Damir Hamamdzic

1.2k total citations
21 papers, 894 citations indexed

About

Damir Hamamdzic is a scholar working on Surgery, Immunology and Infectious Diseases. According to data from OpenAlex, Damir Hamamdzic has authored 21 papers receiving a total of 894 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Surgery, 6 papers in Immunology and 5 papers in Infectious Diseases. Recurrent topics in Damir Hamamdzic's work include Coronary Interventions and Diagnostics (5 papers), Protease and Inhibitor Mechanisms (5 papers) and Viral gastroenteritis research and epidemiology (4 papers). Damir Hamamdzic is often cited by papers focused on Coronary Interventions and Diagnostics (5 papers), Protease and Inhibitor Mechanisms (5 papers) and Viral gastroenteritis research and epidemiology (4 papers). Damir Hamamdzic collaborates with scholars based in United States, India and United Kingdom. Damir Hamamdzic's co-authors include Robert L. Wilensky, Emile R. Mohler, Robert S. Fenning, E. Carwile LeRoy, Colin H. Macphee, Yi Shi, Andrew Zalewski, Mark Burgert, Martin Keane and Laura M. Kasman and has published in prestigious journals such as Nature Medicine, European Heart Journal and Arteriosclerosis Thrombosis and Vascular Biology.

In The Last Decade

Damir Hamamdzic

21 papers receiving 869 citations

Peers

Damir Hamamdzic
Damir Hamamdzic
Citations per year, relative to Damir Hamamdzic Damir Hamamdzic (= 1×) peers Hidehisa Horiguchi

Countries citing papers authored by Damir Hamamdzic

Since Specialization
Citations

This map shows the geographic impact of Damir Hamamdzic's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Damir Hamamdzic with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Damir Hamamdzic more than expected).

Fields of papers citing papers by Damir Hamamdzic

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Damir Hamamdzic. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Damir Hamamdzic. The network helps show where Damir Hamamdzic may publish in the future.

Co-authorship network of co-authors of Damir Hamamdzic

This figure shows the co-authorship network connecting the top 25 collaborators of Damir Hamamdzic. A scholar is included among the top collaborators of Damir Hamamdzic based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Damir Hamamdzic. Damir Hamamdzic is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Saybolt, Matthew D., Scott Lilly, Dhavalkumar Patel, et al.. (2016). The vulnerable artery: early and rapid deposition of lipid in coronary arteries is associated with subsequent development of thin-cap fibroatheromas. EuroIntervention. 11(14). e1612–e1618. 9 indexed citations
2.
Fenning, Robert S., Mark Burgert, Damir Hamamdzic, et al.. (2015). Atherosclerotic Plaque Inflammation Varies Between Vascular Sites and Correlates With Response to Inhibition of Lipoprotein‐Associated Phospholipase A 2. Journal of the American Heart Association. 4(2). 18 indexed citations
3.
Hamamdzic, Damir & Robert L. Wilensky. (2013). Porcine Models of Accelerated Coronary Atherosclerosis: Role of Diabetes Mellitus and Hypercholesterolemia. Journal of Diabetes Research. 2013. 1–7. 60 indexed citations
4.
Patel, Dhavalkumar, et al.. (2013). Subsequent Development of Fibroatheromas With Inflamed Fibrous Caps Can Be Predicted by Intracoronary Near Infrared Spectroscopy. Arteriosclerosis Thrombosis and Vascular Biology. 33(2). 347–353. 27 indexed citations
5.
Winsor-Hines, Dawn, Dhavalkumar Patel, Paul S. Seifert, et al.. (2011). Vascular Responses to Drug-Eluting and Bare Metal Stents in Diabetic/Hypercholesterolemic and Nonatherosclerotic Porcine Coronary Arteries. Circulation Cardiovascular Interventions. 4(5). 438–446. 17 indexed citations
6.
Hamamdzic, Damir, Robert S. Fenning, Dhavalkumar Patel, et al.. (2010). Akt pathway is hypoactivated by synergistic actions of diabetes mellitus and hypercholesterolemia resulting in advanced coronary artery disease. American Journal of Physiology-Heart and Circulatory Physiology. 299(3). H699–H706. 38 indexed citations
7.
Mohler, Emile R., Yuquan Shi, Jonni S. Moore, et al.. (2009). Diabetes reduces bone marrow and circulating porcine endothelial progenitor cells, an effect ameliorated by atorvastatin and independent of cholesterol. PMC. 2 indexed citations
8.
Mohler, Emile R., Yuquan Shi, Jonni S. Moore, et al.. (2008). Diabetes reduces bone marrow and circulating porcine endothelial progenitor cells, an effect ameliorated by atorvastatin and independent of cholesterol. Cytometry Part A. 75A(1). 75–82. 30 indexed citations
9.
Zhou, Rong, Hualei Zhang, Damir Hamamdzic, et al.. (2008). Intracoronary delivery of mesenchymal stem cells at high flow rates after myocardial infarction improves distal coronary blood flow and decreases mortality in pigs. Catheterization and Cardiovascular Interventions. 73(2). 251–257. 19 indexed citations
10.
Wilensky, Robert L., Yi Shi, Emile R. Mohler, et al.. (2008). Inhibition of lipoprotein-associated phospholipase A2 reduces complex coronary atherosclerotic plaque development. Nature Medicine. 14(10). 1059–1066. 294 indexed citations
11.
Mohler, Emile R., Lea Sarov‐Blat, Yi Shi, et al.. (2008). Site-Specific Atherogenic Gene Expression Correlates With Subsequent Variable Lesion Development in Coronary and Peripheral Vasculature. Arteriosclerosis Thrombosis and Vascular Biology. 28(5). 850–855. 51 indexed citations
12.
Wilensky, Robert L. & Damir Hamamdzic. (2007). The molecular basis of vulnerable plaque: potential therapeutic role for immunomodulation. Current Opinion in Cardiology. 22(6). 545–551. 32 indexed citations
13.
Hamamdzic, Damir, Laura M. Kasman, & E. Carwile LeRoy. (2002). The role of infectious agents in the pathogenesis of systemic sclerosis. Current Opinion in Rheumatology. 14(6). 694–698. 47 indexed citations
14.
Hamamdzic, Damir, Russell A. Harley, Debra J. Hazen‐Martin, & E. Carwile LeRoy. (2001). MCMV induces neointima in IFN-γR-/- mice: Intimal cell apoptosis and persistent proliferation of myofibroblasts. BMC Musculoskeletal Disorders. 2(1). 3–3. 26 indexed citations
15.
Hamamdzic, Damir, et al.. (2001). Reovirus triggers cell type-specific proinflammatory responses dependent on the autocrine action of IFN-β. American Journal of Physiology-Lung Cellular and Molecular Physiology. 280(1). L18–L29. 10 indexed citations
16.
Hamamdzic, Damir, et al.. (1999). Prolonged Induction of IL-8 Gene Expression in a Human Fibroblast Cell Line Infected with Reovirus Serotype 1 Strain Lang. Clinical Immunology. 91(1). 25–33. 7 indexed citations
18.
Thompson, A., Lucille London, Stephen Bellum, et al.. (1996). Respiratory-Mucosal Lymphocyte Populations Induced by Reovirus Serotype 1 Infection. Cellular Immunology. 169(2). 278–287. 16 indexed citations
19.
Bellum, Stephen, Damir Hamamdzic, A. Thompson, et al.. (1996). Experimental reovirus serotype 1/strain Lang infection of the lung: a model for the study of the lung in the context of mucosal immunity.. PubMed. 74(1). 221–31. 18 indexed citations
20.
Hamamdzic, Damir, et al.. (1995). Regulation of alpha 2-adrenergic receptor expression and signaling in pancreatic beta-cells. American Journal of Physiology-Endocrinology and Metabolism. 269(1). E162–E171. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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