Ehsan Pourkarimi

903 total citations
17 papers, 664 citations indexed

About

Ehsan Pourkarimi is a scholar working on Molecular Biology, Aging and Physiology. According to data from OpenAlex, Ehsan Pourkarimi has authored 17 papers receiving a total of 664 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Aging and 3 papers in Physiology. Recurrent topics in Ehsan Pourkarimi's work include Genetics, Aging, and Longevity in Model Organisms (8 papers), DNA Repair Mechanisms (2 papers) and Mitochondrial Function and Pathology (2 papers). Ehsan Pourkarimi is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (8 papers), DNA Repair Mechanisms (2 papers) and Mitochondrial Function and Pathology (2 papers). Ehsan Pourkarimi collaborates with scholars based in United Kingdom, Qatar and United States. Ehsan Pourkarimi's co-authors include Anton Gartner, Sarah J. Coulthurst, Sarah Murdoch, Katharina Trunk, Maximilian Fritsch, Grant English, Angus I. Lamond, Ronald T. Hay, Mark Larance and Sebastian Greiss and has published in prestigious journals such as Nature Communications, PLoS ONE and Development.

In The Last Decade

Ehsan Pourkarimi

15 papers receiving 659 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ehsan Pourkarimi United Kingdom	11	413	191	129	59	59	17	664
Lucía Herrera-Domínguez United Kingdom	10	440 1.1×	58 0.3×	14 0.1×	21 0.4×	14 0.2×	10	598
Pengpeng Li China	14	293 0.7×	36 0.2×	20 0.2×	44 0.7×	39 0.7×	29	676
Michael Hoffmann Germany	17	490 1.2×	67 0.4×	7 0.1×	31 0.5×	58 1.0×	40	725
Tatsuyuki Kamiryo Japan	21	857 2.1×	42 0.2×	12 0.1×	66 1.1×	76 1.3×	41	1.0k
Verena Kohler Sweden	14	413 1.0×	16 0.1×	18 0.1×	35 0.6×	131 2.2×	25	675
Matías Cabruja Argentina	8	205 0.5×	19 0.1×	65 0.5×	99 1.7×	8 0.1×	12	334
Michal Čáp Czechia	7	280 0.7×	60 0.3×	18 0.1×	6 0.1×	20 0.3×	9	397
Horia Todor United States	12	734 1.8×	12 0.1×	46 0.4×	46 0.8×	22 0.4×	19	903
Andreas Kastenmüller Germany	8	438 1.1×	20 0.1×	27 0.2×	14 0.2×	109 1.8×	8	522
Alena Pichová Czechia	11	584 1.4×	190 1.0×	9 0.1×	4 0.1×	77 1.3×	20	674

Countries citing papers authored by Ehsan Pourkarimi

Since Specialization

Citations

This map shows the geographic impact of Ehsan Pourkarimi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ehsan Pourkarimi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ehsan Pourkarimi more than expected).

Fields of papers citing papers by Ehsan Pourkarimi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ehsan Pourkarimi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ehsan Pourkarimi. The network helps show where Ehsan Pourkarimi may publish in the future.

Co-authorship network of co-authors of Ehsan Pourkarimi

This figure shows the co-authorship network connecting the top 25 collaborators of Ehsan Pourkarimi. A scholar is included among the top collaborators of Ehsan Pourkarimi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ehsan Pourkarimi. Ehsan Pourkarimi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Ali, Tayyiba Akbar, et al.. (2025). Depletion of tryptophanyl-tRNA synthetase and tryptophan accumulation triggers p53-dependent apoptosis. Cell Death Discovery. 12(1). 34–34.

Ali, Tayyiba Akbar, et al.. (2025). Thiostrepton induces apoptotic cell death at the level of BCL-2/CED-9 in C. elegans. Scientific Reports. 15(1). 24415–24415.

Kuttikrishnan, Shilpa, Kirti S. Prabhu, Tamam El‐Elimat, et al.. (2024). Exploring the in vivo anti-cancer potential of Neosetophomone B in leukemic cells using a zebrafish xenograft model. Experimental Cell Research. 435(1). 113907–113907. 1 indexed citations

Prabhu, Kirti S., Fareed Ahmad, Shilpa Kuttikrishnan, et al.. (2024). Bortezomib exerts its anti-cancer activity through the regulation of Skp2/p53 axis in non-melanoma skin cancer cells and C. elegans. Cell Death Discovery. 10(1). 225–225. 6 indexed citations

Ali, Tayyiba Akbar, et al.. (2024). Tryptophanyl-tRNA synthetase-1 (WARS-1) depletion and high tryptophan concentration lead to genomic instability in Caenorhabditis elegans. Cell Death Discovery. 10(1). 165–165. 3 indexed citations

Bejaoui, Yosra, Noha A. Yousri, Colette Christiansen, et al.. (2023). Accelerated epigenetic aging and DNA methylation alterations in Berardinelli–Seip congenital lipodystrophy. Human Molecular Genetics. 32(11). 1826–1835. 8 indexed citations

Ali, Tayyiba Akbar, et al.. (2021). Over Fifty Years of Life, Death, and Cannibalism: A Historical Recollection of Apoptosis and Autophagy. International Journal of Molecular Sciences. 22(22). 12466–12466. 19 indexed citations

Narayan, Vikram, Tony Ly, Ehsan Pourkarimi, et al.. (2016). Deep Proteome Analysis Identifies Age-Related Processes in C. elegans. Cell Systems. 3(2). 144–159. 85 indexed citations

Pourkarimi, Ehsan, et al.. (2016). Spatiotemporal coupling and decoupling of gene transcription with DNA replication origins during embryogenesis in C. elegans. eLife. 5. 43 indexed citations

10.

Larance, Mark, Ehsan Pourkarimi, Bin Wang, et al.. (2015). Global Proteomics Analysis of the Response to Starvation in C. elegans*. Molecular & Cellular Proteomics. 14(7). 1989–2001. 33 indexed citations

11.

Pelisch, Federico, Romain Sonneville, Ehsan Pourkarimi, et al.. (2014). Dynamic SUMO modification regulates mitotic chromosome assembly and cell cycle progression in Caenorhabditis elegans. Nature Communications. 5(1). 5485–5485. 53 indexed citations

12.

Farkas, Zsolt, Ehsan Pourkarimi, Maja Herak Bosnar, et al.. (2014). NDK-1, the Homolog of NM23-H1/H2 Regulates Cell Migration and Apoptotic Engulfment in C. elegans. PLoS ONE. 9(3). e92687–e92687. 21 indexed citations

13.

Akay, Alper, Ashley Craig, Nicolas J. Lehrbach, et al.. (2013). RNA-binding protein GLD-1/quaking genetically interacts with themir-35andthe let-7 miRNA pathways inCaenorhabditis elegans. Open Biology. 3(11). 130151–130151. 18 indexed citations

14.

Pourkarimi, Ehsan, Tibor Vellai, Anita Alexa, et al.. (2013). The NM23-H1/H2 homolog NDK-1 is required for full activation of Ras signaling inC. elegans. Development. 140(16). 3486–3495. 29 indexed citations

15.

Murdoch, Sarah, Katharina Trunk, Grant English, et al.. (2011). The Opportunistic Pathogen Serratia marcescens Utilizes Type VI Secretion To Target Bacterial Competitors. Journal of Bacteriology. 193(21). 6057–6069. 199 indexed citations

16.

Larance, Mark, Aymeric Bailly, Ehsan Pourkarimi, et al.. (2011). Stable-isotope labeling with amino acids in nematodes. Nature Methods. 8(10). 849–851. 96 indexed citations

17.

Pourkarimi, Ehsan, Sebastian Greiss, & Anton Gartner. (2011). Evidence that CED-9/Bcl2 and CED-4/Apaf-1 localization is not consistent with the current model for C. elegans apoptosis induction. Cell Death and Differentiation. 19(3). 406–415. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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