–856. 80 indexed citations
17.
Ribé, Elena M., Mar Pérez, Berta Puig, et al.. (2005). Accelerated amyloid deposition, neurofibrillary degeneration and neuronal loss in double mutant APP/tau transgenic mice. Neurobiology of Disease. 20(3). 814–822. 128 indexed citations
18.
Ferrer, Isidró, Teresa Gómez‐Isla, Berta Puig, et al.. (2005). Current Advances on Different Kinases Involved in Tau Phosphorylation, and Implications in Alzheimers Disease and Tauopathies. Current Alzheimer Research. 2(1). 3–18. 254 indexed citations
19.
Pérez, Mar, Elena M. Ribé, Alicia Rubio, et al.. (2004). Characterization of a double (amyloid precursor protein-tau) transgenic: Tau phosphorylation and aggregation. Neuroscience. 130(2). 339–347. 61 indexed citations
20.
Puig, Berta, Teresa Gómez‐Isla, Elena M. Ribé, et al.. (2004). Expression of stress‐activated kinases c‐Jun N‐terminal kinase (SAPK/JNK‐P) and p38 kinase (p38‐P), and tau hyperphosphorylation in neurites surrounding βA plaques in APP Tg2576 mice. Neuropathology and Applied Neurobiology. 30(5). 491–502. 74 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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