David Crettaz
- Molecular Biology
- Physiology top 5%
- Hematology top 2%
- Spectroscopy top 5%
- Pulmonary and Respiratory Medicine top 10%
- Co-authors
- Jean‐Daniel TissotNiels LionOlivier RubinMichel PrudentLynne ThadikkaranPhilippe SchneiderGiorgia CanelliniJulien Delobel
- Topics
- Erythrocyte Function and Pathophysiology (20 papers)Blood transfusion and management (15 papers)Blood properties and coagulation (12 papers)
- Cited by
- BiochemistryHematologySpectroscopy
- Journals
- International Journal of Molecular SciencesClinica Chimica ActaJournal of Thrombosis and Haemostasis
- Partner nations
- SwitzerlandFranceUnited States
In The Last Decade
David Crettaz
47 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 112
- Molecular Biology 552
- Physiology 453
- Hematology 375
- Spectroscopy 316
- Pulmonary and Respiratory Medicine 310
Countries citing papers authored by David Crettaz
This map shows the geographic impact of David Crettaz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Crettaz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Crettaz more than expected).
Fields of papers citing papers by David Crettaz
This network shows the impact of papers produced by David Crettaz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Crettaz. The network helps show where David Crettaz may publish in the future.
Co-authorship network of co-authors of David Crettaz
This figure shows the co-authorship network connecting the top 25 collaborators of David Crettaz. A scholar is included among the top collaborators of David Crettaz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Crettaz. David Crettaz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 2 | |
| 3 | 3 | |
| 4 | 13 | |
| 5 | 7 | |
| 6 | 11 | |
| 7 | 11 | |
| 8 | 31 | |
| 9 | 59 | |
| 10 | 54 | |
| 11 | 144 | |
| 12 | 44 | |
| 13 | 88 | |
| 14 | 3 | |
| 15 | 23 | |
| 16 | 24 | |
| 17 | 142 | |
| 18 | 39 | |
| 19 | 9 | |
| 20 | 31 |
About David Crettaz
David Crettaz is a scholar working on Biochemistry, Hematology and Physiology, having authored 48 papers that have together received 1.5k indexed citations. Recurring topics across this work include Erythrocyte Function and Pathophysiology (20 papers), Blood transfusion and management (15 papers) and Blood properties and coagulation (12 papers). The work is most often cited by research in Biochemistry (309 citations), Hematology (375 citations) and Spectroscopy (316 citations). David Crettaz has collaborated with scholars based in Switzerland, France and United States. Frequent co-authors include Jean‐Daniel Tissot, Niels Lion, Olivier Rubin, Michel Prudent, Lynne Thadikkaran, Philippe Schneider, Giorgia Canellini, Julien Delobel, Joël S. Rossier and Denis Gallot. Their work appears in journals such as International Journal of Molecular Sciences, Clinica Chimica Acta and Journal of Thrombosis and Haemostasis.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.