Duk‐Soo Kim

2.1k total citations
96 papers, 1.7k citations indexed

About

Duk‐Soo Kim is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Psychiatry and Mental health. According to data from OpenAlex, Duk‐Soo Kim has authored 96 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 58 papers in Molecular Biology, 42 papers in Cellular and Molecular Neuroscience and 15 papers in Psychiatry and Mental health. Recurrent topics in Duk‐Soo Kim's work include Neuroscience and Neuropharmacology Research (36 papers), Epilepsy research and treatment (12 papers) and Ion channel regulation and function (10 papers). Duk‐Soo Kim is often cited by papers focused on Neuroscience and Neuropharmacology Research (36 papers), Epilepsy research and treatment (12 papers) and Ion channel regulation and function (10 papers). Duk‐Soo Kim collaborates with scholars based in South Korea, Nepal and United States. Duk‐Soo Kim's co-authors include Tae‐Cheon Kang, Dae Won Kim, Soo Young Choi, Jinseu Park, Soo‐Young Choi, Oh‐Shin Kwon, Won Sik Eum, Sung‐Eun Kwak, Ji‐Eun Kim and Ji‐Eun Kim and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Neuron and Journal of Neuroscience.

In The Last Decade

Duk‐Soo Kim

95 papers receiving 1.6k citations

Peers

Duk‐Soo Kim
Mária Baranyi Hungary
Amal Chandra Mondal India
Sara Xapelli Portugal
Esperanza Garcı́a Mexico
Claudio Frank Italy
Xiaolin Deng United States
Sun Hee Lee South Korea
Se Jin Jeon South Korea
Monika Leśkiewicz Poland
Mária Baranyi Hungary
Duk‐Soo Kim
Citations per year, relative to Duk‐Soo Kim Duk‐Soo Kim (= 1×) peers Mária Baranyi

Countries citing papers authored by Duk‐Soo Kim

Since Specialization
Citations

This map shows the geographic impact of Duk‐Soo Kim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Duk‐Soo Kim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Duk‐Soo Kim more than expected).

Fields of papers citing papers by Duk‐Soo Kim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Duk‐Soo Kim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Duk‐Soo Kim. The network helps show where Duk‐Soo Kim may publish in the future.

Co-authorship network of co-authors of Duk‐Soo Kim

This figure shows the co-authorship network connecting the top 25 collaborators of Duk‐Soo Kim. A scholar is included among the top collaborators of Duk‐Soo Kim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Duk‐Soo Kim. Duk‐Soo Kim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Park, Samel, et al.. (2024). AST-120 Protects Cognitive and Emotional Impairment in Chronic Kidney Disease Induced by 5/6 Nephrectomy. Brain Sciences. 14(11). 1043–1043. 2 indexed citations
2.
3.
Kim, Seong-Wook, Dae Young Yoo, Man Ryul Lee, et al.. (2022). Phosphodiesterase-5 Inhibitor Attenuates Anxious Phenotypes and Movement Disorder Induced by Mild Ischemic Stroke in Rats. Journal of Korean Neurosurgical Society. 65(5). 665–679. 6 indexed citations
4.
Kim, Seong-Wook, Minsoo Kim, Jinhee Baek, et al.. (2022). Hemispherically lateralized rhythmic oscillations in the cingulate-amygdala circuit drive affective empathy in mice. Neuron. 111(3). 418–429.e4. 25 indexed citations
5.
Kim, Duk‐Soo, Woosuk Kim, Hyo Young Jung, et al.. (2020). Tat-Cannabinoid Receptor Interacting Protein Reduces Ischemia-Induced Neuronal Damage and Its Possible Relationship with 14-3-3η. Cells. 9(8). 1827–1827. 4 indexed citations
6.
Eum, Won Sik, Min Jea Shin, Chi Hern Lee, et al.. (2018). Neuroprotective effects of Tat-ATOX1 protein against MPP+-induced SH-SY5Y cell deaths and in MPTP-induced mouse model of Parkinson's disease. Biochimie. 156. 158–168. 12 indexed citations
7.
Kim, Dae Won, Min Jea Shin, Hyo Sang Jo, et al.. (2018). PEP-1-PEA15 suppresses inflammatory responses by regulation of MAPK in macrophages and animal models. Immunobiology. 223(11). 709–717. 5 indexed citations
8.
Park, Kyungho, et al.. (2017). Altered functional efficacy of hippocampal interneuron during epileptogenesis following febrile seizures. Brain Research Bulletin. 131. 25–38. 13 indexed citations
9.
Kim, Young Jin, et al.. (2015). The functional relevance of muscarinic acetylcholine receptor and Phospholipase C (PLC) beta expression following status epilepticus in the hippocampus. 57–57. 1 indexed citations
10.
Sohn, Eun Jeong, Min Jea Shin, Dae Won Kim, et al.. (2014). Tat-Fused Recombinant Human SAG Prevents Dopaminergic Neurodegeneration in a MPTP-Induced Parkinson’s Disease Model. Molecules and Cells. 37(3). 226–233. 4 indexed citations
11.
Kim, Ji‐Eun, Duk‐Soo Kim, Hea Jin Ryu, et al.. (2013). The effect of P2X7 receptor activation on nuclear factor‐kappa B phosphorylation induced by status epilepticus in the rat hippocampus. Hippocampus. 23(6). 500–514. 20 indexed citations
12.
Kim, Mi Jin, Dae Won Kim, Hoon Jae Jeong, et al.. (2012). Tat-Frataxin protects dopaminergic neuronal cells against MPTP-induced toxicity in a mouse model of Parkinson's disease. Biochimie. 94(11). 2448–2456. 18 indexed citations
13.
Kim, Ji‐Eun, et al.. (2010). Astroglial loss and edema formation in the rat piriform cortex and hippocampus following pilocarpine‐induced status epilepticus. The Journal of Comparative Neurology. 518(22). 4612–4628. 77 indexed citations
14.
Kim, So Young, Hoon Jae Jeong, Dae Won Kim, et al.. (2010). Transduced PEP-1-FK506BP inhibits the inflammatory response in the Raw 264.7 cell and mouse models. Immunobiology. 216(7). 771–781. 15 indexed citations
15.
Kim, Duk‐Soo, Ji‐Eun Kim, Sung‐Eun Kwak, Moo Ho Won, & Tae‐Cheon Kang. (2007). Seizure activity affects neuroglial Kv1 channel immunoreactivities in the gerbil hippocampus. Brain Research. 1151. 172–187. 12 indexed citations
16.
Kim, Duk‐Soo, Ji‐Eun Kim, Sung‐Eun Kwak, et al.. (2007). Seizure activity selectively reduces 5-HT1A receptor immunoreactivity in CA1 interneurons in the hippocampus of seizure-prone gerbils. Brain Research. 1154. 181–193. 9 indexed citations
17.
Kim, Duk‐Soo, et al.. (2006). Genetic Algorithms and Evolvable Hardware. ICEIC : International Conference on Electronics, Informations and Communications. 367–370. 1 indexed citations
18.
Kang, Tae‐Cheon, Duk‐Soo Kim, Ji‐Eun Kim, et al.. (2006). Altered expression of K+-Cl− cotransporters affects fast paired-pulse inhibition during GABA receptor activation in the gerbil hippocampus. Brain Research. 1072(1). 8–14. 5 indexed citations
19.
Kim, Ji‐Eun, Sung‐Eun Kwak, Duk‐Soo Kim, et al.. (2006). Up-regulation of P/Q-type voltage-gated Ca2+ channel immunoreactivity within parvalbumin positive neurons in the rat hippocampus following status epilepticus. Neuroscience Research. 57(3). 379–386. 3 indexed citations
20.
Kim, Duk‐Soo, Ki‐Yeon Yoo, In Koo Hwang, et al.. (2004). Elevated voltage gated Cl− channel expression enhances fast paired-pulse inhibition in the dentate gyrus of seizure sensitive gerbil. Neuroscience Research. 51(1). 45–53. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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