Edward Nylen
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 10%
- Cardiology and Cardiovascular Medicine top 10%
- Physiology
- Cell Biology top 10%
- Co-authors
- Klaus WrogemannCheryl R. GreenbergTracey WeilerKenneth MorganTakuya FujiwaraEduardo RosenmannPatrick FroskThangirala Sudha
- Topics
- Muscle Physiology and Disorders (10 papers)Genetic Neurodegenerative Diseases (7 papers)Muscle metabolism and nutrition (4 papers)
- Partner nations
- CanadaUnited KingdomNetherlands
In The Last Decade
Edward Nylen
17 papers receiving 859 citations
Peers
Comparison fields: 5 of 68
- Molecular Biology 800
- Cellular and Molecular Neuroscience 229
- Cardiology and Cardiovascular Medicine 192
- Physiology 147
- Cell Biology 135
Countries citing papers authored by Edward Nylen
This map shows the geographic impact of Edward Nylen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward Nylen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward Nylen more than expected).
Fields of papers citing papers by Edward Nylen
This network shows the impact of papers produced by Edward Nylen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward Nylen. The network helps show where Edward Nylen may publish in the future.
Co-authorship network of co-authors of Edward Nylen
This figure shows the co-authorship network connecting the top 25 collaborators of Edward Nylen. A scholar is included among the top collaborators of Edward Nylen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edward Nylen. Edward Nylen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 8 | |
| 2 | 53 | |
| 3 | 10 | |
| 4 | 188 | |
| 5 | 102 | |
| 6 | 142 | |
| 7 | 84 | |
| 8 | 11 | |
| 9 | Limb-girdle muscular dystrophy and Miyoshi myopathy in an aboriginal Canadian kindred map to LGMD2B and segregate with the same haplotype. | 74 |
| 10 | 4 | |
| 11 | 47 | |
| 12 | 23 | |
| 13 | 28 | |
| 14 | 77 | |
| 15 | 14 | |
| 16 | 17 | |
| 17 | 6 |
About Edward Nylen
Edward Nylen is a scholar working on Cellular and Molecular Neuroscience, Geriatrics and Gerontology and Cell Biology, having authored 17 papers that have together received 888 indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (10 papers), Genetic Neurodegenerative Diseases (7 papers) and Muscle metabolism and nutrition (4 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (229 citations), Molecular Biology (800 citations) and Genetics (108 citations). Edward Nylen has collaborated with scholars based in Canada, United Kingdom and Netherlands. Frequent co-authors include Klaus Wrogemann, Cheryl R. Greenberg, Tracey Weiler, Kenneth Morgan, Takuya Fujiwara, Eduardo Rosenmann, Patrick Frosk, Thangirala Sudha, Marshall J. Glesby and Kate Bushby. Their work appears in journals such as The Lancet, Neurology and Biochemical and Biophysical Research Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.