E.A. Frey

1.7k total citations · 2 hit papers
15 papers, 1.5k citations indexed

About

E.A. Frey is a scholar working on Endocrinology, Diabetes and Metabolism, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, E.A. Frey has authored 15 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Endocrinology, Diabetes and Metabolism, 5 papers in Molecular Biology and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in E.A. Frey's work include Receptor Mechanisms and Signaling (5 papers), Growth Hormone and Insulin-like Growth Factors (4 papers) and Neuropeptides and Animal Physiology (3 papers). E.A. Frey is often cited by papers focused on Receptor Mechanisms and Signaling (5 papers), Growth Hormone and Insulin-like Growth Factors (4 papers) and Neuropeptides and Animal Physiology (3 papers). E.A. Frey collaborates with scholars based in United States, Cameroon and Switzerland. E.A. Frey's co-authors include C. E. Thomas, C. W. GREWE, John W. Kebabian, V Bažil, Terje Espevik, Samuel D. Wright, Anders Sundan, David S. Miller, B. Brett Finlay and Robert L. Eskay and has published in prestigious journals such as Nature, Journal of Biological Chemistry and The Journal of Experimental Medicine.

In The Last Decade

E.A. Frey

15 papers receiving 1.4k citations

Hit Papers

Soluble CD14 participates in the response of cells to lip... 1992 2026 2003 2014 1992 2024 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
E.A. Frey United States 13 644 539 475 142 133 15 1.5k
K J Collier United States 10 717 1.1× 413 0.8× 629 1.3× 89 0.6× 119 0.9× 14 1.6k
Satoru Nakai Japan 19 608 0.9× 371 0.7× 803 1.7× 220 1.5× 56 0.4× 41 2.2k
Carmen Martı́nez Spain 27 972 1.5× 277 0.5× 492 1.0× 326 2.3× 323 2.4× 59 2.1k
Carmen Romero Chile 30 575 0.9× 265 0.5× 497 1.0× 105 0.7× 122 0.9× 89 2.3k
S. Ellis United States 21 710 1.1× 326 0.6× 137 0.3× 343 2.4× 395 3.0× 52 1.8k
Jacqueline Higelin Switzerland 13 448 0.7× 288 0.5× 319 0.7× 109 0.8× 103 0.8× 15 936
Lex Nagelkerken Netherlands 27 571 0.9× 192 0.4× 1.2k 2.4× 238 1.7× 77 0.6× 69 2.4k
Sigrid Wattler United States 14 1.2k 1.9× 280 0.5× 401 0.8× 155 1.1× 116 0.9× 17 2.1k
B Delpech France 11 540 0.8× 426 0.8× 155 0.3× 134 0.9× 85 0.6× 14 1.2k
M. T. Castells Spain 22 527 0.8× 156 0.3× 167 0.4× 97 0.7× 68 0.5× 48 1.1k

Countries citing papers authored by E.A. Frey

Since Specialization
Citations

This map shows the geographic impact of E.A. Frey's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E.A. Frey with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E.A. Frey more than expected).

Fields of papers citing papers by E.A. Frey

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E.A. Frey. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E.A. Frey. The network helps show where E.A. Frey may publish in the future.

Co-authorship network of co-authors of E.A. Frey

This figure shows the co-authorship network connecting the top 25 collaborators of E.A. Frey. A scholar is included among the top collaborators of E.A. Frey based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E.A. Frey. E.A. Frey is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Frey, E.A., Philipp A. Gerber, Marco Kaufmann, et al.. (2024). GLP-1 receptor agonists for weight reduction in people living with obesity but without diabetes: a living benefit–harm modelling study. EClinicalMedicine. 73. 102661–102661. 42 indexed citations breakdown →
2.
Frey, E.A., David S. Miller, Anders Sundan, et al.. (1992). Soluble CD14 participates in the response of cells to lipopolysaccharide.. The Journal of Experimental Medicine. 176(6). 1665–1671. 591 indexed citations breakdown →
3.
Crawford, Keith W., E.A. Frey, & C. E. Thomas. (1992). Angiotensin II receptor recognized by DuP753 regulates two distinct guanine nucleotide-binding protein signaling pathways.. Molecular Pharmacology. 41(1). 154–162. 38 indexed citations
4.
Aub, D L, E.A. Frey, Ronald D. Sekura, & C. E. Thomas. (1986). Coupling of the thyrotropin-releasing hormone receptor to phospholipase C by a GTP-binding protein distinct from the inhibitory or stimulatory GTP-binding protein.. Journal of Biological Chemistry. 261(20). 9333–9340. 74 indexed citations
5.
Frey, E.A., John W. Kebabian, & Sarah‐Jane Guild. (1986). Forskolin enhances calcium-evoked prolactin release from 7315c tumor cells without increasing the cytosolic calcium concentration.. Molecular Pharmacology. 29(5). 461–466. 8 indexed citations
6.
Thomas, C. E., E.A. Frey, & Ronald D. Sekura. (1984). Altered activity of the inhibitory guanyl nucleotide-binding component (Ni) induced by pertussis toxin. Uncoupling of Ni from receptor with continued coupling of Ni to the catalytic unit.. Journal of Biological Chemistry. 259(14). 8693–8698. 87 indexed citations
7.
Frey, E.A. & J. W. Kebabian. (1984). Aα-Opiate Receptor in 7315c Tumor Tissue Mediates Inhibition of Immunoreactive Prolactin Release and Adenylate Cyclase Activity. Endocrinology. 115(5). 1797–1804. 52 indexed citations
8.
Beaulieu, Jean‐Martin, Mark E. Goldman, Kazuhiro Miyazaki, et al.. (1984). Bromocriptine-Induced Changes in the Biochemistry, Physiology, and Histology of the Intermediate Lobe of the Rat Pituitary Gland. Endocrinology. 114(5). 1871–1884. 51 indexed citations
9.
Frey, E.A., et al.. (1983). [Elevated prolactin reserves in gonadal dysgenesis and agonadism].. PubMed. 38(4). 315–21. 2 indexed citations
10.
Thomas, C. E., Robert L. Eskay, E.A. Frey, et al.. (1982). Biochemical and Physiological Studies of the Beta-Adrenoceptor and the D-2 Dopamine Receptor in the Intermediate Lobe of the Rat Pituitary Gland: A Review. Neuroendocrinology. 35(3). 217–224. 112 indexed citations
11.
GREWE, C. W., E.A. Frey, C. E. Thomas, & John W. Kebabian. (1982). YM-09151-2: A potent antagonist for a peripheral D2-dopamine receptor. European Journal of Pharmacology. 81(1). 149–152. 30 indexed citations
12.
Frey, E.A., C. E. Thomas, C. W. GREWE, & John W. Kebabian. (1982). [3H]Spiroperidol Identifies a D-2 Dopamine Receptor Inhibiting Adenylate Cyclase Activity in the Intermediate Lobe of the Rat Pituitary Gland. Endocrinology. 110(6). 1897–1904. 39 indexed citations
13.
Frey, E.A. & Robert J. McIsaac. (1981). A comparison of cyclic guanosine 3':5'-monophosphate and muscarinic excitatory responses in the superior cervical ganglion of the rat.. Journal of Pharmacology and Experimental Therapeutics. 218(1). 115–121. 17 indexed citations
14.
Tsuruta, K., E.A. Frey, C. W. GREWE, et al.. (1981). Evidence that LY-141865 specifically stimulates the D-2 dopamine receptor. Nature. 292(5822). 463–465. 310 indexed citations
15.
Koppenhagen, K., et al.. (1977). Klinisch-experimentelle Ergebnisse mit Heparin-Dihydroergotamin. DMW - Deutsche Medizinische Wochenschrift. 102(39). 1374–1378. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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