Cynthia T. Carilli
- Molecular Biology
- Cardiology and Cardiovascular Medicine
- Endocrinology, Diabetes and Metabolism top 10%
- Cell Biology
- Oncology
- Co-authors
- Robert A. FarleyLewis C. CantleyDavid H. HawkeJohn E. ShivelyJohn D. BaxterG. T. HaupertD. PerlmanJohn Lewicki
- Topics
- Receptor Mechanisms and Signaling (4 papers)Ion Transport and Channel Regulation (4 papers)Hormonal Regulation and Hypertension (3 papers)
- Cited by
- Molecular BiologyEndocrinology, Diabetes and MetabolismCardiology and Cardiovascular Medicine
- Partner nations
- United StatesAustraliaCanada
In The Last Decade
Cynthia T. Carilli
11 papers receiving 704 citations
Peers
Comparison fields: 5 of 76
- Molecular Biology 582
- Cardiology and Cardiovascular Medicine 123
- Endocrinology, Diabetes and Metabolism 101
- Cell Biology 87
- Oncology 82
Countries citing papers authored by Cynthia T. Carilli
This map shows the geographic impact of Cynthia T. Carilli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cynthia T. Carilli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cynthia T. Carilli more than expected).
Fields of papers citing papers by Cynthia T. Carilli
This network shows the impact of papers produced by Cynthia T. Carilli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cynthia T. Carilli. The network helps show where Cynthia T. Carilli may publish in the future.
Co-authorship network of co-authors of Cynthia T. Carilli
This figure shows the co-authorship network connecting the top 25 collaborators of Cynthia T. Carilli. A scholar is included among the top collaborators of Cynthia T. Carilli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cynthia T. Carilli. Cynthia T. Carilli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 51 | |
| 2 | 26 | |
| 3 | 36 | |
| 4 | 166 | |
| 5 | 21 | |
| 6 | 45 | |
| 7 | 57 | |
| 8 | 187 | |
| 9 | 40 | |
| 10 | 10 | |
| 11 | 95 |
About Cynthia T. Carilli
Cynthia T. Carilli is a scholar working on Behavioral Neuroscience, Endocrinology, Diabetes and Metabolism and Endocrine and Autonomic Systems, having authored 11 papers that have together received 734 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (4 papers), Ion Transport and Channel Regulation (4 papers) and Hormonal Regulation and Hypertension (3 papers). The work is most often cited by research in Molecular Biology (582 citations), Endocrinology, Diabetes and Metabolism (101 citations) and Cardiology and Cardiovascular Medicine (123 citations). Cynthia T. Carilli has collaborated with scholars based in United States, Australia and Canada. Frequent co-authors include Robert A. Farley, Lewis C. Cantley, David H. Hawke, John E. Shively, John D. Baxter, G. T. Haupert, D. Perlman, John Lewicki, M.E. Fraser and Alastair Muir. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.