Daniela Novick
- Immunology top 5%
- Oncology top 10%
- Molecular Biology
- Epidemiology
- Rheumatology top 10%
- Co-authors
- Menachem RubinsteinSoo Hyun KimYolande ChvatchkoGiamila FantuzziW. F. KeaneIlana BelzerBoris SchwartsburdCharles A. Dinarello
- Topics
- Inflammasome and immune disorders (5 papers)Cytokine Signaling Pathways and Interactions (3 papers)Dermatological and COVID-19 studies (2 papers)
- Cited by
- ImmunologyOncologyHematology
- Partner nations
- IsraelUnited StatesAustria
In The Last Decade
Daniela Novick
13 papers receiving 1.0k citations
Hit Papers
Peers
Comparison fields: 5 of 80
- Immunology 655
- Oncology 455
- Molecular Biology 364
- Epidemiology 126
- Rheumatology 103
Countries citing papers authored by Daniela Novick
This map shows the geographic impact of Daniela Novick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniela Novick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniela Novick more than expected).
Fields of papers citing papers by Daniela Novick
This network shows the impact of papers produced by Daniela Novick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniela Novick. The network helps show where Daniela Novick may publish in the future.
Co-authorship network of co-authors of Daniela Novick
This figure shows the co-authorship network connecting the top 25 collaborators of Daniela Novick. A scholar is included among the top collaborators of Daniela Novick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniela Novick. Daniela Novick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 8 | |
| 2 | 9 | |
| 3 | 45 | |
| 4 | 221 | |
| 5 | 33 | |
| 6 | 14 | |
| 7 | 57 | |
| 8 | 15 | |
| 9 | 17 | |
| 10 | 14 | |
| 11 | The human interferon $alpha;/$beta; receptor: Characterization and molecular cloningbreakdown → | 557 |
| 12 | Aderka, D. et al. Interleukin-6 inhibits the proliferation of B-chronic lymphocytic leukemia cells that is induced by tumor necrosis factor- or -. Blood 81, 2076−2084 | 17 |
| 13 | 77 |
About Daniela Novick
Daniela Novick is a scholar working on Dermatology, Immunology and Allergy and Immunology, having authored 13 papers that have together received 1.1k indexed citations. Recurring topics across this work include Inflammasome and immune disorders (5 papers), Cytokine Signaling Pathways and Interactions (3 papers) and Dermatological and COVID-19 studies (2 papers). The work is most often cited by research in Immunology (655 citations), Oncology (455 citations) and Hematology (90 citations). Daniela Novick has collaborated with scholars based in Israel, United States and Austria. Frequent co-authors include Menachem Rubinstein, Soo Hyun Kim, Yolande Chvatchko, Giamila Fantuzzi, W. F. Keane, Ilana Belzer, Boris Schwartsburd, Charles A. Dinarello, Ron Pinkus and Zev Sthoeger. Their work appears in journals such as Cell, International Journal of Molecular Sciences and European Journal of Immunology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.