D. Eccleston

4.5k total citations
94 papers, 3.5k citations indexed

About

D. Eccleston is a scholar working on Cellular and Molecular Neuroscience, Psychiatry and Mental health and Molecular Biology. According to data from OpenAlex, D. Eccleston has authored 94 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Cellular and Molecular Neuroscience, 22 papers in Psychiatry and Mental health and 14 papers in Molecular Biology. Recurrent topics in D. Eccleston's work include Neurotransmitter Receptor Influence on Behavior (21 papers), Neuroscience and Neuropharmacology Research (19 papers) and Bipolar Disorder and Treatment (13 papers). D. Eccleston is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (21 papers), Neuroscience and Neuropharmacology Research (19 papers) and Bipolar Disorder and Treatment (13 papers). D. Eccleston collaborates with scholars based in United Kingdom, United States and Australia. D. Eccleston's co-authors include G Ashcroft, A. T. B. Moir, T. B. B. Crawford, Jan Scott, D.S. Walter, Isobel M. Ritchie, H.C. Guldberg, D.F. Sharman, James B. Stanton and J. K. Binns and has published in prestigious journals such as Nature, The Lancet and SHILAP Revista de lepidopterología.

In The Last Decade

D. Eccleston

92 papers receiving 3.1k citations

Peers

D. Eccleston
Comparison fields: 5 of 117
  • Cellular and Molecular Neuroscience 1.4k
  • Psychiatry and Mental health 949
  • Molecular Biology 661
  • Biological Psychiatry 575
  • Pharmacology 573
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Citations per field, relative to D. Eccleston
D. Eccleston · 1×
Citations per year, relative to D. Eccleston
D. Eccleston · 1×

Countries citing papers authored by D. Eccleston

Since Specialization
Citations

This map shows the geographic impact of D. Eccleston's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D. Eccleston with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D. Eccleston more than expected).

Fields of papers citing papers by D. Eccleston

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by D. Eccleston. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D. Eccleston. The network helps show where D. Eccleston may publish in the future.

Co-authorship network of co-authors of D. Eccleston

This figure shows the co-authorship network connecting the top 25 collaborators of D. Eccleston. A scholar is included among the top collaborators of D. Eccleston based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with D. Eccleston. D. Eccleston is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 0
2 138
3 106
4 7
5 3
6 46
7 42
8 38
9 108
10 24
11 39
12 10
13 41
14 11
15 5
16
High dose frusemide in the treatment of hypertension in chronic renal insufficiency and of terminal renal failure.
17
17 12
18 112
19 61
20 144

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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