Dale M. Seth
- Cardiology and Cardiovascular Medicine top 1%
- Endocrinology, Diabetes and Metabolism top 1%
- Molecular Biology top 10%
- Nutrition and Dietetics top 2%
- Pharmacology top 5%
- Co-authors
- L. Gabriel NavarAkira NishiyamaHiroyuki KoboriAlexis A. GonzálezWeijian ShaoLucienne S. LaraMinolfa C. PrietoMinolfa C. Prieto‐Carrasquero
- Topics
- Renin-Angiotensin System Studies (38 papers)Hormonal Regulation and Hypertension (30 papers)Receptor Mechanisms and Signaling (11 papers)
- Partner nations
- United StatesJapanBrazil
In The Last Decade
Dale M. Seth
41 papers receiving 2.3k citations
Peers
Comparison fields: 5 of 90
- Cardiology and Cardiovascular Medicine 1.5k
- Endocrinology, Diabetes and Metabolism 1.3k
- Molecular Biology 702
- Nutrition and Dietetics 374
- Pharmacology 266
Countries citing papers authored by Dale M. Seth
This map shows the geographic impact of Dale M. Seth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dale M. Seth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dale M. Seth more than expected).
Fields of papers citing papers by Dale M. Seth
This network shows the impact of papers produced by Dale M. Seth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dale M. Seth. The network helps show where Dale M. Seth may publish in the future.
Co-authorship network of co-authors of Dale M. Seth
This figure shows the co-authorship network connecting the top 25 collaborators of Dale M. Seth. A scholar is included among the top collaborators of Dale M. Seth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dale M. Seth. Dale M. Seth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 48 | |
| 2 | 19 | |
| 3 | 61 | |
| 4 | 169 | |
| 5 | 38 | |
| 6 | 22 | |
| 7 | 64 | |
| 8 | 48 | |
| 9 | 106 | |
| 10 | 73 | |
| 11 | 50 | |
| 12 | 95 | |
| 13 | 93 | |
| 14 | 23 | |
| 15 | 47 | |
| 16 | 104 | |
| 17 | 77 | |
| 18 | 129 | |
| 19 | 39 | |
| 20 | 57 |
About Dale M. Seth
Dale M. Seth is a scholar working on Endocrinology, Diabetes and Metabolism, Cardiology and Cardiovascular Medicine and Nutrition and Dietetics, having authored 42 papers that have together received 2.3k indexed citations. Recurring topics across this work include Renin-Angiotensin System Studies (38 papers), Hormonal Regulation and Hypertension (30 papers) and Receptor Mechanisms and Signaling (11 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (1.3k citations), Cardiology and Cardiovascular Medicine (1.5k citations) and Nephrology (234 citations). Dale M. Seth has collaborated with scholars based in United States, Japan and Brazil. Frequent co-authors include L. Gabriel Navar, Akira Nishiyama, Hiroyuki Kobori, Alexis A. González, Weijian Shao, Lucienne S. Lara, Minolfa C. Prieto, Minolfa C. Prieto‐Carrasquero, Yumei Feng and M. Prieto. Their work appears in journals such as Journal of Clinical Investigation, PLoS ONE and Circulation Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.