David Y.H. Choong
- Cancer Research top 5%
- Cancer Genomics and Diagnostics 8
- Reproductive Medicine top 5%
- Oncology top 5%
- Molecular Biology top 5%
- Cancer-related gene regulation 5
- Epigenetics and DNA Methylation 3
- PI3K/AKT/mTOR signaling in cancer 3
- RNA modifications and cancer 3
- Genetics top 5%
- BRCA gene mutations in cancer 6
- Genomic variations and chromosomal abnormalities 4
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- Genetic factors in colorectal cancer 7
- Co-authors
- Ian CampbellWayne A. PhillipsSarah E. RussellKaren G. MontgomeryRichard B. PearsonBriony E. CristianoSimon W. BaxterDiana Eccles
- Partner nations
- AustraliaUnited KingdomUnited States
In The Last Decade
David Y.H. Choong
25 papers receiving 1.9k citations
Hit Papers
Peers
Comparison fields: 5 of 77
- Cancer Research 521
- Reproductive Medicine 224
- Oncology 593
- Molecular Biology 1.3k
- Genetics 191
Countries citing papers authored by David Y.H. Choong
This map shows the geographic impact of David Y.H. Choong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Y.H. Choong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Y.H. Choong more than expected).
Fields of papers citing papers by David Y.H. Choong
This network shows the impact of papers produced by David Y.H. Choong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Y.H. Choong. The network helps show where David Y.H. Choong may publish in the future.
Co-authorship network
The 25 scholars most cited alongside David Y.H. Choong, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2020 | 30 | |
| 2 | 2015 | 67 | |
| 3 | 2015 | 31 | |
| 4 | 2012 | 147 | |
| 5 | 2011 | 53 | |
| 6 | 2009 | 27 | |
| 7 | 2008 | 67 | |
| 8 | 2007 | 122 | |
| 9 | 2007 | 21 | |
| 10 | 2007 | 3 | |
| 11 | 2006 | 13 | |
| 12 | 2005 | 66 | |
| 13 | 2005 | 64 | |
| 14 | 2005 | 4 | |
| 15 | 2004 | 180 | |
| 16 | 2003 | 12 | |
| 17 | 2002 | 61 | |
| 18 | 2002 | 43 | |
| 19 | 2001 | 14 | |
| 20 | 2001 | 70 |
About David Y.H. Choong
David Y.H. Choong is a scholar working on Cancer Research, Pathology and Forensic Medicine and Genetics, having authored 25 papers that have together received 1.9k indexed citations. Recurring topics across this work include Cancer Genomics and Diagnostics (8 papers), Genetic factors in colorectal cancer (7 papers), BRCA gene mutations in cancer (6 papers), Cancer-related gene regulation (5 papers), Genomic variations and chromosomal abnormalities (4 papers), Epigenetics and DNA Methylation (3 papers), PI3K/AKT/mTOR signaling in cancer (3 papers) and RNA modifications and cancer (3 papers). The work is most often cited by research in Cancer Research (521 citations), Reproductive Medicine (224 citations) and Oncology (593 citations). David Y.H. Choong has collaborated with scholars based in Australia, United Kingdom and United States. Frequent co-authors include Ian Campbell, Wayne A. Phillips, Sarah E. Russell, Karen G. Montgomery, Richard B. Pearson, Briony E. Cristiano, Simon W. Baxter, Diana Eccles, Kylie L. Gorringe and Geoffrey J. Lindeman. Their work appears in journals such as Nature Genetics, Cancer Research and Oncogene.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.