David M. Eisenmann

2.2k total citations
31 papers, 1.8k citations indexed

About

David M. Eisenmann is a scholar working on Aging, Molecular Biology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, David M. Eisenmann has authored 31 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Aging, 20 papers in Molecular Biology and 15 papers in Public Health, Environmental and Occupational Health. Recurrent topics in David M. Eisenmann's work include Genetics, Aging, and Longevity in Model Organisms (23 papers), Reproductive Biology and Fertility (15 papers) and Circadian rhythm and melatonin (7 papers). David M. Eisenmann is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (23 papers), Reproductive Biology and Fertility (15 papers) and Circadian rhythm and melatonin (7 papers). David M. Eisenmann collaborates with scholars based in United States, Netherlands and Russia. David M. Eisenmann's co-authors include Stuart K. Kim, Fred Winston, Julie E. Gleason, Catherine Dollard, Karen M. Arndt, Cynthia Kenyon, Julin Maloof, Jeffrey S. Simske, John W. Rooney and Hendrik C. Korswagen and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Genes & Development.

In The Last Decade

David M. Eisenmann

31 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David M. Eisenmann United States 22 1.3k 937 355 309 135 31 1.8k
Dave Hansen Canada 19 800 0.6× 730 0.8× 246 0.7× 141 0.5× 109 0.8× 30 1.1k
Yanxia Bei United States 10 1.5k 1.2× 881 0.9× 273 0.8× 121 0.4× 95 0.7× 14 1.9k
Cathy Savage‐Dunn United States 17 519 0.4× 711 0.8× 134 0.4× 181 0.6× 55 0.4× 36 1.1k
Sudhir Nayak United States 12 658 0.5× 615 0.7× 182 0.5× 123 0.4× 155 1.1× 19 1.0k
Diane G. Morton United States 15 1.4k 1.1× 768 0.8× 166 0.5× 90 0.3× 185 1.4× 17 1.9k
Anna P. Newman United States 18 1.0k 0.8× 561 0.6× 232 0.7× 189 0.6× 82 0.6× 30 1.6k
Gillian M. Stanfield United States 10 722 0.6× 541 0.6× 86 0.2× 99 0.3× 145 1.1× 17 1.2k
Vida Praitis United States 7 649 0.5× 757 0.8× 97 0.3× 122 0.4× 89 0.7× 7 970
Jeb Gaudet Canada 12 666 0.5× 556 0.6× 68 0.2× 139 0.4× 135 1.0× 17 955
Lynn Boyd United States 12 741 0.6× 521 0.6× 163 0.5× 80 0.3× 59 0.4× 15 1.2k

Countries citing papers authored by David M. Eisenmann

Since Specialization
Citations

This map shows the geographic impact of David M. Eisenmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David M. Eisenmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David M. Eisenmann more than expected).

Fields of papers citing papers by David M. Eisenmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David M. Eisenmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David M. Eisenmann. The network helps show where David M. Eisenmann may publish in the future.

Co-authorship network of co-authors of David M. Eisenmann

This figure shows the co-authorship network connecting the top 25 collaborators of David M. Eisenmann. A scholar is included among the top collaborators of David M. Eisenmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David M. Eisenmann. David M. Eisenmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Thompson, Kenneth W., et al.. (2016). The Paired-box protein PAX-3 regulates the choice between lateral and ventral epidermal cell fates in C. elegans. Developmental Biology. 412(2). 191–207. 7 indexed citations
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Jackson, Belinda M. & David M. Eisenmann. (2012).  -Catenin-Dependent Wnt Signaling in C. elegans: Teaching an Old Dog a New Trick. Cold Spring Harbor Perspectives in Biology. 4(8). a007948–a007948. 29 indexed citations
6.
Gleason, Julie E. & David M. Eisenmann. (2010). Wnt signaling controls the stem cell-like asymmetric division of the epithelial seam cells during C. elegans larval development. Developmental Biology. 348(1). 58–66. 45 indexed citations
7.
Weidhaas, Joanne B., David M. Eisenmann, Justin M. Holub, & Sunitha Nallur. (2006). A Conserved RAS/Mitogen-Activated Protein Kinase Pathway Regulates DNA Damage–Induced Cell Death Postirradiation in Radelegans. Cancer Research. 66(21). 10434–10438. 23 indexed citations
8.
Gleason, Julie E., et al.. (2006). Transcriptional upregulation of the C. elegans Hox gene lin-39 during vulval cell fate specification. Mechanisms of Development. 123(2). 135–150. 39 indexed citations
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Gleason, Julie E., et al.. (2006). Multiple redundant Wnt signaling components function in two processes during C. elegans vulval development. Developmental Biology. 298(2). 442–457. 86 indexed citations
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Eisenmann, David M.. (2005). Wnt signaling. WormBook. 1–17. 141 indexed citations
12.
Jackson, Belinda M., et al.. (2004). Identification of evolutionarily conserved promoter elements and amino acids required for function of the C. elegans β-catenin homolog BAR-1. Developmental Biology. 272(2). 536–557. 21 indexed citations
13.
Gleason, Julie E., Hendrik C. Korswagen, & David M. Eisenmann. (2002). Activation of Wnt signaling bypasses the requirement for RTK/Ras signaling during C. elegans vulval induction. Genes & Development. 16(10). 1281–1290. 95 indexed citations
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Eisenmann, David M., et al.. (1997). Identification of RTF1 , a Novel Gene Important for TATA Site Selection by TATA Box-Binding Protein in Saccharomyces cerevisiae. Molecular and Cellular Biology. 17(8). 4490–4500. 63 indexed citations
16.
Eisenmann, David M. & Stuart K. Kim. (1997). Mechanism of Activation of the Caenorhabditis elegans ras Homologue let-60 by a Novel, Temperature-Sensitive, Gain-of-Function Mutation. Genetics. 146(2). 553–565. 54 indexed citations
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Eisenmann, David M. & Stuart K. Kim. (1994). Signal transduction and cell fate specification during Caenorhabditis elegans vulval development. Current Opinion in Genetics & Development. 4(4). 508–516. 46 indexed citations
18.
Eisenmann, David M., C Chapon, Samuel Roberts, Catherine Dollard, & Fred Winston. (1994). The Saccharomyces cerevisiae SPT8 gene encodes a very acidic protein that is functionally related to SPT3 and TATA-binding protein.. Genetics. 137(3). 647–657. 78 indexed citations
19.
Arndt, Karen M., et al.. (1992). Biochemical and Genetic Characterization of a Yeast TFIID Mutant That Alters Transcription In Vivo and DNA Binding In Vitro. Molecular and Cellular Biology. 12(5). 2372–2382. 9 indexed citations
20.
Eisenmann, David M., Catherine Dollard, & Fred Winston. (1989). SPT15, the gene encoding the yeast TATA binding factor TFIID, is required for normal transcription initiation in vivo. Cell. 58(6). 1183–1191. 223 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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