DIAN-LEI LIU

500 total citations
8 papers, 431 citations indexed

About

DIAN-LEI LIU is a scholar working on Molecular Biology, Toxicology and Plant Science. According to data from OpenAlex, DIAN-LEI LIU has authored 8 papers receiving a total of 431 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Toxicology and 4 papers in Plant Science. Recurrent topics in DIAN-LEI LIU's work include Bioactive Compounds and Antitumor Agents (4 papers), Cell death mechanisms and regulation (4 papers) and Phytochemistry and biological activity of medicinal plants (4 papers). DIAN-LEI LIU is often cited by papers focused on Bioactive Compounds and Antitumor Agents (4 papers), Cell death mechanisms and regulation (4 papers) and Phytochemistry and biological activity of medicinal plants (4 papers). DIAN-LEI LIU collaborates with scholars based in China. DIAN-LEI LIU's co-authors include Weitian Wei, Zhaohong Wang, Shengzhang Lin, Hongfei Tong, Haibin Liu, Heqi Bu, Sheng‐Zhang Lin, Zhonglin Ni, Hai Huang and Hui Chen and has published in prestigious journals such as PLoS ONE, International Journal of Biological Sciences and International Journal of Oncology.

In The Last Decade

DIAN-LEI LIU

8 papers receiving 428 citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
DIAN-LEI LIU	195	137	85	63	57	8	431
Meng-Liang Lin	257 1.3×	129 0.9×	73 0.9×	43 0.7×	53 0.9×	17	455
Weitian Wei	170 0.9×	105 0.8×	65 0.8×	44 0.7×	43 0.8×	11	389
Ming-Jen Fan	249 1.3×	120 0.9×	73 0.9×	41 0.7×	51 0.9×	14	494
Guang-Wei Chen	215 1.1×	255 1.9×	89 1.0×	33 0.5×	49 0.9×	8	577
Ching‐Lung Liao	296 1.5×	100 0.7×	53 0.6×	44 0.7×	37 0.6×	17	461
Meng‐Liang Lin	319 1.6×	106 0.8×	58 0.7×	83 1.3×	38 0.7×	27	619
Ching-Lung Liao	284 1.5×	83 0.6×	43 0.5×	76 1.2×	58 1.0×	14	506
Tsan Hung Chiu	191 1.0×	119 0.9×	56 0.7×	28 0.4×	41 0.7×	13	361
Huaxin Hou	157 0.8×	72 0.5×	62 0.7×	61 1.0×	38 0.7×	27	329
Gabriela B. Iwanski	294 1.5×	92 0.7×	140 1.6×	86 1.4×	54 0.9×	8	612

Countries citing papers authored by DIAN-LEI LIU

Since Specialization

Citations

This map shows the geographic impact of DIAN-LEI LIU's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by DIAN-LEI LIU with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites DIAN-LEI LIU more than expected).

Fields of papers citing papers by DIAN-LEI LIU

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by DIAN-LEI LIU. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by DIAN-LEI LIU. The network helps show where DIAN-LEI LIU may publish in the future.

Co-authorship network of co-authors of DIAN-LEI LIU

This figure shows the co-authorship network connecting the top 25 collaborators of DIAN-LEI LIU. A scholar is included among the top collaborators of DIAN-LEI LIU based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with DIAN-LEI LIU. DIAN-LEI LIU is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

LIU, DIAN-LEI, et al.. (2020). Oridonin enhances the anti-tumor activity of gemcitabine towards pancreatic cancer by stimulating Bax- and Smac-dependent apoptosis. Translational Cancer Research. 9(7). 4148–4161. 6 indexed citations

LIU, DIAN-LEI, et al.. (2014). Enhancement of the effects of gemcitabine against pancreatic cancer by oridonin via the mitochondrial caspase-dependent signaling pathway. Molecular Medicine Reports. 10(6). 3027–3034. 23 indexed citations

Zhang, Wei, Hui Chen, DIAN-LEI LIU, et al.. (2013). Emodin sensitizes the gemcitabine-resistant cell line Bxpc-3/Gem to gemcitabine via downregulation of NF-κB and its regulated targets. International Journal of Oncology. 42(4). 1189–1196. 38 indexed citations

Wei, Weitian, Shengzhang Lin, DIAN-LEI LIU, & Zhaohong Wang. (2013). The distinct mechanisms of the antitumor activity of emodin in different types of cancer (Review). Oncology Reports. 30(6). 2555–2562. 120 indexed citations

Bu, Heqi, DIAN-LEI LIU, Weitian Wei, et al.. (2013). Oridonin induces apoptosis in SW1990 pancreatic cancer cells via p53- and caspase-dependent induction of p38 MAPK. Oncology Reports. 31(2). 975–982. 54 indexed citations

Wei, Weitian, Hui Chen, Zhaohong Wang, et al.. (2012). Enhanced Antitumor Efficacy of Gemcitabine by Evodiamine on Pancreatic Cancer via Regulating PI3K/Akt Pathway. International Journal of Biological Sciences. 8(1). 1–14. 70 indexed citations

Lin, Sheng‐Zhang, Weitian Wei, Hui Chen, et al.. (2012). Antitumor Activity of Emodin against Pancreatic Cancer Depends on Its Dual Role: Promotion of Apoptosis and Suppression of Angiogenesis. PLoS ONE. 7(8). e42146–e42146. 74 indexed citations

LIU, DIAN-LEI, Heqi Bu, Hui Chen, et al.. (2011). Emodin reverses gemcitabine resistance in pancreatic cancer cells via the mitochondrial apoptosis pathway in vitro. International Journal of Oncology. 40(4). 1049–1057. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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