Christopher M. Hammell

1.6k total citations
26 papers, 1.1k citations indexed

About

Christopher M. Hammell is a scholar working on Molecular Biology, Aging and Cancer Research. According to data from OpenAlex, Christopher M. Hammell has authored 26 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 18 papers in Aging and 4 papers in Cancer Research. Recurrent topics in Christopher M. Hammell's work include Genetics, Aging, and Longevity in Model Organisms (18 papers), RNA Research and Splicing (11 papers) and CRISPR and Genetic Engineering (7 papers). Christopher M. Hammell is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (18 papers), RNA Research and Splicing (11 papers) and CRISPR and Genetic Engineering (7 papers). Christopher M. Hammell collaborates with scholars based in United States, France and Switzerland. Christopher M. Hammell's co-authors include Victor Ambros, Charles N. Cole, Rosalind C. Lee, Catherine V. Heath, Xantha Karp, Claudio A. Saavedra, Peter R. Boag, T. Keith Blackwell, Christine A. Hodge and Ewan F. Dunn and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Christopher M. Hammell

23 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Christopher M. Hammell United States 15 932 368 231 105 78 26 1.1k
Florian Aeschimann Switzerland 13 738 0.8× 206 0.6× 133 0.6× 66 0.6× 79 1.0× 17 907
Kiyokazu Morita Japan 10 523 0.6× 377 1.0× 148 0.6× 34 0.3× 85 1.1× 12 788
Aaron M. Kershner United States 13 842 0.9× 630 1.7× 53 0.2× 145 1.4× 121 1.6× 14 1.1k
Joshua A. Arribere United States 12 989 1.1× 428 1.2× 48 0.2× 85 0.8× 65 0.8× 20 1.2k
Gabriel D. Hayes United States 7 752 0.8× 135 0.4× 581 2.5× 116 1.1× 25 0.3× 7 953
Sudhir Nayak United States 12 658 0.7× 615 1.7× 33 0.1× 96 0.9× 123 1.6× 19 1.0k
Jennifer A Schisa United States 14 831 0.9× 344 0.9× 40 0.2× 70 0.7× 44 0.6× 23 953
Vipin T. Sreedharan Germany 8 439 0.5× 150 0.4× 64 0.3× 44 0.4× 33 0.4× 10 565
Tory Herman United States 7 502 0.5× 121 0.3× 54 0.2× 44 0.4× 31 0.4× 9 640
Jeb Gaudet Canada 12 666 0.7× 556 1.5× 25 0.1× 79 0.8× 139 1.8× 17 955

Countries citing papers authored by Christopher M. Hammell

Since Specialization
Citations

This map shows the geographic impact of Christopher M. Hammell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher M. Hammell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher M. Hammell more than expected).

Fields of papers citing papers by Christopher M. Hammell

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher M. Hammell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher M. Hammell. The network helps show where Christopher M. Hammell may publish in the future.

Co-authorship network of co-authors of Christopher M. Hammell

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher M. Hammell. A scholar is included among the top collaborators of Christopher M. Hammell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher M. Hammell. Christopher M. Hammell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yarychkivska, Olya, Simin Liu, Stephen A. Newland, et al.. (2025). Non-apoptotic death of the C. elegans linker cell is primed by MYRF-1 activation of pqn-41 /polyQ. bioRxiv (Cold Spring Harbor Laboratory).
2.
Kim, Hyun Soo, Benjamin Roche, Sonali Bhattacharjee, et al.. (2024). Clr4SUV39H1 ubiquitination and non-coding RNA mediate transcriptional silencing of heterochromatin via Swi6 phase separation. Nature Communications. 15(1). 9384–9384. 4 indexed citations
3.
4.
Stec, Natalia, Jing Wang, M. Jaremko, et al.. (2023). A circadian-like gene network programs the timing and dosage of heterochronic miRNA transcription during C. elegans development. Developmental Cell. 58(22). 2563–2579.e8. 13 indexed citations
5.
Yee, Callista, Michael A. Q. Martinez, Wan Zhang, et al.. (2023). An expandable FLP-ON::TIR1 system for precise spatiotemporal protein degradation in Caenorhabditis elegans. Genetics. 223(4). 5 indexed citations
6.
7.
Martinez, Michael A. Q., Natalia Stec, Taylor N. Medwig-Kinney, et al.. (2021). An engineered, orthogonal auxin analog/ At TIR1(F79G) pairing improves both specificity and efficacy of the auxin degradation system in Caenorhabditis elegans. Genetics. 220(2). 36 indexed citations
8.
Carlston, Colleen M., Robin Weinmann, Natalia Stec, et al.. (2021). PQN-59 antagonizes microRNA-mediated repression during post-embryonic temporal patterning and modulates translation and stress granule formation in C. elegans. PLoS Genetics. 17(11). e1009599–e1009599. 7 indexed citations
9.
Stec, Natalia, O. Ramos, Nuri Kim, et al.. (2020). ACaenorhabditis elegansModel for Integrating the Functions of Neuropsychiatric Risk Genes Identifies Components Required for Normal Dendritic Morphology. G3 Genes Genomes Genetics. 10(5). 1617–1628. 12 indexed citations
10.
Stec, Natalia, et al.. (2020). An Epigenetic Priming Mechanism Mediated by Nutrient Sensing Regulates Transcriptional Output during C. elegans Development. Current Biology. 31(4). 809–826.e6. 17 indexed citations
11.
Martinez, Michael A. Q., Taylor N. Medwig-Kinney, James Matthew Ragle, et al.. (2019). Rapid Degradation ofCaenorhabditis elegansProteins at Single-Cell Resolution with a Synthetic Auxin. G3 Genes Genomes Genetics. 10(1). 267–280. 47 indexed citations
12.
Hammell, Christopher M. & Gregory J. Hannon. (2016). Inducing RNAi in Caenorhabditis elegans by Injection of dsRNA. Cold Spring Harbor Protocols. 2016(1). pdb.prot086306–pdb.prot086306. 4 indexed citations
13.
Perales, Roberto, et al.. (2014). LIN-42, the Caenorhabditis elegans PERIOD homolog, Negatively Regulates MicroRNA Transcription. PLoS Genetics. 10(7). e1004486–e1004486. 30 indexed citations
14.
Zinovyeva, Anna, Samir Bouasker, Martin J. Simard, Christopher M. Hammell, & Victor Ambros. (2014). Mutations in Conserved Residues of the C. elegans microRNA Argonaute ALG-1 Identify Separable Functions in ALG-1 miRISC Loading and Target Repression. PLoS Genetics. 10(4). e1004286–e1004286. 33 indexed citations
15.
Hammell, Christopher M., et al.. (2009). nhl-2 Modulates MicroRNA Activity in Caenorhabditis elegans. Cell. 136(5). 926–938. 142 indexed citations
16.
Hammell, Christopher M.. (2008). The microRNA-argonaute complex: A platform for mRNA modulation. RNA Biology. 5(3). 123–127. 33 indexed citations
17.
Dunn, Ewan F., Christopher M. Hammell, Christine A. Hodge, & Charles N. Cole. (2005). Yeast poly(A)-binding protein, Pab1, and PAN, a poly(A) nuclease complex recruited by Pab1, connect mRNA biogenesis to export. Genes & Development. 19(1). 90–103. 89 indexed citations
18.
Cole, Charles N., Catherine V. Heath, Christine A. Hodge, Christopher M. Hammell, & David C. Amberg. (2002). Analysis of RNA export. Methods in enzymology on CD-ROM/Methods in enzymology. 351. 568–587. 17 indexed citations
19.
Cole, Charles N. & Christopher M. Hammell. (1998). Nucleocytoplasmic transport: Driving and directing transport. Current Biology. 8(11). R368–R372. 86 indexed citations
20.
Saavedra, Claudio A., Christopher M. Hammell, Catherine V. Heath, & Charles N. Cole. (1997). Yeast heat shock mRNAs are exported through a distinct pathway defined by Rip1p. Genes & Development. 11(21). 2845–2856. 118 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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