Daniel Fernández-Pérez

1.6k total citations
18 papers, 1.0k citations indexed

About

Daniel Fernández-Pérez is a scholar working on Molecular Biology, Physiology and Genetics. According to data from OpenAlex, Daniel Fernández-Pérez has authored 18 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 2 papers in Physiology and 2 papers in Genetics. Recurrent topics in Daniel Fernández-Pérez's work include Epigenetics and DNA Methylation (7 papers), CRISPR and Genetic Engineering (4 papers) and Genomics and Chromatin Dynamics (4 papers). Daniel Fernández-Pérez is often cited by papers focused on Epigenetics and DNA Methylation (7 papers), CRISPR and Genetic Engineering (4 papers) and Genomics and Chromatin Dynamics (4 papers). Daniel Fernández-Pérez collaborates with scholars based in Italy, Spain and United States. Daniel Fernández-Pérez's co-authors include Diego Pasini, Marika Zanotti, Simone Tamburri, Daria Manganaro, Elisa Lavarone, Eric Conway, Karin Johanna Ferrari, Tiziana Bonaldi, Claudio Isella and Andrea Scelfo and has published in prestigious journals such as Science, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Daniel Fernández-Pérez

17 papers receiving 1.0k citations

Peers

Daniel Fernández-Pérez

ONCOL

GENET

IMMUN

Zhen Sun China

Charlie Degui Chen China

Jeremy E. Coffin United States

Erikjan Rijkers Netherlands

Julia Schultz Germany

Eva M. Monsalve Spain

Claudio Cantù Sweden

Elisia D. Tichy United States

Daniel Alpern Switzerland

Oliver M. Dovey United Kingdom

Zhen Sun China

Daniel Fernández-Pérez

631 ×0.9

297 ×1.5

113 ×0.9

ONCOL

51 ×0.4

GENET

142 ×1.7

IMMUN

Citations per year, relative to Daniel Fernández-Pérez Daniel Fernández-Pérez (= 1×) peers Zhen Sun

Countries citing papers authored by Daniel Fernández-Pérez

Since Specialization

Citations

This map shows the geographic impact of Daniel Fernández-Pérez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel Fernández-Pérez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel Fernández-Pérez more than expected).

Fields of papers citing papers by Daniel Fernández-Pérez

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel Fernández-Pérez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel Fernández-Pérez. The network helps show where Daniel Fernández-Pérez may publish in the future.

Co-authorship network of co-authors of Daniel Fernández-Pérez

This figure shows the co-authorship network connecting the top 25 collaborators of Daniel Fernández-Pérez. A scholar is included among the top collaborators of Daniel Fernández-Pérez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniel Fernández-Pérez. Daniel Fernández-Pérez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Fernández-Pérez, Daniel, Jean‐Claude Lavoie, Nilay Gandhi, et al.. (2025). First-in-US Pilot Study of Aquablation in Ambulatory Surgery Centers: Clinical Outcomes and Economic Feasibility. Urology. 207. 220–224.

Fernández-Pérez, Daniel, et al.. (2025). Pre-existing stem cell heterogeneity dictates clonal responses to the acquisition of leukemic driver mutations. Cell stem cell. 32(4). 564–580.e6. 2 indexed citations

Fernández-Pérez, Daniel, et al.. (2024). SpikeFlow: automated and flexible analysis of ChIP-Seq data with spike-in control. NAR Genomics and Bioinformatics. 6(3). lqae118–lqae118. 3 indexed citations

Fernández-Pérez, Daniel, Georgia Lattanzi, Marika Zanotti, et al.. (2024). PCGF6 controls murine Tuft cell differentiation via H3K9me2 modification independently of Polycomb repression. Developmental Cell. 59(3). 368–383.e7. 3 indexed citations

Larriba, Eduardo, et al.. (2022). Unraveling mitochondrial piRNAs in mouse embryonic gonadal cells. Scientific Reports. 12(1). 10730–10730. 4 indexed citations

Fernández-Pérez, Daniel, Fabrizia Marullo, Roberta Maggio, et al.. (2021). Prdm16-mediated H3K9 methylation controls fibro-adipogenic progenitors identity during skeletal muscle repair. Science Advances. 7(23). 44 indexed citations

Carloni, Sara, Alice Bertocchi, Sara Mancinelli, et al.. (2021). Identification of a choroid plexus vascular barrier closing during intestinal inflammation. Science. 374(6566). 439–448. 185 indexed citations

Fernández-Pérez, Daniel, Samuel Demharter, Ulrich Pfisterer, et al.. (2021). Coordinated maintenance of H3K36/K27 methylation by histone demethylases preserves germ cell identity and immortality. Cell Reports. 37(8). 110050–110050. 5 indexed citations

Conway, Eric, Daniel Fernández-Pérez, Karin Johanna Ferrari, et al.. (2021). BAP1 enhances Polycomb repression by counteracting widespread H2AK119ub1 deposition and chromatin condensation. Molecular Cell. 81(17). 3526–3541.e8. 58 indexed citations

10.

Fernández-Pérez, Daniel, et al.. (2020). Diversification of piRNAs expressed in PGCs and somatic cells during embryonic gonadal development. RNA Biology. 17(9). 1309–1323. 9 indexed citations

11.

Ferrari, Karin Johanna, Roberta Noberini, Daniel Fernández-Pérez, et al.. (2020). Intestinal differentiation involves cleavage of histone H3 N-terminal tails by multiple proteases. Nucleic Acids Research. 49(2). 791–804. 23 indexed citations

12.

Fernández-Pérez, Daniel, Alessandro d’Ambrosio, Caterina Barbieri, et al.. (2019). Loss of PRC1 activity in different stem cell compartments activates a common transcriptional program with cell type–dependent outcomes. Science Advances. 5(5). eaav1594–eaav1594. 21 indexed citations

13.

Scelfo, Andrea, Daniel Fernández-Pérez, Simone Tamburri, et al.. (2019). Functional Landscape of PCGF Proteins Reveals Both RING1A/B-Dependent-and RING1A/B-Independent-Specific Activities. Molecular Cell. 74(5). 1037–1052.e7. 103 indexed citations

14.

Tamburri, Simone, Elisa Lavarone, Daniel Fernández-Pérez, et al.. (2019). Histone H2AK119 Mono-Ubiquitination Is Essential for Polycomb-Mediated Transcriptional Repression. Molecular Cell. 77(4). 840–856.e5. 234 indexed citations

15.

Apicella, Maria, Elisa Giannoni, Stephany Fiore, et al.. (2018). Increased Lactate Secretion by Cancer Cells Sustains Non-cell-autonomous Adaptive Resistance to MET and EGFR Targeted Therapies. Cell Metabolism. 28(6). 848–865.e6. 223 indexed citations

16.

Fernández-Pérez, Daniel, et al.. (2017). Cellular and molecular characterization of gametogenic progression in ex vivo cultured prepuberal mouse testes. Reproductive Biology and Endocrinology. 15(1). 85–85. 17 indexed citations

17.

Fernández-Pérez, Daniel, et al.. (2017). MicroRNA dynamics at the onset of primordial germ and somatic cell sex differentiation during mouse embryonic gonad development. RNA. 24(3). 287–303. 30 indexed citations

18.

Kishibe, Mari, Yoshio Bandô, Ryuji Terayama, et al.. (2006). Kallikrein 8 Is Involved in Skin Desquamation in Cooperation with Other Kallikreins. Journal of Biological Chemistry. 282(8). 5834–5841. 45 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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