David MacTavish

1.6k total citations
31 papers, 1.4k citations indexed

About

David MacTavish is a scholar working on Cellular and Molecular Neuroscience, Physiology and Molecular Biology. According to data from OpenAlex, David MacTavish has authored 31 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Cellular and Molecular Neuroscience, 16 papers in Physiology and 10 papers in Molecular Biology. Recurrent topics in David MacTavish's work include Alzheimer's disease research and treatments (14 papers), Neuropeptides and Animal Physiology (13 papers) and Cholinesterase and Neurodegenerative Diseases (8 papers). David MacTavish is often cited by papers focused on Alzheimer's disease research and treatments (14 papers), Neuropeptides and Animal Physiology (13 papers) and Cholinesterase and Neurodegenerative Diseases (8 papers). David MacTavish collaborates with scholars based in Canada, Japan and United States. David MacTavish's co-authors include Jack H. Jhamandas, Kim H. Harris, Teresa L. Krukoff, Satyabrata Kar, David Westaway, Wenbin Fu, Jing Yang, Balvinder S. Jassar, Jean‐Jacques Bourguignon and Araya Ruangkittisakul and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.

In The Last Decade

David MacTavish

31 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David MacTavish Canada 23 594 573 521 254 183 31 1.4k
I. Bednar Sweden 18 349 0.6× 474 0.8× 456 0.9× 189 0.7× 103 0.6× 36 1.1k
Daniel L. Voisin France 25 779 1.3× 601 1.0× 358 0.7× 349 1.4× 436 2.4× 48 1.6k
Laura M. Frago Spain 24 546 0.9× 255 0.4× 500 1.0× 648 2.6× 80 0.4× 77 1.9k
Rebecca L. Cole United States 11 438 0.7× 746 1.3× 557 1.1× 513 2.0× 182 1.0× 14 1.5k
Jinbin Tian United States 21 327 0.6× 489 0.9× 579 1.1× 153 0.6× 102 0.6× 60 1.7k
M. Markerink–van Ittersum Netherlands 25 549 0.9× 516 0.9× 876 1.7× 185 0.7× 165 0.9× 43 1.8k
Mi-Hyeon Jang South Korea 24 318 0.5× 323 0.6× 417 0.8× 142 0.6× 45 0.2× 55 1.5k
Beata Legutko Poland 25 362 0.6× 742 1.3× 356 0.7× 390 1.5× 80 0.4× 41 1.8k
Akiyoshi Saitoh Japan 26 486 0.8× 951 1.7× 760 1.5× 113 0.4× 113 0.6× 73 1.7k
Zhi‐Ren Rao China 21 365 0.6× 645 1.1× 527 1.0× 223 0.9× 147 0.8× 62 1.4k

Countries citing papers authored by David MacTavish

Since Specialization
Citations

This map shows the geographic impact of David MacTavish's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David MacTavish with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David MacTavish more than expected).

Fields of papers citing papers by David MacTavish

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David MacTavish. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David MacTavish. The network helps show where David MacTavish may publish in the future.

Co-authorship network of co-authors of David MacTavish

This figure shows the co-authorship network connecting the top 25 collaborators of David MacTavish. A scholar is included among the top collaborators of David MacTavish based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David MacTavish. David MacTavish is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kimura, Ryoichi, Wenbin Fu, Rania Soudy, et al.. (2021). Genetic Depletion of Amylin/Calcitonin Receptors Improves Memory and Learning in Transgenic Alzheimer’s Disease Mouse Models. Molecular Neurobiology. 58(10). 5369–5382. 5 indexed citations
2.
Fu, Wenbin, Rania Soudy, David MacTavish, et al.. (2017). Role of microglial amylin receptors in mediating beta amyloid (Aβ)-induced inflammation. Journal of Neuroinflammation. 14(1). 199–199. 42 indexed citations
3.
Kimura, Ryoichi, David MacTavish, Jing Yang, David Westaway, & Jack H. Jhamandas. (2016). Pramlintide Antagonizes Beta Amyloid (Aβ)- and Human Amylin-Induced Depression of Hippocampal Long-Term Potentiation. Molecular Neurobiology. 54(1). 748–754. 18 indexed citations
4.
Fu, Wenbin, Araya Ruangkittisakul, David MacTavish, et al.. (2013). Activity and metabolism-related Ca2+ and mitochondrial dynamics in co-cultured human fetal cortical neurons and astrocytes. Neuroscience. 250. 520–535. 25 indexed citations
5.
Kimura, Ryoichi, David MacTavish, Jing Yang, David Westaway, & Jack H. Jhamandas. (2012). Beta Amyloid-Induced Depression of Hippocampal Long-Term Potentiation Is Mediated through the Amylin Receptor. Journal of Neuroscience. 32(48). 17401–17406. 61 indexed citations
6.
Jhamandas, Jack H. & David MacTavish. (2011). β-Amyloid protein (Aβ) and human amylin regulation of apoptotic genes occurs through the amylin receptor. APOPTOSIS. 17(1). 37–47. 44 indexed citations
7.
Jhamandas, Jack H., et al.. (2011). Actions of β-Amyloid Protein on Human Neurons Are Expressed through the Amylin Receptor. American Journal Of Pathology. 178(1). 140–149. 74 indexed citations
8.
Li, Zong‐Ming, et al.. (2010). Ionic mechanisms of action of prion protein fragment PrP(106–126) in rat basal forebrain neurons. Journal of Neuroscience Research. 88(10). 2217–2227. 11 indexed citations
9.
MacTavish, David, et al.. (2009). Prolactin‐releasing peptide effects in the rat brain are mediated through the Neuropeptide FF receptor. European Journal of Neuroscience. 30(8). 1585–1593. 33 indexed citations
10.
11.
Jhamandas, Jack H., David MacTavish, & Kim H. Harris. (2006). Neuropeptide FF (NPFF) control of magnocellular neurosecretory cells of the rat hypothalamic paraventricular nucleus (PVN). Peptides. 27(5). 973–979. 18 indexed citations
12.
Jhamandas, Jack H., Myung Bok Wie, Kim H. Harris, David MacTavish, & Satyabrata Kar. (2005). Fucoidan inhibits cellular and neurotoxic effects of β‐amyloid (Aβ) in rat cholinergic basal forebrain neurons. European Journal of Neuroscience. 21(10). 2649–2659. 89 indexed citations
13.
MacTavish, David, et al.. (2005). Galanin attenuates β-amyloid (Aβ) toxicity in rat cholinergic basal forebrain neurons. Neurobiology of Disease. 21(2). 413–420. 66 indexed citations
14.
Jhamandas, Jack H. & David MacTavish. (2004). Antagonist of the Amylin Receptor Blocks β-Amyloid Toxicity in Rat Cholinergic Basal Forebrain Neurons. Journal of Neuroscience. 24(24). 5579–5584. 56 indexed citations
15.
Jhamandas, Jack H. & David MacTavish. (2003). Central Administration of Neuropeptide FF Causes Activation of Oxytocin Paraventricular Hypothalamic Neurones That Project to the Brainstem. Journal of Neuroendocrinology. 15(1). 24–32. 35 indexed citations
16.
Harris, Kristin, et al.. (2002). Nociceptin/Orphanin FQ Modulation of Ionic Conductances in Rat Basal Forebrain Neurons. Journal of Pharmacology and Experimental Therapeutics. 303(1). 188–195. 22 indexed citations
17.
Krukoff, Teresa L., David MacTavish, & Jack H. Jhamandas. (1999). Hypertensive rats exhibit heightened expression of corticotropin-releasing factor in activated central neurons in response to restraint stress. Molecular Brain Research. 65(1). 70–79. 39 indexed citations
18.
Krukoff, Teresa L., David MacTavish, & Jack H. Jhamandas. (1997). Activation by hypotension of neurons in the hypothalamic paraventricular nucleus that project to the brainstem. The Journal of Comparative Neurology. 385(2). 285–296. 57 indexed citations
19.
Petrov, Tatjana, Kristin Harris, David MacTavish, Teresa L. Krukoff, & Jack H. Jhamandas. (1995). Hypotension induces fos immunoreactivity in NADPH-diaphorase positive neurons in the paraventricular and supraoptic hypothalamic nuclei of the rat. Neuropharmacology. 34(5). 509–514. 36 indexed citations
20.
Krukoff, Teresa L., David MacTavish, Kim H. Harris, & Jack H. Jhamandas. (1995). Changes in blood volume and pressure induce c-fos expression in brainstem neurons that project to the paraventricular nucleus of the hypothalamus. Molecular Brain Research. 34(1). 99–108. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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