Donald T. Fullerton
- Clinical Psychology top 5%
- Pathology and Forensic Medicine
- Epidemiology
- Endocrine and Autonomic Systems top 10%
- Psychiatry and Mental health
- Co-authors
- William J. SwiftStephen A. WonderlichCarl J. GettoIan H. CarlsonMarjorie H. KleinBlake A. GosnellRichard F. HarveyTheodore L. Munsat
- Topics
- Stress Responses and Cortisol (4 papers)Regulation of Appetite and Obesity (4 papers)Eating Disorders and Behaviors (4 papers)
- Partner nations
- United States
In The Last Decade
Donald T. Fullerton
20 papers receiving 501 citations
Peers
Comparison fields: 5 of 73
- Clinical Psychology 260
- Pathology and Forensic Medicine 93
- Epidemiology 85
- Endocrine and Autonomic Systems 81
- Psychiatry and Mental health 75
Countries citing papers authored by Donald T. Fullerton
This map shows the geographic impact of Donald T. Fullerton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Donald T. Fullerton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Donald T. Fullerton more than expected).
Fields of papers citing papers by Donald T. Fullerton
This network shows the impact of papers produced by Donald T. Fullerton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Donald T. Fullerton. The network helps show where Donald T. Fullerton may publish in the future.
Co-authorship network of co-authors of Donald T. Fullerton
This figure shows the co-authorship network connecting the top 25 collaborators of Donald T. Fullerton. A scholar is included among the top collaborators of Donald T. Fullerton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Donald T. Fullerton. Donald T. Fullerton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 65 | |
| 2 | 70 | |
| 3 | 7 | |
| 4 | 1 | |
| 5 | 10 | |
| 6 | 8 | |
| 7 | 33 | |
| 8 | 42 | |
| 9 | 38 | |
| 10 | 38 | |
| 11 | 101 | |
| 12 | 7 | |
| 13 | 3 | |
| 14 | 9 | |
| 15 | 59 | |
| 16 | 13 | |
| 17 | 3 | |
| 18 | 5 | |
| 19 | 17 | |
| 20 | 31 |
About Donald T. Fullerton
Donald T. Fullerton is a scholar working on Behavioral Neuroscience, Endocrine and Autonomic Systems and Psychiatry and Mental health, having authored 21 papers that have together received 564 indexed citations. Recurring topics across this work include Stress Responses and Cortisol (4 papers), Regulation of Appetite and Obesity (4 papers) and Eating Disorders and Behaviors (4 papers). The work is most often cited by research in Behavioral Neuroscience (52 citations), Clinical Psychology (260 citations) and Endocrine and Autonomic Systems (81 citations). Donald T. Fullerton has collaborated with scholars based in United States. Frequent co-authors include William J. Swift, Stephen A. Wonderlich, Carl J. Getto, Ian H. Carlson, Marjorie H. Klein, Blake A. Gosnell, Richard F. Harvey, Theodore L. Munsat, Edward J. Kollar and Frederick J. Wenzel. Their work appears in journals such as JAMA, American Journal of Psychiatry and Psychological Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.