B. Obermaier–Kusser

1.2k total citations
28 papers, 925 citations indexed

About

B. Obermaier–Kusser is a scholar working on Molecular Biology, Clinical Biochemistry and Surgery. According to data from OpenAlex, B. Obermaier–Kusser has authored 28 papers receiving a total of 925 indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 9 papers in Clinical Biochemistry and 3 papers in Surgery. Recurrent topics in B. Obermaier–Kusser's work include Mitochondrial Function and Pathology (15 papers), ATP Synthase and ATPases Research (11 papers) and Metabolism and Genetic Disorders (9 papers). B. Obermaier–Kusser is often cited by papers focused on Mitochondrial Function and Pathology (15 papers), ATP Synthase and ATPases Research (11 papers) and Metabolism and Genetic Disorders (9 papers). B. Obermaier–Kusser collaborates with scholars based in Germany, France and Austria. B. Obermaier–Kusser's co-authors include B Ermel, Hans-Ulrich Häring, Monika Kellerer, Klaus-Dieter Gerbitz, D. Pongratz, Patrick Lestienne, E. Seffer, Joanne Mushack, Isabelle Nelson and B. Vogt and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Neurology.

In The Last Decade

B. Obermaier–Kusser

28 papers receiving 895 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
B. Obermaier–Kusser Germany 20 750 268 164 156 155 28 925
Y. Goto Japan 10 859 1.1× 431 1.6× 126 0.8× 60 0.4× 196 1.3× 23 1.2k
Carmen Valcarce Spain 13 279 0.4× 144 0.5× 152 0.9× 82 0.5× 78 0.5× 27 579
Hironori Tamei Japan 9 159 0.2× 315 1.2× 77 0.5× 52 0.3× 218 1.4× 12 654
Emma Footitt United Kingdom 17 387 0.5× 412 1.5× 92 0.6× 46 0.3× 29 0.2× 29 735
Yasuyoshi Yamaji Japan 18 622 0.8× 31 0.1× 140 0.9× 129 0.8× 119 0.8× 29 864
Alecia Willis United States 14 373 0.5× 132 0.5× 77 0.5× 42 0.3× 47 0.3× 17 643
Arthur Hays United States 8 166 0.2× 133 0.5× 145 0.9× 35 0.2× 40 0.3× 11 447
S.J. Heydrick United States 13 516 0.7× 30 0.1× 229 1.4× 125 0.8× 50 0.3× 15 701
Saskia J.G. Hoefs Netherlands 8 707 0.9× 293 1.1× 61 0.4× 37 0.2× 22 0.1× 9 1.1k
Katharina Völker Germany 14 370 0.5× 54 0.2× 127 0.8× 70 0.4× 45 0.3× 32 726

Countries citing papers authored by B. Obermaier–Kusser

Since Specialization
Citations

This map shows the geographic impact of B. Obermaier–Kusser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by B. Obermaier–Kusser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites B. Obermaier–Kusser more than expected).

Fields of papers citing papers by B. Obermaier–Kusser

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by B. Obermaier–Kusser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by B. Obermaier–Kusser. The network helps show where B. Obermaier–Kusser may publish in the future.

Co-authorship network of co-authors of B. Obermaier–Kusser

This figure shows the co-authorship network connecting the top 25 collaborators of B. Obermaier–Kusser. A scholar is included among the top collaborators of B. Obermaier–Kusser based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with B. Obermaier–Kusser. B. Obermaier–Kusser is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wolf, Joachim, et al.. (2012). A new mitochondrial point mutation in the transfer RNALys gene associated with progressive external ophthalmoplegia with impaired respiratory regulation. Journal of the Neurological Sciences. 316(1-2). 108–111. 7 indexed citations
2.
Kiechl, Stefan, et al.. (1999). Different clinical aspects of debrancher deficiency myopathy. Journal of Neurology Neurosurgery & Psychiatry. 67(3). 364–368. 22 indexed citations
3.
Jaksch, Michaela, et al.. (1996). A novel combination of mitochondrial tRNA and ND1 gene mutations in a syndrome with MELAS, cardiomyopathy, and diabetes mellitus. Human Mutation. 7(4). 358–360. 19 indexed citations
4.
Meire, Françoise, et al.. (1995). Neurological disorders in members of families with Leber's hereditary optic neuropathy (LHON) caused by different mitochondrial mutations. Ophthalmic Genetics. 16(3). 119–126. 30 indexed citations
5.
Kellerer, Monika, Giorgio Sesti, E. Seffer, et al.. (1993). Altered pattern of insulin receptor isotypes in skeletal muscle membranes of Type 2 (non-insulin-dependent) diabetic subjects. Diabetologia. 36(7). 628–632. 54 indexed citations
6.
Vogt, B., B. Obermaier–Kusser, E. Seffer, et al.. (1992). Subcellular distribution of GLUT 4 in the skeletal muscle of lean type 2 (non-insulin-dependent) diabetic patients in the basal state. Diabetologia. 35(5). 456–463. 32 indexed citations
7.
Kellerer, Monika, B Ermel, Stefanie Tippmer, et al.. (1992). Distinct .alpha.-subunit structures of human insulin receptor A and B variants determine differences in tyrosine kinase activities. Biochemistry. 31(19). 4588–4596. 68 indexed citations
8.
Müller‐Höcker, Josef, Peter Seibel, Karin Schneiderbanger, et al.. (1992). In situ hybridization of mitochondrial DNA in the heart of a patient with Kearns-Sayre syndrome and dilatative cardiomyopathy. Human Pathology. 23(12). 1431–1437. 35 indexed citations
10.
Degoul, Françoise, Isabelle Nelson, Serge Amselem, et al.. (1991). Different mechanisms inferred from sequences of human mitochondrial DNA deletions in ocular myopathies. Nucleic Acids Research. 19(3). 493–496. 64 indexed citations
11.
Obermaier–Kusser, B., et al.. (1991). Respiratory chain activity in tissues from patients (MELAS) with a point mutation of the mitochondrial genome [tRNALeu(UUR)]. FEBS Letters. 286(1-2). 67–70. 28 indexed citations
12.
M�ller-H�cker, J., et al.. (1991). A specific point mutation in the mitochondrial genome of Caucasians with MELAS. Human Genetics. 88(2). 233–6. 25 indexed citations
13.
Müller, Heidi Kaastrup, Monika Kellerer, B Ermel, et al.. (1991). Prevention by Protein Kinase C Inhibitors of Glucose-Induced Insulin-Receptor Tyrosine Kinase Resistance in Rat Fat Cells. Diabetes. 40(11). 1440–1448. 86 indexed citations
14.
Gerbitz, Klaus-Dieter, B. Obermaier–Kusser, Patrick Lestienne, et al.. (1990). Mutations of the Mitochondrial DNA: The Contribution of DNA Techniques to the Diagnosis of Mitochondrial Encephalomyopathies. Clinical Chemistry and Laboratory Medicine (CCLM). 28(4). 241–50. 9 indexed citations
15.
Obermaier–Kusser, B., et al.. (1990). Different copy numbers of apparently identically deleted mitochondrial DNA in tissues from a patient with Kearns-Sayre syndrome detected by PCR. Biochemical and Biophysical Research Communications. 169(3). 1007–1015. 30 indexed citations
16.
Gerbitz, Klaus-Dieter, B. Obermaier–Kusser, S. Zierz, et al.. (1990). Mitochondrial myopathies: divergences of genetic deletions, biochemical defects and the clinical syndromes. Journal of Neurology. 237(1). 5–10. 44 indexed citations
17.
Kellerer, Monika, E. Seffer, Joanne Mushack, B. Obermaier–Kusser, & Hans‐Ulrich Häring. (1990). TPA inhibits insulin stimulated PIP hydrolysis in fat cell membranes: Evidence for modulation of insulin dependent phospholipase C by proteinkinase C. Biochemical and Biophysical Research Communications. 172(2). 446–454. 15 indexed citations
18.
Nelson, Isabelle, Françoise Degoul, B. Obermaier–Kusser, et al.. (1989). Mapping of heteroplasmic mitochondrial DNA deletions in Kearns-Sayre syndrome. Nucleic Acids Research. 17(20). 8117–8124. 56 indexed citations
19.
Häring, Hans-Ulrich & B. Obermaier–Kusser. (1989). Insulin receptor kinase defects in insulin‐resistant tissues and their role in the pathogenesis of NIDDM. Diabetes/Metabolism Reviews. 5(5). 431–441. 25 indexed citations
20.
Obermaier–Kusser, B., et al.. (1989). [The mechanism of the cellular insulin effect--receptor kinase defect in type II diabetes].. PubMed. 84(3). 144–52. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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