Cole S. Nelson
- Cellular and Molecular Neuroscience top 10%
- Molecular Biology
- Endocrine and Autonomic Systems top 5%
- Cognitive Neuroscience
- Physiology
- Co-authors
- Charles N. AllenJennifer L. MarinoKim A. NeveZhi‐Gen JiangDavid B. JacobyZhenying NieA.D. FryerJ.P. Adelman
- Topics
- Receptor Mechanisms and Signaling (6 papers)Ion channel regulation and function (4 papers)Circadian rhythm and melatonin (4 papers)
- Journals
- Brain ResearchJournal of Allergy and Clinical ImmunologyJournal of Pharmacology and Experimental Therapeutics
- Partner nations
- United StatesJapan
In The Last Decade
Cole S. Nelson
16 papers receiving 457 citations
Peers
Comparison fields: 5 of 70
- Cellular and Molecular Neuroscience 259
- Molecular Biology 212
- Endocrine and Autonomic Systems 196
- Cognitive Neuroscience 69
- Physiology 59
Countries citing papers authored by Cole S. Nelson
This map shows the geographic impact of Cole S. Nelson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cole S. Nelson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cole S. Nelson more than expected).
Fields of papers citing papers by Cole S. Nelson
This network shows the impact of papers produced by Cole S. Nelson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cole S. Nelson. The network helps show where Cole S. Nelson may publish in the future.
Co-authorship network of co-authors of Cole S. Nelson
This figure shows the co-authorship network connecting the top 25 collaborators of Cole S. Nelson. A scholar is included among the top collaborators of Cole S. Nelson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cole S. Nelson. Cole S. Nelson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 17 | |
| 2 | 12 | |
| 3 | 9 | |
| 4 | 8 | |
| 5 | 31 | |
| 6 | 5 | |
| 7 | 26 | |
| 8 | 18 | |
| 9 | 12 | |
| 10 | 81 | |
| 11 | 23 | |
| 12 | 22 | |
| 13 | 22 | |
| 14 | 70 | |
| 15 | 88 | |
| 16 | Cloning and expression of a 5HT7 receptor from Xenopus laevis. | 24 |
About Cole S. Nelson
Cole S. Nelson is a scholar working on Endocrine and Autonomic Systems, Cellular and Molecular Neuroscience and Molecular Biology, having authored 16 papers that have together received 468 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (6 papers), Ion channel regulation and function (4 papers) and Circadian rhythm and melatonin (4 papers). The work is most often cited by research in Endocrine and Autonomic Systems (196 citations), Cellular and Molecular Neuroscience (259 citations) and Behavioral Neuroscience (13 citations). Cole S. Nelson has collaborated with scholars based in United States and Japan. Frequent co-authors include Charles N. Allen, Jennifer L. Marino, Kim A. Neve, Zhi‐Gen Jiang, David B. Jacoby, Zhenying Nie, A.D. Fryer, J.P. Adelman, Roger D. Cone and Linda S. Robbins. Their work appears in journals such as Brain Research, Journal of Allergy and Clinical Immunology and Journal of Pharmacology and Experimental Therapeutics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.