David M. Bader

6.6k total citations · 1 hit paper
94 papers, 5.3k citations indexed

About

David M. Bader is a scholar working on Molecular Biology, Surgery and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, David M. Bader has authored 94 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 76 papers in Molecular Biology, 21 papers in Surgery and 20 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in David M. Bader's work include Congenital heart defects research (41 papers), Cardiomyopathy and Myosin Studies (15 papers) and RNA Research and Splicing (14 papers). David M. Bader is often cited by papers focused on Congenital heart defects research (41 papers), Cardiomyopathy and Myosin Studies (15 papers) and RNA Research and Splicing (14 papers). David M. Bader collaborates with scholars based in United States, United Kingdom and Canada. David M. Bader's co-authors include Donald A. Fischman, Τοmoh Masaki, Katherine E. Yutzey, David E. Reese, Maureen Gannon, Bettina Wilm, Aya Wada, Megan E. Osler, Takashi Mikawa and John O. Oberpriller and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Cell Biology.

In The Last Decade

David M. Bader

93 papers receiving 5.2k citations

Hit Papers

Immunochemical analysis of myosin heavy chain during avia... 1982 2026 1996 2011 1982 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David M. Bader United States 38 3.9k 1.3k 1.0k 734 621 94 5.3k
Antonis K. Hatzopoulos United States 40 3.5k 0.9× 970 0.7× 573 0.5× 509 0.7× 487 0.8× 84 5.1k
Laurenţiu M. Popescu Romania 52 3.8k 1.0× 2.4k 1.8× 839 0.8× 410 0.6× 631 1.0× 95 7.2k
Qing Ma United States 41 5.9k 1.5× 1.7k 1.3× 1.6k 1.6× 899 1.2× 875 1.4× 69 7.2k
Ramón Muñoz‐Chápuli Spain 37 2.9k 0.7× 1.1k 0.9× 694 0.7× 259 0.4× 373 0.6× 108 4.3k
Yunfu Sun China 27 3.3k 0.8× 1.0k 0.8× 672 0.6× 308 0.4× 344 0.6× 48 4.1k
Enrique Brandan Chile 52 4.2k 1.1× 808 0.6× 462 0.4× 859 1.2× 1.8k 2.9× 147 6.3k
Jun Fujita Japan 34 2.4k 0.6× 1.4k 1.1× 324 0.3× 530 0.7× 295 0.5× 133 4.5k
Vikram Prasad United States 29 2.6k 0.7× 580 0.4× 1.0k 1.0× 365 0.5× 430 0.7× 66 4.0k
Didier Montarras France 35 4.9k 1.3× 1.2k 0.9× 472 0.5× 676 0.9× 580 0.9× 82 5.7k
Henrik Semb Sweden 43 4.6k 1.2× 2.1k 1.6× 420 0.4× 1.1k 1.5× 816 1.3× 76 6.9k

Countries citing papers authored by David M. Bader

Since Specialization
Citations

This map shows the geographic impact of David M. Bader's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David M. Bader with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David M. Bader more than expected).

Fields of papers citing papers by David M. Bader

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David M. Bader. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David M. Bader. The network helps show where David M. Bader may publish in the future.

Co-authorship network of co-authors of David M. Bader

This figure shows the co-authorship network connecting the top 25 collaborators of David M. Bader. A scholar is included among the top collaborators of David M. Bader based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David M. Bader. David M. Bader is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Choksi, Yash A., Vishruth K. Reddy, Kshipra Singh, et al.. (2018). BVES is required for maintenance of colonic epithelial integrity in experimental colitis by modifying intestinal permeability. Mucosal Immunology. 11(5). 1363–1374. 15 indexed citations
2.
Pfaltzgraff, Elise R., et al.. (2016). Loss of CENP-F results in distinct microtubule-related defects without chromosomal abnormalities. Molecular Biology of the Cell. 27(13). 1990–1999. 14 indexed citations
3.
Bader, David M., et al.. (2014). Isolation and Physiological Analysis of Mouse Cardiomyocytes. Journal of Visualized Experiments. e51109–e51109. 17 indexed citations
4.
Winters, Nichelle I., et al.. (2014). Resident progenitors, not exogenous migratory cells, generate the majority of visceral mesothelium in organogenesis. Developmental Biology. 391(2). 125–132. 11 indexed citations
5.
Miller, Paul M., Elise R. Pfaltzgraff, Nathan E. Grega‐Larson, et al.. (2013). Bves and NDRG4 regulate directional epicardial cell migration through autocrine extracellular matrix deposition. Molecular Biology of the Cell. 24(22). 3496–3510. 38 indexed citations
6.
Thomason, Rebecca T., et al.. (2011). Application of Small Organic Molecules Reveals Cooperative TGFβ and BMP Regulation of Mesothelial Cell Behaviors. ACS Chemical Biology. 6(9). 952–961. 18 indexed citations
7.
8.
Roberts, Ryan, et al.. (2010). Identification of a novel Bves function: regulation of vesicular transport. The EMBO Journal. 29(3). 532–545. 40 indexed citations
9.
Miller, Paul M., et al.. (2009). Murine CENP-F Regulates Centrosomal Microtubule Nucleation and Interacts with Hook2 at the Centrosome. Molecular Biology of the Cell. 20(22). 4790–4803. 29 indexed citations
10.
Kawaguchi, Michiya, et al.. (2008). Identification of a Novel Intracellular Interaction Domain Essential for Bves Function. PLoS ONE. 3(5). e2261–e2261. 30 indexed citations
11.
Robertson, James B., et al.. (2008). CMF1–Rb interaction promotes myogenesis in avian skeletal myoblasts. Developmental Dynamics. 237(5). 1424–1433. 6 indexed citations
12.
Roland, Joseph T., et al.. (2006). CytLEK1 Is a Regulator of Plasma Membrane Recycling through Its Interaction with SNAP-25. Molecular Biology of the Cell. 17(7). 3176–3186. 18 indexed citations
13.
Robertson, James B., et al.. (2006). Specific deletion of CMF1 nuclear localization domain causes incomplete cell cycle withdrawal and impaired differentiation in avian skeletal myoblasts. Experimental Cell Research. 312(16). 3000–3014. 2 indexed citations
14.
Bader, David M., et al.. (2003). LEK1 Is a Potential Inhibitor of Pocket Protein-mediated Cellular Processes. Journal of Biological Chemistry. 279(1). 664–676. 29 indexed citations
15.
Bader, David M., et al.. (2002). Hole is a novel gene product expressed in the developing heart and brain. Mechanisms of Development. 117(1-2). 347–350. 4 indexed citations
16.
Reese, David E., et al.. (1999). bves:A Novel Gene Expressed during Coronary Blood Vessel Development. Developmental Biology. 209(1). 159–171. 80 indexed citations
17.
Gannon, Maureen, et al.. (1996). Molecular Cloning and Expression of Two Novel Avian Cytochrome P450 1A Enzymes Induced by 2,3,7,8-Tetrachlorodibenzo-p-dioxin. Journal of Biological Chemistry. 271(51). 33054–33059. 119 indexed citations
18.
Yutzey, Katherine E., et al.. (1995). Commitment, Differentiation, and Diversification of Avian Cardiac Progenitor Cells. Annals of the New York Academy of Sciences. 752(1). 1–8. 8 indexed citations
19.
Yutzey, Katherine E., Maureen Gannon, & David M. Bader. (1995). Diversification of Cardiomyogenic Cell Lineages in Vitro. Developmental Biology. 170(2). 531–541. 70 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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