David Herrmann

4.2k total citations
41 papers, 1.3k citations indexed

About

David Herrmann is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, David Herrmann has authored 41 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 16 papers in Oncology and 8 papers in Cell Biology. Recurrent topics in David Herrmann's work include Cancer Cells and Metastasis (9 papers), Pancreatic and Hepatic Oncology Research (8 papers) and Cellular Mechanics and Interactions (5 papers). David Herrmann is often cited by papers focused on Cancer Cells and Metastasis (9 papers), Pancreatic and Hepatic Oncology Research (8 papers) and Cellular Mechanics and Interactions (5 papers). David Herrmann collaborates with scholars based in Australia, United Kingdom and United States. David Herrmann's co-authors include Paul Timpson, Jennifer P. Morton, Claire Vennin, Sean Warren, James R. W. Conway, Brooke Pereira, Vincent Hyenne, Jacky G. Goetz, Gautier Follain and Naël Osmani and has published in prestigious journals such as Nature Communications, Journal of Clinical Oncology and Nature reviews. Cancer.

In The Last Decade

David Herrmann

38 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David Herrmann Australia 19 671 542 281 272 180 41 1.3k
V. М. Perelmuter Russia 20 600 0.9× 511 0.9× 155 0.6× 390 1.4× 160 0.9× 108 1.3k
Ai Sato Japan 16 592 0.9× 630 1.2× 183 0.7× 352 1.3× 275 1.5× 45 1.4k
João Incio United States 9 434 0.6× 458 0.8× 248 0.9× 179 0.7× 311 1.7× 13 1.3k
Felix Zeppernick Germany 17 691 1.0× 609 1.1× 119 0.4× 384 1.4× 168 0.9× 40 1.6k
Rafał Sądej Poland 18 339 0.5× 560 1.0× 124 0.4× 256 0.9× 141 0.8× 51 1.2k
Ethel R. Pereira United States 13 526 0.8× 398 0.7× 137 0.5× 236 0.9× 282 1.6× 17 1.1k
Crina Tiron Romania 12 540 0.8× 552 1.0× 119 0.4× 186 0.7× 407 2.3× 27 1.3k
María Virtudes Céspedes Spain 7 1.0k 1.5× 872 1.6× 149 0.5× 410 1.5× 240 1.3× 9 1.7k
José L. Orgaz Spain 20 544 0.8× 938 1.7× 129 0.5× 320 1.2× 180 1.0× 26 1.6k
Mingfu Wu China 18 254 0.4× 495 0.9× 207 0.7× 190 0.7× 209 1.2× 70 1.3k

Countries citing papers authored by David Herrmann

Since Specialization
Citations

This map shows the geographic impact of David Herrmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Herrmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Herrmann more than expected).

Fields of papers citing papers by David Herrmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Herrmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Herrmann. The network helps show where David Herrmann may publish in the future.

Co-authorship network of co-authors of David Herrmann

This figure shows the co-authorship network connecting the top 25 collaborators of David Herrmann. A scholar is included among the top collaborators of David Herrmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Herrmann. David Herrmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sue, Nancy, Ebru Boslem, Kwan Yi Chu, et al.. (2025). ER stress disrupts insulin release in murine models of type 2 diabetes by impairing retromer action and constitutive secretion. Cell Reports. 44(5). 115691–115691. 1 indexed citations
3.
Reed, Daniel A., David Herrmann, Frank J. Lovicu, et al.. (2024). Fibroblast growth factor-induced lens fiber cell elongation is driven by the stepwise activity of Rho and Rac. Development. 151(3). 4 indexed citations
4.
Song, Ji‐Ying, Mark C. de Gooijer, Hendrik A. Messal, et al.. (2023). BCRP drives intrinsic chemoresistance in chemotherapy-naïve breast cancer brain metastasis. Science Advances. 9(42). eabp9530–eabp9530. 8 indexed citations
5.
Scheele, Colinda L. G. J., David Herrmann, Erika Yamashita, et al.. (2022). Multiphoton intravital microscopy of rodents. Nature Reviews Methods Primers. 2(1). 33 indexed citations
6.
Wang, Kai, Yifan Zhan, Nhi Huynh, et al.. (2019). Inhibition of PAK1 suppresses pancreatic cancer by stimulation of anti-tumour immunity through down-regulation of PD-L1. Cancer Letters. 472. 8–18. 36 indexed citations
7.
Pereira, Brooke, Claire Vennin, Michael Papanicolaou, et al.. (2019). CAF Subpopulations: A New Reservoir of Stromal Targets in Pancreatic Cancer. Trends in cancer. 5(11). 724–741. 222 indexed citations
8.
Conway, James R. W., et al.. (2018). Combating pancreatic cancer with PI3K pathway inhibitors in the era of personalised medicine. Gut. 68(4). 742–758. 65 indexed citations
9.
Pinho, Andreia V., Lorraine A. Chantrill, David Herrmann, et al.. (2018). ROBO2 is a stroma suppressor gene in the pancreas and acts via TGF-β signalling. Nature Communications. 9(1). 5083–5083. 40 indexed citations
10.
Warren, Sean, Max Nobis, Astrid Magenau, et al.. (2018). Removing physiological motion from intravital and clinical functional imaging data. eLife. 7. 31 indexed citations
11.
Kawai, Mariko, et al.. (2018). Fgfr1 conditional-knockout in neural crest cells induces heterotopic chondrogenesis and osteogenesis in mouse frontal bones. Medical Molecular Morphology. 52(3). 156–163. 8 indexed citations
12.
Benthani, Fahad, David Herrmann, Phuong Tran, et al.. (2017). ‘MCC’ protein interacts with E-cadherin and β-catenin strengthening cell–cell adhesion of HCT116 colon cancer cells. Oncogene. 37(5). 663–672. 22 indexed citations
13.
Vennin, Claire, James R. W. Conway, Kara L. Vine, et al.. (2017). SerpinB2 regulates stromal remodelling and local invasion in pancreatic cancer. Oncogene. 36(30). 4288–4298. 77 indexed citations
14.
Herrmann, David, et al.. (2017). Heterogeneity of Systematic Reviews in Oncology. Baylor University Medical Center Proceedings. 30(2). 163–166. 4 indexed citations
15.
16.
Young, Adelaide I.J., Andrew M. K. Law, Lesley Castillo, et al.. (2016). MCL-1 inhibition provides a new way to suppress breast cancer metastasis and increase sensitivity to dasatinib. Breast Cancer Research. 18(1). 125–125. 64 indexed citations
17.
Nobis, Max, Ewan J. McGhee, David Herrmann, et al.. (2014). Monitoring the dynamics of Src activity in response to anti-invasive dasatinib treatment at a subcellular level using dual intravital imaging. Cell Adhesion & Migration. 8(5). 478–486. 5 indexed citations
18.
Herrmann, David, et al.. (2009). Expression and regulation of ANTXR1 in the chick embryo. Developmental Dynamics. 239(2). 680–687. 8 indexed citations
19.
Herrmann, David, et al.. (2009). 09-P054 Analysis of facial and palatal defects in Fgfr1 mutant mice. Mechanisms of Development. 126. S166–S166. 1 indexed citations
20.
Jung, Beth F., David Herrmann, Jennifer J. Griggs, Anne Louise Oaklander, & Robert H. Dworkin. (2005). Neuropathic pain associated with non-surgical treatment of breast cancer. Pain. 118(1). 10–14. 70 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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