David B. Wang

825 total citations
16 papers, 673 citations indexed

About

David B. Wang is a scholar working on Molecular Biology, Physiology and Genetics. According to data from OpenAlex, David B. Wang has authored 16 papers receiving a total of 673 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 5 papers in Physiology and 4 papers in Genetics. Recurrent topics in David B. Wang's work include Alzheimer's disease research and treatments (5 papers), Neurogenetic and Muscular Disorders Research (4 papers) and Amyotrophic Lateral Sclerosis Research (4 papers). David B. Wang is often cited by papers focused on Alzheimer's disease research and treatments (5 papers), Neurogenetic and Muscular Disorders Research (4 papers) and Amyotrophic Lateral Sclerosis Research (4 papers). David B. Wang collaborates with scholars based in United States. David B. Wang's co-authors include Ronald L. Klein, Robert D. Dayton, C. Kinoshita, Yoshito Kinoshita, Richard S. Morrison, Bryce L. Sopher, Gwenn A. Garden, Omar Skalli, Michael Hutton and Dennis W. Dickson and has published in prestigious journals such as Journal of Neuroscience, Brain and Journal of Neurochemistry.

In The Last Decade

David B. Wang

16 papers receiving 666 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David B. Wang United States 14 369 172 145 129 126 16 673
Sara Sepe Italy 15 428 1.2× 178 1.0× 222 1.5× 145 1.1× 39 0.3× 22 807
Hans-Dieter Mennel Germany 15 268 0.7× 125 0.7× 90 0.6× 209 1.6× 68 0.5× 26 711
Kasey L. Jackson United States 10 291 0.8× 149 0.9× 81 0.6× 70 0.5× 102 0.8× 19 494
Breanna Cooper United States 12 266 0.7× 177 1.0× 297 2.0× 92 0.7× 73 0.6× 16 872
Christopher J. Holler United States 11 307 0.8× 253 1.5× 356 2.5× 65 0.5× 78 0.6× 16 687
Saba Tadesse United States 20 1.3k 3.5× 125 0.7× 70 0.5× 147 1.1× 60 0.5× 28 1.5k
Margaret Wong United States 14 797 2.2× 563 3.3× 140 1.0× 139 1.1× 378 3.0× 27 1.3k
Gloria M. Palomo Spain 8 300 0.8× 205 1.2× 93 0.6× 107 0.8× 60 0.5× 11 537
M. E. Percy Canada 12 287 0.8× 177 1.0× 221 1.5× 76 0.6× 117 0.9× 30 658
Elena Panzeri Italy 15 225 0.6× 47 0.3× 104 0.7× 140 1.1× 58 0.5× 33 587

Countries citing papers authored by David B. Wang

Since Specialization
Citations

This map shows the geographic impact of David B. Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David B. Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David B. Wang more than expected).

Fields of papers citing papers by David B. Wang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David B. Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David B. Wang. The network helps show where David B. Wang may publish in the future.

Co-authorship network of co-authors of David B. Wang

This figure shows the co-authorship network connecting the top 25 collaborators of David B. Wang. A scholar is included among the top collaborators of David B. Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David B. Wang. David B. Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Wang, David B., C. Kinoshita, Yoshito Kinoshita, et al.. (2018). Neuronal susceptibility to beta‐amyloid toxicity and ischemic injury involves histone deacetylase‐2 regulation of endophilin‐B1. Brain Pathology. 29(2). 164–175. 22 indexed citations
2.
Wang, David B., Yoshito Kinoshita, C. Kinoshita, et al.. (2015). Loss of endophilin-B1 exacerbates Alzheimer’s disease pathology. Brain. 138(7). 2005–2019. 28 indexed citations
3.
Su, Wei, Bryce L. Sopher, J. M. Gillespie, et al.. (2015). Recombinant adeno‐associated viral (rAAV) vectors mediate efficient gene transduction in cultured neonatal and adult microglia. Journal of Neurochemistry. 136(S1). 49–62. 23 indexed citations
4.
Wang, David B., C. Kinoshita, Yoshito Kinoshita, & Richard S. Morrison. (2014). p53 and mitochondrial function in neurons. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1842(8). 1186–1197. 145 indexed citations
5.
Wang, David B., Takuma Uo, C. Kinoshita, et al.. (2014). Bax Interacting Factor-1 Promotes Survival and Mitochondrial Elongation in Neurons. Journal of Neuroscience. 34(7). 2674–2683. 36 indexed citations
6.
Wang, David B., Gwenn A. Garden, C. Kinoshita, et al.. (2013). Declines in Drp1 and Parkin Expression Underlie DNA Damage-Induced Changes in Mitochondrial Length and Neuronal Death. Journal of Neuroscience. 33(4). 1357–1365. 49 indexed citations
7.
Dayton, Robert D., Michael A. Gitcho, Elysse A. Orchard, et al.. (2013). Selective Forelimb Impairment in Rats Expressing a Pathological TDP-43 25 kDa C-terminal Fragment to Mimic Amyotrophic Lateral Sclerosis. Molecular Therapy. 21(7). 1324–1334. 36 indexed citations
8.
Dayton, Robert D., David B. Wang, & Ronald L. Klein. (2012). The advent of AAV9 expands applications for brain and spinal cord gene delivery. Expert Opinion on Biological Therapy. 12(6). 757–766. 111 indexed citations
9.
Robbins, Delira, Magdalena L. Circu, Tak Yee Aw, et al.. (2012). Isocitrate dehydrogenase 1 is downregulated during early skin tumorigenesis which can be inhibited by overexpression of manganese superoxide dismutase. Cancer Science. 103(8). 1429–1433. 15 indexed citations
10.
Wang, David B., Michael A. Gitcho, Brian C. Kraemer, & Ronald L. Klein. (2011). Genetic strategies to study TDP-43 in rodents and to develop preclinical therapeutics for amyotrophic lateral sclerosis. European Journal of Neuroscience. 34(8). 1179–1188. 15 indexed citations
11.
Dayton, Robert D., David B. Wang, Lisa M. Schrott, et al.. (2011). Frontotemporal lobar degeneration-related proteins induce only subtle memory-related deficits when bilaterally overexpressed in the dorsal hippocampus. Experimental Neurology. 233(2). 807–814. 12 indexed citations
12.
Wang, David B., Robert D. Dayton, Richard M. Zweig, & Ronald L. Klein. (2010). Transcriptome analysis of a tau overexpression model in rats implicates an early pro-inflammatory response. Experimental Neurology. 224(1). 197–206. 20 indexed citations
13.
Wang, David B., Robert D. Dayton, Lisa M. Schrott, et al.. (2010). Expansive Gene Transfer in the Rat CNS Rapidly Produces Amyotrophic Lateral Sclerosis Relevant Sequelae When TDP-43 is Overexpressed. Molecular Therapy. 18(12). 2064–2074. 52 indexed citations
14.
Wang, David B., Robert D. Dayton, Omar Skalli, et al.. (2009). Mimicking Aspects of Frontotemporal Lobar Degeneration and Lou Gehrig's Disease in Rats via TDP-43 Overexpression. Molecular Therapy. 17(4). 607–613. 70 indexed citations
15.
Klein, Ronald L., et al.. (2009). Pronounced microgliosis and neurodegeneration in aged rats after tau gene transfer. Neurobiology of Aging. 31(12). 2091–2102. 33 indexed citations
16.
Klein, Ronald L., David B. Wang, & Michael A. King. (2009). Versatile Somatic Gene Transfer for Modeling Neurodegenerative Diseases. Neurotoxicity Research. 16(3). 329–342. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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