David L. Waning

2.0k total citations
35 papers, 1.1k citations indexed

About

David L. Waning is a scholar working on Molecular Biology, Oncology and Physiology. According to data from OpenAlex, David L. Waning has authored 35 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 13 papers in Oncology and 7 papers in Physiology. Recurrent topics in David L. Waning's work include Bone health and treatments (11 papers), Muscle Physiology and Disorders (10 papers) and Orthopaedic implants and arthroplasty (4 papers). David L. Waning is often cited by papers focused on Bone health and treatments (11 papers), Muscle Physiology and Disorders (10 papers) and Orthopaedic implants and arthroplasty (4 papers). David L. Waning collaborates with scholars based in United States, Spain and Russia. David L. Waning's co-authors include Theresa A. Guise, Robert A. Lamb, Andrea Bonetto, George P. Leser, Anthony P. Schmitt, Brian A. Hain, Daniel Andersson, Rafael Barreto, Yunlong Liu and Hongyu Gao and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Blood.

In The Last Decade

David L. Waning

33 papers receiving 1.1k citations

Peers

David L. Waning

ONCOL

PHYSI

EPIDE

GENET

Suzanne Herren Switzerland

William T. Yewdell United States

Steffen Boettcher Switzerland

Simon Blanchard France

Barrett J. Rollins United States

Shinji Kagami Japan

Véronique Barateau France

Mohamed El Behi France

Jianguang Du United States

Lisa Borghesi United States

Suzanne Herren Switzerland

David L. Waning

363 ×0.7

262 ×0.8

ONCOL

197 ×0.6

PHYSI

127 ×0.5

EPIDE

73 ×0.7

GENET

Citations per year, relative to David L. Waning David L. Waning (= 1×) peers Suzanne Herren

Countries citing papers authored by David L. Waning

Since Specialization

Citations

This map shows the geographic impact of David L. Waning's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David L. Waning with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David L. Waning more than expected).

Fields of papers citing papers by David L. Waning

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David L. Waning. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David L. Waning. The network helps show where David L. Waning may publish in the future.

Co-authorship network of co-authors of David L. Waning

This figure shows the co-authorship network connecting the top 25 collaborators of David L. Waning. A scholar is included among the top collaborators of David L. Waning based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David L. Waning. David L. Waning is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ganguly, Upasana, Abdirashid Warsame, Rachel J. Blosser, et al.. (2025). Evaluating the usage of human placental tissue-derived xenograft in a surgically induced murine fracture model. Bone. 200. 117599–117599.

Hain, Brian A., Scot R. Kimball, & David L. Waning. (2024). Preventing loss of sirt1 lowers mitochondrial oxidative stress and preserves C2C12 myotube diameter in an in vitro model of cancer cachexia. Physiological Reports. 12(13). e16103–e16103. 3 indexed citations

Hain, Brian A. & David L. Waning. (2022). Electroporation of Plasmid DNA into Mouse Skeletal Muscle. Journal of Visualized Experiments. 1 indexed citations

Hain, Brian A. & David L. Waning. (2022). Bone-Muscle Crosstalk: Musculoskeletal Complications of Chemotherapy. Current Osteoporosis Reports. 20(6). 433–441. 8 indexed citations

Hain, Brian A. & David L. Waning. (2022). Electroporation of Plasmid DNA into Mouse Skeletal Muscle. Journal of Visualized Experiments. 3 indexed citations

Fanburg–Smith, Julie C., Kempland C. Walley, Jesse King, et al.. (2021). Animal model detects early pathologic changes of Charcot neuropathic arthropathy. Annals of Diagnostic Pathology. 56. 151878–151878. 3 indexed citations

Hain, Brian A., et al.. (2021). REDD1 deletion attenuates cancer cachexia in mice. Journal of Applied Physiology. 131(6). 1718–1730. 17 indexed citations

Chakraborty, Nabarun, David L. Waning, Aarti Gautam, et al.. (2020). Gene-Metabolite Network Linked to Inhibited Bioenergetics in Association With Spaceflight-Induced Loss of Male Mouse Quadriceps Muscle. Journal of Bone and Mineral Research. 35(10). 2049–2057. 15 indexed citations

Hain, Brian A., et al.. (2019). Chemotherapy‐induced loss of bone and muscle mass in a mouse model of breast cancer bone metastases and cachexia. PubMed. 2(1). 1–12. 38 indexed citations

10.

Regan, Jenna N., Trupti Trivedi, Theresa A. Guise, & David L. Waning. (2017). The Role of TGFβ in Bone-Muscle Crosstalk. Current Osteoporosis Reports. 15(1). 18–23. 25 indexed citations

11.

Regan, Jenna N., Steven Reiken, Andrew R. Marks, et al.. (2017). Osteolytic Breast Cancer Causes Skeletal Muscle Weakness in an Immunocompetent Syngeneic Mouse Model. Frontiers in Endocrinology. 8. 358–358. 21 indexed citations

12.

Barreto, Rafael, David L. Waning, Hongyu Gao, et al.. (2016). Chemotherapy-related cachexia is associated with mitochondrial depletion and the activation of ERK1/2 and p38 MAPKs. Oncotarget. 7(28). 43442–43460. 149 indexed citations

13.

Regan, Jenna N., David L. Waning, & Theresa A. Guise. (2015). Skeletal muscle Ca2+ mishandling: Another effect of bone-to-muscle signaling. Seminars in Cell and Developmental Biology. 49. 24–29. 11 indexed citations

14.

Bonetto, Andrea, Daniel Andersson, & David L. Waning. (2015). Assessment of muscle mass and strength in mice. BoneKEy Reports. 4. 732–732. 100 indexed citations

15.

Stayrook, Keith R., Donna M. Edwards, Hai H. Bui, et al.. (2015). TGFβ-Mediated induction of SphK1 as a potential determinant in human MDA-MB-231 breast cancer cell bone metastasis. BoneKEy Reports. 4. 719–719. 14 indexed citations

16.

Cheng, Ying‐Hua, David L. Waning, Brahmananda R. Chitteti, et al.. (2014). Signaling Pathways Involved in Megakaryocyte‐Mediated Proliferation of Osteoblast Lineage Cells. Journal of Cellular Physiology. 230(3). 578–586. 14 indexed citations

17.

Waning, David L. & Theresa A. Guise. (2014). Molecular Mechanisms of Bone Metastasis and Associated Muscle Weakness. Clinical Cancer Research. 20(12). 3071–3077. 85 indexed citations

18.

Chiechi, Antonella, David L. Waning, Keith R. Stayrook, et al.. (2013). Role of TGF-<i>β</i> in breast cancer bone metastases. Advances in Bioscience and Biotechnology. 4(10). 15–30. 54 indexed citations

19.

Lehman, Jason A., et al.. (2011). Induction of Apoptotic Genes by a p73-Phosphatase and Tensin Homolog (p73-PTEN) Protein Complex in Response to Genotoxic Stress. Journal of Biological Chemistry. 286(42). 36631–36640. 21 indexed citations

20.

Waning, David L., et al.. (2010). c-Abl Phosphorylation of Mdm2 Facilitates Mdm2-Mdmx Complex Formation. Journal of Biological Chemistry. 286(1). 216–222. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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