Claire E. Walters
Impact in
- Immunology and Allergy top 5%
- Cell Adhesion Molecules Research
- Neurology top 10%
- Neuroinflammation and Neurodegeneration Mechanisms
Papers in
- Oncology 4
- Cytokine Signaling Pathways and Interactions 4
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- Protein Tyrosine Phosphatases 1
- Glycosylation and Glycoproteins Research 1
- Co-authors
- Peter Adamson (3 shared papers)David Stephens (1 shared paper)Emma R. Frears (1 shared paper)John Greenwood (3 shared papers)Brian Austen (1 shared paper)Huw Davies (1 shared paper)Gareth Pryce (2 shared papers)David Baker (2 shared papers)
- Journals
- The Journal of Immunology (2 papers)The FASEB Journal (1 paper)Neuroreport (1 paper)Genomics (1 paper)Journal of Biological Chemistry (1 paper)
- Partner nations
- United KingdomAustraliaUnited States
In The Last Decade
Claire E. Walters
7 papers receiving 768 citations
Peers
Comparison fields: 5 of 80
- Immunology and Allergy 104
- Neurology 91
- Physiology 219
- Immunology 150
- Developmental Neuroscience 28
Countries citing papers authored by Claire E. Walters
This map shows the geographic impact of Claire E. Walters's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Claire E. Walters with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Claire E. Walters more than expected).
Fields of papers citing papers by Claire E. Walters
This network shows the impact of papers produced by Claire E. Walters. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Claire E. Walters. The network helps show where Claire E. Walters may publish in the future.
Co-authors
The 25 scholars most cited alongside Claire E. Walters, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1999 | 274 | |
| 2 | 2003 | 187 | |
| 3 | 2003 | 115 | |
| 4 | 2002 | 96 | |
| 5 | 2004 | 68 | |
| 6 | 2005 | 34 | |
| 7 | 1996 | 27 |
About Claire E. Walters
Claire E. Walters is a scholar working on Oncology, Molecular Biology, Immunology and Allergy, Immunology and Computational Theory and Mathematics, having authored 7 papers that have together received 801 indexed citations. Recurring topics across this work include Cytokine Signaling Pathways and Interactions (4 papers), Cell Adhesion Molecules Research (3 papers), interferon and immune responses (2 papers), NF-κB Signaling Pathways (1 paper), Alzheimer's disease research and treatments (1 paper), Computational Drug Discovery Methods (1 paper), Protein Tyrosine Phosphatases (1 paper) and Glycosylation and Glycoproteins Research (1 paper). The work is most often cited by research in Immunology and Allergy (104 citations), Neurology (91 citations), Physiology (219 citations), Immunology (150 citations) and Developmental Neuroscience (28 citations). Claire E. Walters has collaborated with scholars based in United Kingdom, Australia and United States. Frequent co-authors include Peter Adamson, David Stephens, Emma R. Frears, John Greenwood, Brian Austen, Huw Davies, Gareth Pryce, David Baker, Naheed Kanuga and Ann M. Turnley. Their work appears in journals such as The Journal of Immunology, The FASEB Journal, Neuroreport, Genomics and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.