David A. Norris

14.6k total citations · 3 hit papers
220 papers, 10.9k citations indexed

About

David A. Norris is a scholar working on Immunology, Molecular Biology and Dermatology. According to data from OpenAlex, David A. Norris has authored 220 papers receiving a total of 10.9k indexed citations (citations by other indexed papers that have themselves been cited), including 80 papers in Immunology, 72 papers in Molecular Biology and 61 papers in Dermatology. Recurrent topics in David A. Norris's work include melanin and skin pigmentation (38 papers), Cell Adhesion Molecules Research (28 papers) and Immunotherapy and Immune Responses (24 papers). David A. Norris is often cited by papers focused on melanin and skin pigmentation (38 papers), Cell Adhesion Molecules Research (28 papers) and Immunotherapy and Immune Responses (24 papers). David A. Norris collaborates with scholars based in United States, United Kingdom and Australia. David A. Norris's co-authors include Yiqun G. Shellman, Nathaniel B. Goldstein, Joseph G. Morelli, William L. Weston, Mayumi Fujita, Joseph Yohn, Martin B. Lyons, J. Clark Huff, Donald Y.M. Leung and Madeleine Duvic and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

David A. Norris

216 papers receiving 10.5k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A simple technique for quantifying apoptosis in 96-well plates

2005 726 citations Nathaniel B. Goldstein, David A. Norris et al. profile →
Alopecia areata investigational assessment guidelines–Part II

2004 584 citations Vera H. Price, Madeleine Duvic et al. Journal of the American Academy of Dermatology profile →
Genome-wide association study in alopecia areata implicates both innate and adaptive immunity

2010 538 citations Madeleine Duvic, Maria Hordinsky et al. profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
David A. Norris	3.7k	3.2k	3.0k	2.2k	1.4k	220	10.9k
Louis Dubertret	3.5k 1.0×	2.2k 0.7×	4.1k 1.4×	1.2k 0.6×	443 0.3×	368	12.3k
Y. Tokura	3.8k 1.0×	1.4k 0.4×	4.7k 1.5×	700 0.3×	697 0.5×	423	10.3k
Lloyd E. King	1.2k 0.3×	3.7k 1.1×	2.8k 0.9×	1.9k 0.9×	2.4k 1.7×	208	10.2k
Brian J. Nickoloff	9.1k 2.5×	5.3k 1.7×	5.6k 1.8×	1.8k 0.8×	547 0.4×	239	19.0k
Constantin E. Orfanos	1.3k 0.4×	2.4k 0.7×	2.6k 0.8×	1.4k 0.6×	512 0.4×	269	7.7k
Norbert E. Fusenig	1.8k 0.5×	6.9k 2.1×	1.8k 0.6×	3.3k 1.5×	831 0.6×	175	15.9k
Gerd Plewig	1.8k 0.5×	1.6k 0.5×	6.5k 2.1×	1.4k 0.6×	265 0.2×	404	11.2k
Harald Gollnick	1.6k 0.4×	2.1k 0.6×	5.7k 1.9×	1.4k 0.6×	218 0.2×	291	9.8k
Seth J. Orlow	941 0.3×	2.9k 0.9×	2.3k 0.7×	3.3k 1.5×	202 0.1×	210	8.4k
Madeleine Duvic	7.9k 2.2×	6.7k 2.1×	14.4k 4.7×	1.7k 0.8×	2.1k 1.5×	627	26.2k

Countries citing papers authored by David A. Norris

Since Specialization

Citations

This map shows the geographic impact of David A. Norris's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David A. Norris with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David A. Norris more than expected).

Fields of papers citing papers by David A. Norris

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David A. Norris. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David A. Norris. The network helps show where David A. Norris may publish in the future.

Co-authorship network of co-authors of David A. Norris

This figure shows the co-authorship network connecting the top 25 collaborators of David A. Norris. A scholar is included among the top collaborators of David A. Norris based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David A. Norris. David A. Norris is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Mukherjee, Nabanita, Theresa Pacheco, Morkos A. Henen, et al.. (2024). SASH1 S519N Variant Links Skin Hyperpigmentation and Premature Hair Graying to Dysfunction of Melanocyte Lineage. Journal of Investigative Dermatology. 145(1). 144–154.e3. 2 indexed citations

Goldstein, Nathaniel B., Dennis R. Roop, Yonghua Zhuang, et al.. (2024). Phosphodiesterase-4 Inhibitors Increase Pigment Cell Proliferation and Melanization in Cultured Melanocytes and within a 3-Dimensional Skin Equivalent Model. Journal of Investigative Dermatology. 145(4). 883–896. 1 indexed citations

Mukherjee, Nabanita, Tonya M. Brunetti, Kasey L. Couts, et al.. (2024). MCL1 inhibition targets Myeloid Derived Suppressors Cells, promotes antitumor immunity and enhances the efficacy of immune checkpoint blockade. Cell Death and Disease. 15(3). 198–198. 5 indexed citations

Li, Hong, Caiguo Zhang, Li Bian, et al.. (2021). Inhibition of CtBP-Regulated Proinflammatory Gene Transcription Attenuates Psoriatic Skin Inflammation. Journal of Investigative Dermatology. 142(2). 390–401. 4 indexed citations

Mukherjee, Nabanita, Carol M. Amato, James R. Lambert, et al.. (2021). Expression Differences in BCL2 Family Members between Uveal and Cutaneous Melanomas Account for Varying Sensitivity to BH3 Mimetics. Journal of Investigative Dermatology. 142(7). 1912–1922.e7. 4 indexed citations

Goldstein, Nathaniel B., Andrea Steel, Maranke I. Koster, et al.. (2020). Melanocyte Precursors in the Hair Follicle Bulge of Repigmented Vitiligo Skin Are Controlled by RHO-GTPase, KCTD10, and CTNNB1 Signaling. Journal of Investigative Dermatology. 141(3). 638–647.e13. 15 indexed citations

Wieser, Iris, David A. Norris, Vera H. Price, et al.. (2018). Childhood alopecia areata—Data from the National Alopecia Areata Registry. Pediatric Dermatology. 35(2). 164–169. 24 indexed citations

Chapman, Stephanie, et al.. (2018). Performance of a computer-aided digital dermoscopic image analyzer for melanoma detection in 1,076 pigmented skin lesion biopsies. Journal of the American Academy of Dermatology. 78(5). 927–934.e6. 17 indexed citations

Eguether, Thibaut, Evan Jones, David A. Norris, et al.. (2017). INTU is essential for oncogenic Hh signaling through regulating primary cilia formation in basal cell carcinoma. Oncogene. 36(35). 4997–5005. 18 indexed citations

10.

Zhai, Zili, Manjinder Kaur, Yiqun G. Shellman, et al.. (2016). 647 NLRP1 promotes tumor growth by enhancing inflammasome activation and suppressing caspase-3 activity in human melanoma. Journal of Investigative Dermatology. 136(5). S114–S114. 2 indexed citations

11.

Liu, Weimin, Yuchun Luo, Jeffrey H. Dunn, et al.. (2012). Dual Role of Apoptosis-Associated Speck-Like Protein Containing a CARD (ASC) in Tumorigenesis of Human Melanoma. Journal of Investigative Dermatology. 133(2). 518–527. 85 indexed citations

12.

Luo, Yuchun, Lixia Z. Ellis, Katiuscia Dallaglio, et al.. (2012). Side Population Cells from Human Melanoma Tumors Reveal Diverse Mechanisms for Chemoresistance. Journal of Investigative Dermatology. 132(10). 2440–2450. 67 indexed citations

13.

Hu, Ling‐Jia, Li Li, James E. Fitzpatrick, et al.. (2007). The Proinflammatory Cytokine Interleukin-32 is expressed in Keratinocytes and Dendritic Cells Obtained from Patients with Chronic Plaque Psoriasis (CPPs) (92.15). The Journal of Immunology. 178(1_Supplement). S165–S165. 2 indexed citations

14.

Norris, David A.. (2003). How Close Are We to Solving the Puzzle? Review of the Alopecia Areata Research Workshop David Norris. Journal of Investigative Dermatology Symposium Proceedings. 8(2). 222–225. 5 indexed citations

15.

Khlgatian, Mary K., Ina Hadshiew, Pravit Asawanonda, et al.. (2002). Tyrosinase Gene Expression is Regulated by p53. Journal of Investigative Dermatology. 118(1). 126–132. 82 indexed citations

16.

Norris, David A.. (2001). In this Issue. Journal of Investigative Dermatology. 117(5). 1025–1026. 3 indexed citations

17.

Leung, Donald Y.M., Jeffrey B. Travers, Ralph Giorno, et al.. (1995). Evidence for a streptococcal superantigen-driven process in acute guttate psoriasis.. Journal of Clinical Investigation. 96(5). 2106–2112. 225 indexed citations

18.

Norris, David A., et al.. (1994). The role of Bcl-2 and Fas in control of apoptosis in keratinocytes and melanocytes. Journal of Investigative Dermatology. 103(3). 442. 1 indexed citations

19.

Leung, Donald Y.M., Ronald J. Harbeck, Peyvand Bina, et al.. (1993). Presence of IgE antibodies to staphylococcal exotoxins on the skin of patients with atopic dermatitis. Evidence for a new group of allergens.. Journal of Clinical Investigation. 92(3). 1374–1380. 405 indexed citations

20.

Norris, David A.. (1989). Six citation classics from the journal of investigative dermatology. Journal of Investigative Dermatology. 92(4). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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