David Bode

1.1k total citations
23 papers, 703 citations indexed

About

David Bode is a scholar working on Cardiology and Cardiovascular Medicine, Physiology and Molecular Biology. According to data from OpenAlex, David Bode has authored 23 papers receiving a total of 703 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Cardiology and Cardiovascular Medicine, 8 papers in Physiology and 6 papers in Molecular Biology. Recurrent topics in David Bode's work include Cardiovascular Function and Risk Factors (10 papers), Cardiac electrophysiology and arrhythmias (7 papers) and Alzheimer's disease research and treatments (4 papers). David Bode is often cited by papers focused on Cardiovascular Function and Risk Factors (10 papers), Cardiac electrophysiology and arrhythmias (7 papers) and Alzheimer's disease research and treatments (4 papers). David Bode collaborates with scholars based in Germany, United Kingdom and United States. David Bode's co-authors include John H. Viles, Mark D. Baker, Jon Nield, Matteo Palma, Mark Freeley, Gabriele G. Schiattarella, Felix Hohendanner, Frank R. Heinzel, Burkert Pieske and Uwe Primeßnig and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Molecular Biology and Scientific Reports.

In The Last Decade

David Bode

21 papers receiving 694 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David Bode Germany 13 322 316 199 80 78 23 703
James Wakefield United States 12 227 0.7× 236 0.7× 44 0.2× 41 0.5× 93 1.2× 26 626
Rita Papp Hungary 15 337 1.0× 157 0.5× 235 1.2× 21 0.3× 26 0.3× 21 691
Yan Bai China 20 572 1.8× 216 0.7× 132 0.7× 34 0.4× 12 0.2× 33 956
Baiping Cui China 13 159 0.5× 88 0.3× 137 0.7× 45 0.6× 11 0.1× 19 487
Rishi Trivedi United States 12 632 2.0× 283 0.9× 313 1.6× 53 0.7× 11 0.1× 19 1.1k
Harrie C. M. Boonen Denmark 13 249 0.8× 153 0.5× 94 0.5× 18 0.2× 11 0.1× 27 549
Kazuki Noda Japan 17 238 0.7× 159 0.5× 197 1.0× 93 1.2× 14 0.2× 54 662
Suzan K. Hanna United States 15 276 0.9× 94 0.3× 34 0.2× 69 0.9× 64 0.8× 25 652

Countries citing papers authored by David Bode

Since Specialization
Citations

This map shows the geographic impact of David Bode's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Bode with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Bode more than expected).

Fields of papers citing papers by David Bode

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Bode. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Bode. The network helps show where David Bode may publish in the future.

Co-authorship network of co-authors of David Bode

This figure shows the co-authorship network connecting the top 25 collaborators of David Bode. A scholar is included among the top collaborators of David Bode based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Bode. David Bode is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fu, Shu Man, David Bode, Felix Hohendanner, et al.. (2024). Impairment of the adrenergic reserve associated with exercise intolerance in a murine model of heart failure with preserved ejection fraction. Acta Physiologica. 240(4). e14124–e14124. 4 indexed citations
2.
Bode, David, Julius Ryan D. Pronto, Gabriele G. Schiattarella, & Niels Voigt. (2024). Metabolic remodelling in atrial fibrillation: manifestations, mechanisms and clinical implications. Nature Reviews Cardiology. 21(10). 682–700. 29 indexed citations
3.
Wang, Ruirui, Federico Capone, Limin Luo, et al.. (2024). Ketone-based metabolism and signalling in heart failure with preserved ejection fraction (HFpEF). Cardiovascular Research. 120(Supplement_1).
4.
Capone, Federico, et al.. (2022). Cardiac metabolism in HFpEF: from fuel to signalling. Cardiovascular Research. 118(18). 3556–3575. 63 indexed citations
5.
Bode, David, Christian U. Oeing, Alessio Alogna, et al.. (2021). Implications of SGLT Inhibition on Redox Signalling in Atrial Fibrillation. International Journal of Molecular Sciences. 22(11). 5937–5937. 9 indexed citations
6.
Primeßnig, Uwe, David Bode, Paulina Wakula, et al.. (2021). Right-Ventricular Dysfunction in HFpEF is Linked to Altered Cardiomyocyte Ca2+ Homeostasis and Myofilament Sensitivity. ESC Heart Failure. 8(4). 3130–3144. 13 indexed citations
7.
Bode, David, Paulina Wakula, Uwe Primeßnig, et al.. (2021). Dual SGLT-1 and SGLT-2 inhibition improves left atrial dysfunction in HFpEF. Cardiovascular Diabetology. 20(1). 7–7. 89 indexed citations
8.
Bode, David, Natale Rolim, Paulina Wakula, et al.. (2021). Effects of Different Exercise Modalities on Cardiac Dysfunction in Heart Failure with Preserved Ejection Fraction. ESC Heart Failure. 8(3). 1806–1818. 7 indexed citations
9.
Parwani, Abdul Shokor, Felix Hohendanner, David Bode, et al.. (2020). The force stability of tissue contact and lesion size index during radiofrequency ablation: An ex‐vivo study. Pacing and Clinical Electrophysiology. 43(3). 327–331. 11 indexed citations
10.
Bode, David, Yan Wen, Uwe Primeßnig, et al.. (2020). Oxidative Stress and Inflammatory Modulation of Ca2+ Handling in Metabolic HFpEF-Related Left Atrial Cardiomyopathy. Antioxidants. 9(9). 860–860. 24 indexed citations
11.
Bode, David, Mark Freeley, Jon Nield, Matteo Palma, & John H. Viles. (2019). Amyloid-β oligomers have a profound detergent-like effect on lipid membrane bilayers, imaged by atomic force and electron microscopy. Journal of Biological Chemistry. 294(19). 7566–7572. 121 indexed citations
12.
Bode, David, et al.. (2018). Copper Redox Cycling Inhibits Aβ Fibre Formation and Promotes Fibre Fragmentation, while Generating a Dityrosine Aβ Dimer. Scientific Reports. 8(1). 16190–16190. 46 indexed citations
13.
Bode, David, et al.. (2018). Serum Albumin's Protective Inhibition of Amyloid-β Fiber Formation Is Suppressed by Cholesterol, Fatty Acids and Warfarin. Journal of Molecular Biology. 430(7). 919–934. 34 indexed citations
14.
Bode, David, et al.. (2018). Isolation of Atrial Cardiomyocytes from a Rat Model of Metabolic Syndrome-related Heart Failure with Preserved Ejection Fraction. Journal of Visualized Experiments. 5 indexed citations
15.
Hohendanner, Felix, David Bode, Uwe Primeßnig, et al.. (2017). Cellular mechanisms of metabolic syndrome-related atrial decompensation in a rat model of HFpEF. Journal of Molecular and Cellular Cardiology. 115. 10–19. 25 indexed citations
16.
Bode, David, Mark D. Baker, & John H. Viles. (2016). Ion Channel Formation by Amyloid-β42 Oligomers but Not Amyloid-β40 in Cellular Membranes. Journal of Biological Chemistry. 292(4). 1404–1413. 170 indexed citations
17.
Chiang, Shih‐Chieh, Bernadett Kalmár, David Bode, et al.. (2014). Expression of a pathogenic mutation of SOD1 sensitizes aprataxin-deficient cells and mice to oxidative stress and triggers hallmarks of premature ageing. Human Molecular Genetics. 24(3). 828–840. 14 indexed citations
18.
Bode, David, et al.. (2010). Elephantiasis Nostras Verrucosa (ENV): A Complication of Congestive Heart Failure and Obesity. The Journal of the American Board of Family Medicine. 23(3). 413–417. 19 indexed citations
19.
Seehusen, Dean A., et al.. (2010). Primary care of adult survivors of childhood cancer.. PubMed. 81(10). 1250–5. 16 indexed citations
20.
Peiper, U., et al.. (1987). Barium ions reduce contraction kinetics in rat tracheal smooth muscle. Pflügers Archiv - European Journal of Physiology. 408(4). 373–378. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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