Chu-Sook Kim

508 total citations
10 papers, 430 citations indexed

About

Chu-Sook Kim is a scholar working on Epidemiology, Physiology and Molecular Biology. According to data from OpenAlex, Chu-Sook Kim has authored 10 papers receiving a total of 430 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Epidemiology, 6 papers in Physiology and 3 papers in Molecular Biology. Recurrent topics in Chu-Sook Kim's work include Adipokines, Inflammation, and Metabolic Diseases (6 papers), Adipose Tissue and Metabolism (6 papers) and Muscle Physiology and Disorders (2 papers). Chu-Sook Kim is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (6 papers), Adipose Tissue and Metabolism (6 papers) and Muscle Physiology and Disorders (2 papers). Chu-Sook Kim collaborates with scholars based in South Korea, Japan and Canada. Chu-Sook Kim's co-authors include Rina Yu, Teruo Kawada, Tsuyoshi Goto, Taesun Park, Mi‐Kyung Sung, Hoon Yoo, Hye‐Seon Choi, Jung Han Yoon Park, Yeonsoo Joe and Hun‐Taeg Chung and has published in prestigious journals such as Nutrients, Inflammation Research and Mediators of Inflammation.

In The Last Decade

Chu-Sook Kim

10 papers receiving 426 citations

Peers

Countries citing papers authored by Chu-Sook Kim

Since Specialization

Citations

This map shows the geographic impact of Chu-Sook Kim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chu-Sook Kim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chu-Sook Kim more than expected).

Fields of papers citing papers by Chu-Sook Kim

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chu-Sook Kim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chu-Sook Kim. The network helps show where Chu-Sook Kim may publish in the future.

Co-authorship network of co-authors of Chu-Sook Kim

This figure shows the co-authorship network connecting the top 25 collaborators of Chu-Sook Kim. A scholar is included among the top collaborators of Chu-Sook Kim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chu-Sook Kim. Chu-Sook Kim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	Filbertone Protects Obesity-induced Hypothalamic Inflammation by Reduction of Microglia-mediated Inflammatory Responses 2021 ·Biotechnology and Bioprocess Engineering ·(unknown), (unknown), (unknown), Chu-Sook Kim, (unknown), Min‐Seon Kim, Taesun Park, Tsuyoshi Goto, Rina Yu	7
2	Absence of 4-1BB reduces obesity-induced atrophic response in skeletal muscle 2017 ·Journal of Inflammation ·(unknown), Chu-Sook Kim, Thai Hien Tu, (unknown), Taesun Park, Jung Han Yoon Park, Tsuyoshi Goto, Teruo Kawada, Tae Youl Ha, Rina Yu	1
3	Quercetin Protects Obesity-Induced Hypothalamic Inflammation by Reducing Microglia-Mediated Inflammatory Responses via HO-1 Induction 2017 ·Nutrients ·(unknown), Chu-Sook Kim, Thai Hien Tu, Min‐Seon Kim, Tsuyoshi Goto, Teruo Kawada, Myung-Sook Choi, Taesun Park, Mi‐Kyung Sung, Jong Won Yun, (unknown), Jee Hye Lee, Yeonsoo Joe, Hye‐Seon Choi, Sung Hoon Back, Hun‐Taeg Chung, Rina Yu	61
4	The Soy Peptide Phe–Leu–Val Reduces TNFα-Induced Inflammatory Response and Insulin Resistance in Adipocytes 2016 ·Journal of Medicinal Food ·(unknown), Chu-Sook Kim, Myung-Sook Choi, Taesun Park, Mi‐Kyung Sung, Jong Won Yun, Hoon Yoo, Yoshinori Mine, Rina Yu	41
5	Quercetin reduces obesity-induced hepatosteatosis by enhancing mitochondrial oxidative metabolism via heme oxygenase-1 2015 ·Nutrition & Metabolism ·Chu-Sook Kim, Yoon‐Hee Kwon, (unknown), (unknown), Hoon Yoo, Tsuyoshi Goto, Teruo Kawada, Hye‐Seon Choi, Yeonsoo Joe, Hun‐Taeg Chung, Rina Yu	100
6	The Inhibitory Effect of Quercetin on Adipose Tissue Inflammation in Mice Fed on a High-fat Diet 2014 ·Chu-Sook Kim, Rina Yu	11
7	Quercetin Protects against Obesity-Induced Skeletal Muscle Inflammation and Atrophy 2014 ·Mediators of Inflammation ·(unknown), Chu-Sook Kim, Taesun Park, Jung Han Yoon Park, Mi‐Kyung Sung, Dong Gun Lee, (unknown), (unknown), Tsuyoshi Goto, Teruo Kawada, Rina Yu	128
8	Blockade of 4-1BB and 4-1BBL Interaction Reduces Obesity-Induced Skeletal Muscle Inflammation 2013 ·Mediators of Inflammation ·(unknown), Chu-Sook Kim, Thai Hien Tu, Hye‐Seon Choi, (unknown), Teruo Kawada, Tsuyoshi Goto, Taesun Park, Jung Han Yoon Park, Rina Yu	16
9	Quercetin Suppresses MIP-1α–Induced Adipose Inflammation by Downregulating Its Receptors CCR1/CCR5 and Inhibiting Inflammatory Signaling 2013 ·Journal of Medicinal Food ·(unknown), Chu-Sook Kim, (unknown), (unknown), (unknown), (unknown), Hoon Yoo, Taesun Park, Rina Yu	32
10	Potential involvement of CCL23 in atherosclerotic lesion formation/progression by the enhancement of chemotaxis, adhesion molecule expression, and MMP-2 release from monocytes 2011 ·Inflammation Research ·Chu-Sook Kim, (unknown), (unknown), Thomas N. Blankenship, Kent L. Erickson, Teruo Kawada, Rina Yu	33

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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