Douglas J. Franks
- Molecular Biology
- Cardiology and Cardiovascular Medicine top 10%
- Endocrinology, Diabetes and Metabolism top 10%
- Physiology
- Cell Biology
- Co-authors
- Madhu B. Anand‐SrivastavaJacques GenestMarc CantinJolanta GutkowskaErnesto L. SchiffrinJon P. DurkinJ. F. WhitfieldPavel Hamet
- Topics
- Protein Kinase Regulation and GTPase Signaling (5 papers)Virus-based gene therapy research (4 papers)Nitric Oxide and Endothelin Effects (4 papers)
- Partner nations
- CanadaUnited StatesFrance
In The Last Decade
Douglas J. Franks
31 papers receiving 746 citations
Peers
Comparison fields: 5 of 83
- Molecular Biology 473
- Cardiology and Cardiovascular Medicine 211
- Endocrinology, Diabetes and Metabolism 144
- Physiology 112
- Cell Biology 90
Countries citing papers authored by Douglas J. Franks
This map shows the geographic impact of Douglas J. Franks's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Douglas J. Franks with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Douglas J. Franks more than expected).
Fields of papers citing papers by Douglas J. Franks
This network shows the impact of papers produced by Douglas J. Franks. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Douglas J. Franks. The network helps show where Douglas J. Franks may publish in the future.
Co-authorship network of co-authors of Douglas J. Franks
This figure shows the co-authorship network connecting the top 25 collaborators of Douglas J. Franks. A scholar is included among the top collaborators of Douglas J. Franks based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Douglas J. Franks. Douglas J. Franks is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 47 | |
| 2 | 9 | |
| 3 | 19 | |
| 4 | 5 | |
| 5 | 7 | |
| 6 | 5 | |
| 7 | 9 | |
| 8 | 8 | |
| 9 | 29 | |
| 10 | 24 | |
| 11 | 8 | |
| 12 | 7 | |
| 13 | 73 | |
| 14 | 11 | |
| 15 | 22 | |
| 16 | 15 | |
| 17 | 10 | |
| 18 | 6 | |
| 19 | 51 | |
| 20 | 14 |
About Douglas J. Franks
Douglas J. Franks is a scholar working on Physiology, Molecular Biology and Cardiology and Cardiovascular Medicine, having authored 31 papers that have together received 793 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (5 papers), Virus-based gene therapy research (4 papers) and Nitric Oxide and Endothelin Effects (4 papers). The work is most often cited by research in Physiology (50 citations), Cardiology and Cardiovascular Medicine (211 citations) and Endocrinology, Diabetes and Metabolism (144 citations). Douglas J. Franks has collaborated with scholars based in Canada, United States and France. Frequent co-authors include Madhu B. Anand‐Srivastava, Jacques Genest, Marc Cantin, Jolanta Gutkowska, Ernesto L. Schiffrin, Jon P. Durkin, J. F. Whitfield, Pavel Hamet, Daniel Malamud and Leonard P. Kleine. Their work appears in journals such as Journal of Biological Chemistry, Diabetes and Analytical Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.