Claudine Billat

615 total citations
22 papers, 515 citations indexed

About

Claudine Billat is a scholar working on Physiology, Molecular Biology and Genetics. According to data from OpenAlex, Claudine Billat has authored 22 papers receiving a total of 515 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 10 papers in Molecular Biology and 7 papers in Genetics. Recurrent topics in Claudine Billat's work include Erythrocyte Function and Pathophysiology (15 papers), Hemoglobinopathies and Related Disorders (6 papers) and Protein Kinase Regulation and GTPase Signaling (5 papers). Claudine Billat is often cited by papers focused on Erythrocyte Function and Pathophysiology (15 papers), Hemoglobinopathies and Related Disorders (6 papers) and Protein Kinase Regulation and GTPase Signaling (5 papers). Claudine Billat collaborates with scholars based in France and Morocco. Claudine Billat's co-authors include Patrick Mayeux, R Jacquot, Catherine Lacombe, Bernard Haye, Stany Chrétien, Nicole Casadevall, Isabelle Dusanter‐Fourt, O. Müller, Siegmund Fischer and Frédèrique Verdier and has published in prestigious journals such as Journal of Biological Chemistry, Biochemical and Biophysical Research Communications and FEBS Letters.

In The Last Decade

Claudine Billat

22 papers receiving 504 citations

Peers

Claudine Billat
Comparison fields: 5 of 63
  • Hematology 239
  • Molecular Biology 215
  • Physiology 204
  • Oncology 179
  • Genetics 89
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Citations per field, relative to Claudine Billat
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Citations per year, relative to Claudine Billat
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Countries citing papers authored by Claudine Billat

Since Specialization
Citations

This map shows the geographic impact of Claudine Billat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Claudine Billat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Claudine Billat more than expected).

Fields of papers citing papers by Claudine Billat

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Claudine Billat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Claudine Billat. The network helps show where Claudine Billat may publish in the future.

Co-authorship network of co-authors of Claudine Billat

This figure shows the co-authorship network connecting the top 25 collaborators of Claudine Billat. A scholar is included among the top collaborators of Claudine Billat based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Claudine Billat. Claudine Billat is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 18
2 18
3 21
4
Erythropoietin induction of tissue inhibitors of metalloproteinase-1 expression and secretion is mediated by mitogen-activated protein kinase and phosphatidylinositol 3-kinase pathways.
30
5 2
6 16
7 30
8 85
9 109
10
Glucose uptake by rat erythroid cells: the effects of erythropoietin and dexamethasone.
3
11 6
12 1
13 6
14
Induction by hemin of proliferation and of differentiation of progenitor erythroid cells responsible for erythropoietin.
17
15 5
16 3
17 6
18 8
19 3
20
[Study with electron microscope of rat fetal hepatocytes in primary culture: action of cortisol].
5

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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