Daniela Wuttig

1.4k total citations
15 papers, 873 citations indexed

About

Daniela Wuttig is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cancer Research. According to data from OpenAlex, Daniela Wuttig has authored 15 papers receiving a total of 873 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 6 papers in Pulmonary and Respiratory Medicine and 6 papers in Cancer Research. Recurrent topics in Daniela Wuttig's work include Renal cell carcinoma treatment (6 papers), Renal and related cancers (5 papers) and MicroRNA in disease regulation (5 papers). Daniela Wuttig is often cited by papers focused on Renal cell carcinoma treatment (6 papers), Renal and related cancers (5 papers) and MicroRNA in disease regulation (5 papers). Daniela Wuttig collaborates with scholars based in Germany, Netherlands and Austria. Daniela Wuttig's co-authors include Holger Sültmann, Jan C. Brase, Ruprecht Kuner, Manfred P. Wirth, Marieta Toma, Susanne Fuessel, Axel Meye, Marc‐Oliver Grimm, Alexander Herr and Matthias Meinhardt and has published in prestigious journals such as Biochemical and Biophysical Research Communications, The Journal of Urology and International Journal of Cancer.

In The Last Decade

Daniela Wuttig

14 papers receiving 860 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Daniela Wuttig Germany 11 642 511 180 93 69 15 873
Sabine Hellwig United States 10 455 0.7× 516 1.0× 172 1.0× 237 2.5× 33 0.5× 18 800
Marion Krieg Germany 8 494 0.8× 495 1.0× 102 0.6× 103 1.1× 61 0.9× 8 748
Harald Surowy Germany 14 480 0.7× 407 0.8× 118 0.7× 168 1.8× 38 0.6× 28 745
David Brocks Germany 10 584 0.9× 266 0.5× 94 0.5× 151 1.6× 69 1.0× 14 807
Michael J. Burdick United States 9 434 0.7× 207 0.4× 166 0.9× 192 2.1× 61 0.9× 12 792
Christina Manley United States 12 438 0.7× 214 0.4× 143 0.8× 183 2.0× 32 0.5× 13 633
Ilaria Alborelli Switzerland 13 309 0.5× 258 0.5× 167 0.9× 198 2.1× 46 0.7× 30 634
Farkas Sükösd Hungary 14 348 0.5× 169 0.3× 168 0.9× 82 0.9× 88 1.3× 45 588
Ensel Oh South Korea 17 397 0.6× 263 0.5× 247 1.4× 323 3.5× 55 0.8× 26 806
Dat T. Vo United States 14 447 0.7× 295 0.6× 65 0.4× 95 1.0× 41 0.6× 45 650

Countries citing papers authored by Daniela Wuttig

Since Specialization
Citations

This map shows the geographic impact of Daniela Wuttig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniela Wuttig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniela Wuttig more than expected).

Fields of papers citing papers by Daniela Wuttig

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniela Wuttig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniela Wuttig. The network helps show where Daniela Wuttig may publish in the future.

Co-authorship network of co-authors of Daniela Wuttig

This figure shows the co-authorship network connecting the top 25 collaborators of Daniela Wuttig. A scholar is included among the top collaborators of Daniela Wuttig based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniela Wuttig. Daniela Wuttig is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Schirmer, Uwe, Kai Doberstein, Anne-Kathleen Rupp, et al.. (2014). Role of miR-34a as a suppressor of L1CAM in endometrial carcinoma. Oncotarget. 5(2). 462–472. 57 indexed citations
2.
Kuner, Ruprecht, Jan C. Brase, Holger Sültmann, & Daniela Wuttig. (2012). microRNA biomarkers in body fluids of prostate cancer patients. Methods. 59(1). 132–137. 45 indexed citations
3.
Sultan, Marc, Simon Dökel, Vyacheslav Amstislavskiy, et al.. (2012). A simple strand-specific RNA-Seq library preparation protocol combining the Illumina TruSeq RNA and the dUTP methods. Biochemical and Biophysical Research Communications. 422(4). 643–646. 53 indexed citations
4.
Toma, Marieta, Daniela Wuttig, Alexander Herr, et al.. (2012). PARK2 and PACRG are commonly downregulated in clear‐cell renal cell carcinoma and are associated with aggressive disease and poor clinical outcome. Genes Chromosomes and Cancer. 52(3). 265–273. 25 indexed citations
5.
Wuttig, Daniela, Stefan Zastrow, S. Füssel, et al.. (2011). CD31, EDNRB and TSPAN7 are promising prognostic markers in clear‐cell renal cell carcinoma revealed by genome‐wide expression analyses of primary tumors and metastases. International Journal of Cancer. 131(5). E693–704. 74 indexed citations
6.
Gade, Stephan, Christine Porzelius, Maria Fälth, et al.. (2011). Graph based fusion of miRNA and mRNA expression data improves clinical outcome prediction in prostate cancer. BMC Bioinformatics. 12(1). 488–488. 36 indexed citations
7.
Brase, Jan C., Daniela Wuttig, Ruprecht Kuner, & Holger Sültmann. (2010). Serum microRNAs as non-invasive biomarkers for cancer. Molecular Cancer. 9(1). 306–306. 370 indexed citations
8.
Wuttig, Daniela, Susanne Fuessel, Marieta Toma, et al.. (2009). 1027 GENE SIGNATURES OF PULMONARY RENAL CELL CARCINOMA (RCC) METASTASES REFLECT METASTASES-FREE INTERVAL AND NUMBER OF METASTASES OF THE PATIENT. European Urology Supplements. 8(4). 377–377.
9.
Wuttig, Daniela, Susanne Fuessel, Matthias Meinhardt, et al.. (2009). Gene signatures of pulmonary metastases of renal cell carcinoma reflect the disease‐free interval and the number of metastases per patient. International Journal of Cancer. 125(2). 474–482. 86 indexed citations
10.
Toma, Marieta, Marianne Großer, Alexander Herr, et al.. (2008). Loss of Heterozygosity and Copy Number Abnormality in Clear Cell Renal Cell Carcinoma Discovered by High-Density Affymetrix 10K Single Nucleotide Polymorphism Mapping Array. Neoplasia. 10(7). 634–642. 66 indexed citations
11.
Wuttig, Daniela, Matthias Meinhardt, Stefan Zastrow, et al.. (2008). GENE SIGNATURES PREDICT METASTASES-FREE INTERVAL AND METASTASES NUMBER IN PULMONARY RENAL CELL CARCINOMA (RCC) METASTASES. The Journal of Urology. 179(4S). 136–136. 2 indexed citations
12.
Kunze, Doreen, Daniela Wuttig, Ingo Kausch, et al.. (2008). Antisense-mediated inhibition of survivin, hTERT and VEGF in bladder cancer cells in vitro and in vivo. International Journal of Oncology. 32(5). 1049–56. 22 indexed citations
13.
Kunze, Doreen, Daniela Wuttig, Susanne Fuessel, et al.. (2008). Multitarget siRNA inhibition of antiapoptotic genes (XIAP, BCL2, BCL-X(L)) in bladder cancer cells.. PubMed. 28(4B). 2259–63. 25 indexed citations
14.
Wuttig, Daniela, Doreen Kunze, Susanne Fuessel, et al.. (2007). Are overexpressed alternative survivin transcripts in human bladder cancer suitable targets for siRNA-mediated in vitro inhibition?. International Journal of Oncology. 30(6). 1317–24. 10 indexed citations
15.
Kunze, Doreen, Daniela Wuttig, S. Füssel, Axel Meye, & Manfred P. Wirth. (2006). SIRNA-MEDIATED INHIBITION OF ANTIAPOPTOTIC GENES IN HUMAN BLADDER CANCER CELLS. European Urology Supplements. 5(14). 800–800. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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