Clare L. Fasching

5.3k total citations · 2 hit papers
26 papers, 3.9k citations indexed

About

Clare L. Fasching is a scholar working on Molecular Biology, Oncology and Physiology. According to data from OpenAlex, Clare L. Fasching has authored 26 papers receiving a total of 3.9k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 12 papers in Oncology and 6 papers in Physiology. Recurrent topics in Clare L. Fasching's work include Cancer-related Molecular Pathways (9 papers), Telomeres, Telomerase, and Senescence (6 papers) and DNA Repair Mechanisms (4 papers). Clare L. Fasching is often cited by papers focused on Cancer-related Molecular Pathways (9 papers), Telomeres, Telomerase, and Senescence (6 papers) and DNA Repair Mechanisms (4 papers). Clare L. Fasching collaborates with scholars based in United States, Australia and Germany. Clare L. Fasching's co-authors include Roger R. Reddel, Axel A. Neumann, Janice S. Chen, James P. Broughton, Venice Servellita, Charles Y. Chiu, Alicia Sotomayor-González, Jessica Streithorst, Steve Miller and Elaine Hsu and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Clare L. Fasching

24 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

CRISPR–Cas12-based detection of SARS-CoV-2

2020 2.0k citations James P. Broughton, Xianding Deng et al. Nature Biotechnology profile →
Telomere maintenance by recombination in human cells

2000 698 citations Axel A. Neumann, Clare L. Fasching et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Clare L. Fasching United States	19	2.8k	1.0k	960	830	402	26	3.9k
Mingxiao Ding China	31	2.9k 1.0×	556 0.5×	494 0.5×	176 0.2×	252 0.6×	60	4.4k
Richard Wubbolts Netherlands	35	3.2k 1.1×	329 0.3×	212 0.2×	295 0.4×	387 1.0×	59	5.2k
Mark D. Wright Australia	39	1.9k 0.7×	274 0.3×	258 0.3×	132 0.2×	682 1.7×	123	4.9k
Hong M. Moulton United States	43	4.1k 1.5×	985 1.0×	254 0.3×	200 0.2×	209 0.5×	83	5.5k
Renaud Poincloux France	29	1.2k 0.4×	350 0.3×	457 0.5×	141 0.2×	504 1.3×	50	3.4k
Yeongjin Hong South Korea	34	1.2k 0.4×	231 0.2×	1.6k 1.7×	213 0.3×	318 0.8×	78	3.6k
Giju Thomas United States	24	1.2k 0.4×	359 0.4×	154 0.2×	260 0.3×	303 0.8×	73	3.2k
Nelson O. Gekara Sweden	25	1.2k 0.4×	514 0.5×	221 0.2×	119 0.1×	330 0.8×	40	2.7k
Karla J. Daniels United States	39	2.1k 0.8×	2.2k 2.1×	183 0.2×	176 0.2×	222 0.6×	73	4.1k
Massimo Spada Italy	32	1.9k 0.7×	395 0.4×	245 0.3×	82 0.1×	812 2.0×	58	4.1k

Countries citing papers authored by Clare L. Fasching

Since Specialization

Citations

This map shows the geographic impact of Clare L. Fasching's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Clare L. Fasching with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Clare L. Fasching more than expected).

Fields of papers citing papers by Clare L. Fasching

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Clare L. Fasching. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Clare L. Fasching. The network helps show where Clare L. Fasching may publish in the future.

Co-authorship network of co-authors of Clare L. Fasching

This figure shows the co-authorship network connecting the top 25 collaborators of Clare L. Fasching. A scholar is included among the top collaborators of Clare L. Fasching based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Clare L. Fasching. Clare L. Fasching is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Gore, Steven K, Shannon J. Ceballos, Giordano Reginato, et al.. (2026). Rad51 determines pathway usage in post-replication repair. Nature Communications. 17(1). 1359–1359.

Broughton, James P., Xianding Deng, Guixia Yu, et al.. (2020). CRISPR–Cas12-based detection of SARS-CoV-2. Nature Biotechnology. 38(7). 870–874. 1994 indexed citations breakdown →

Fasching, Clare L.. (2018). Telomere length measurement as a clinical biomarker of aging and disease. Critical Reviews in Clinical Laboratory Sciences. 55(7). 443–465. 112 indexed citations

Fasching, Clare L., Jue Lin, Karl Guegler, et al.. (2017). Analytical Validation of Relative Average Telomere Length Measurement in a Clinical Laboratory Environment. The Journal of Applied Laboratory Medicine. 2(1). 4–16. 1 indexed citations

Fasching, Clare L., Petr Ćejka, Stephen C. Kowalczykowski, & Wolf‐Dietrich Heyer. (2015). Top3-Rmi1 Dissolve Rad51-Mediated D Loops by a Topoisomerase-Based Mechanism. Molecular Cell. 57(4). 595–606. 95 indexed citations

Zhang, Xiaoping, et al.. (2013). A Conserved Sequence Extending Motif III of the Motor Domain in the Snf2-Family DNA Translocase Rad54 Is Critical for ATPase Activity. PLoS ONE. 8(12). e82184–e82184. 9 indexed citations

Fasching, Clare L., et al.. (2007). DNA Damage Induces Alternative Lengthening of Telomeres (ALT)–Associated Promyelocytic Leukemia Bodies that Preferentially Associate with Linear Telomeric DNA. Cancer Research. 67(15). 7072–7077. 74 indexed citations

Fasching, Clare L., et al.. (2005). Telomerase-Independent Telomere Length Maintenance in the Absence of Alternative Lengthening of Telomeres–Associated Promyelocytic Leukemia Bodies. Cancer Research. 65(7). 2722–2729. 79 indexed citations

Neumann, Axel A. & Clare L. Fasching. (2001). Pictures in Molecular Medicine: Telomeres copying telomeres in human cells. Trends in Molecular Medicine. 7(4). 184–184.

10.

Neumann, Axel A., et al.. (2000). Telomere maintenance by recombination in human cells. Nature Genetics. 26(4). 447–450. 698 indexed citations breakdown →

11.

Mendonca, Marc S., Ken Howard, Clare L. Fasching, et al.. (1998). Loss of Suppressor Loci on Chromosomes 11 and 14 May Be Required for Radiation-Induced Neoplastic Transformation of HeLa × Skin Fibroblast Human Cell Hybrids. Radiation Research. 149(3). 246–246. 26 indexed citations

12.

Boukamp, Petra, et al.. (1997). Sustained nontumorigenic phenotype correlates with a largely stable chromosome content during long-term culture of the human keratinocyte line HaCaT. Genes Chromosomes and Cancer. 19(4). 201–214. 119 indexed citations

13.

Plattner, Rina, Michael J. Anderson, Kevin Sato, et al.. (1996). Loss of oncogenic ras expression does not correlate with loss of tumorigenicity in human cells.. Proceedings of the National Academy of Sciences. 93(13). 6665–6670. 67 indexed citations

14.

Mendonca, Marc S., Clare L. Fasching, Eri S. Srivatsan, Eric J. Stanbridge, & J. Leslie Redpath. (1995). Loss of a Putative Tumor Suppressor Locus after Gamma-Ray-Induced Neoplastic Transformation of HeLa × Skin Fibroblast Human Cell Hybrids. Radiation Research. 143(1). 34–34. 31 indexed citations

15.

Anderson, Michael J., et al.. (1994). Evidence that wild‐type TP53, and not genes on either chromosome 1 or 11, controls the tumorigenic phenotype of the human fibrosarcoma HT1080. Genes Chromosomes and Cancer. 9(4). 266–281. 33 indexed citations

16.

Anderson, Michael J., Clare L. Fasching, Eric J. Stanbridge, Hong‐Ji Xu, & William F. Benedict. (1994). Chromosome 13 transfer provides evidence for regulation of RBI protein expression. Genes Chromosomes and Cancer. 9(4). 251–260. 9 indexed citations

17.

Casey, Graham, et al.. (1993). Functional evidence for a breast cancer growth suppressor gene on chromosome 17. Human Molecular Genetics. 2(11). 1921–1927. 32 indexed citations

18.

Bader, Scott, et al.. (1991). Dissociation of suppression of tumorigenicity and differentiation in vitro effected by transfer of single human chromosomes into human neuroblastoma cells.. PubMed. 2(5). 245–55. 90 indexed citations

19.

Dowdy, Steven F., Clare L. Fasching, D Araujo, et al.. (1991). Suppression of Tumorigenicity in Wilms Tumor by the p15.5-p14 Region of Chromosome 11. Science. 254(5029). 293–295. 101 indexed citations

20.

Dowdy, Steven F., et al.. (1990). Irradiation microcell‐mediated chromosome transfer (XMMCT): The generation of specific chromosomal arm deletions. Genes Chromosomes and Cancer. 2(4). 318–327. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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