David W. Hampton

2.1k total citations
27 papers, 1.3k citations indexed

About

David W. Hampton is a scholar working on Cellular and Molecular Neuroscience, Developmental Neuroscience and Neurology. According to data from OpenAlex, David W. Hampton has authored 27 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Cellular and Molecular Neuroscience, 11 papers in Developmental Neuroscience and 10 papers in Neurology. Recurrent topics in David W. Hampton's work include Neuroinflammation and Neurodegeneration Mechanisms (10 papers), Neurogenesis and neuroplasticity mechanisms (10 papers) and Neuroscience and Neuropharmacology Research (6 papers). David W. Hampton is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (10 papers), Neurogenesis and neuroplasticity mechanisms (10 papers) and Neuroscience and Neuropharmacology Research (6 papers). David W. Hampton collaborates with scholars based in United Kingdom, United States and Canada. David W. Hampton's co-authors include James W. Fawcett, Siddharthan Chandran, Robin J.M. Franklin, Chao Zhao, Chris G. Dulla, Lauren A. Lau, Kate Rhodes, Amaro Taylor‐Weiner, Giuseppina Tesco and Kendall R. Walker and has published in prestigious journals such as Nature Communications, Journal of Neuroscience and Brain.

In The Last Decade

David W. Hampton

27 papers receiving 1.3k citations

Peers

David W. Hampton
Hiroko Nobuta United States
Derron L. Bishop United States
Jana Vukovic Australia
Marcel Dihné Germany
Jelle Praet Belgium
Nadine Wilczak Netherlands
Catherine H. Horner Ireland
Sebastian Thams Sweden
Hisaaki Takahashi Japan
Rüdiger Schweigreiter Austria
Hiroko Nobuta United States
David W. Hampton
Citations per year, relative to David W. Hampton David W. Hampton (= 1×) peers Hiroko Nobuta

Countries citing papers authored by David W. Hampton

Since Specialization
Citations

This map shows the geographic impact of David W. Hampton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David W. Hampton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David W. Hampton more than expected).

Fields of papers citing papers by David W. Hampton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David W. Hampton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David W. Hampton. The network helps show where David W. Hampton may publish in the future.

Co-authorship network of co-authors of David W. Hampton

This figure shows the co-authorship network connecting the top 25 collaborators of David W. Hampton. A scholar is included among the top collaborators of David W. Hampton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David W. Hampton. David W. Hampton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ramesh, Vidya, Karen Burr, Biju Viswanath, et al.. (2025). The Fragile X Messenger Ribonucleoprotein 1 Regulates the Morphology and Maturation of Human and Rat Oligodendrocytes. Glia. 73(6). 1203–1220. 1 indexed citations
2.
Hampton, David W., et al.. (2024). Alterations in network robustness upon simultaneous temperature and pH perturbations. Journal of Neurophysiology. 131(3). 509–515. 3 indexed citations
3.
Hampton, David W., et al.. (2023). Microfluidic device with reconfigurable spatial temporal gradients reveals plastic astrocyte response to stroke and reperfusion. Lab on a Chip. 23(18). 3945–3960. 4 indexed citations
4.
Munro, David A. D., Barry Bradford, Samanta A. Mariani, et al.. (2020). CNS macrophages differentially rely on an intronicCsf1renhancer for their development. Development. 147(23). 50 indexed citations
5.
Hampton, David W., Sandra Amor, David Story, et al.. (2020). HspB5 Activates a Neuroprotective Glial Cell Response in Experimental Tauopathy. Frontiers in Neuroscience. 14. 574–574. 8 indexed citations
6.
Torvell, Megan, David W. Hampton, Peter Connick, et al.. (2019). A single systemic inflammatory insult causes acute motor deficits and accelerates disease progression in a mouse model of human tauopathy. Alzheimer s & Dementia Translational Research & Clinical Interventions. 5(1). 579–591. 12 indexed citations
7.
8.
Hasel, Philip, Owen Dando, Zoeb Jiwaji, et al.. (2017). Neurons and neuronal activity control gene expression in astrocytes to regulate their development and metabolism. Nature Communications. 8(1). 15132–15132. 216 indexed citations
9.
Pirone, Antonella, Lauren A. Lau, David W. Hampton, et al.. (2016). APC conditional knock-out mouse is a model of infantile spasms with elevated neuronal β-catenin levels, neonatal spasms, and chronic seizures. Neurobiology of Disease. 98. 149–157. 33 indexed citations
10.
Walker, Kendall R., Lauren A. Lau, Amaro Taylor‐Weiner, et al.. (2014). Traumatic Brain Injury Increases Cortical Glutamate Network Activity by Compromising GABAergic Control. Cerebral Cortex. 25(8). 2306–2320. 148 indexed citations
11.
Lau, Lauren A., David W. Hampton, Amaro Taylor‐Weiner, et al.. (2014). Gabapentin attenuates hyperexcitability in the freeze-lesion model of developmental cortical malformation. Neurobiology of Disease. 71. 305–316. 29 indexed citations
12.
Armbruster, Moritz, David W. Hampton, Yongjie Yang, & Chris G. Dulla. (2014). Laser-scanning astrocyte mapping reveals increased glutamate-responsive domain size and disrupted maturation of glutamate uptake following neonatal cortical freeze-lesion. Frontiers in Cellular Neuroscience. 8. 277–277. 10 indexed citations
13.
Hampton, David W., Andrea Serio, Gareth Pryce, et al.. (2013). Neurodegeneration progresses despite complete elimination of clinical relapses in a mouse model of multiple sclerosis. Acta Neuropathologica Communications. 1(1). 84–84. 25 indexed citations
14.
Hampton, David W., et al.. (2012). Focal Immune-Mediated White Matter Demyelination Reveals an Age-Associated Increase in Axonal Vulnerability and Decreased Remyelination Efficiency. American Journal Of Pathology. 180(5). 1897–1905. 31 indexed citations
15.
Hampton, David W., JANE ANDERSON, Gareth Pryce, et al.. (2008). An experimental model of secondary progressive multiple sclerosis that shows regional variation in gliosis, remyelination, axonal and neuronal loss. Journal of Neuroimmunology. 201-202. 200–211. 54 indexed citations
16.
Hampton, David W., Richard Asher, Tôru Kondo, et al.. (2007). A potential role for bone morphogenetic protein signalling in glial cell fate determination following adult central nervous system injury in vivo. European Journal of Neuroscience. 26(11). 3024–3035. 83 indexed citations
17.
Hampton, David W., John D. Steeves, James W. Fawcett, & Matt S. Ramer. (2006). Spinally upregulated noggin suppresses axonal and dendritic plasticity following dorsal rhizotomy. Experimental Neurology. 204(1). 366–379. 18 indexed citations
18.
19.
Hampton, David W., Kate Rhodes, Chao Zhao, Robin J.M. Franklin, & James W. Fawcett. (2004). The responses of oligodendrocyte precursor cells, astrocytes and microglia to a cortical stab injury, in the brain. Neuroscience. 127(4). 813–820. 135 indexed citations
20.
Stanfa, Louise C., David W. Hampton, & Anthony H. Dickenson. (2000). Role of Ca2+-permeable non-NMDA glutamate receptors in spinal nociceptive transmission. Neuroreport. 11(14). 3199–3202. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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