Christoph Block

1.5k total citations
30 papers, 1.2k citations indexed

About

Christoph Block is a scholar working on Molecular Biology, Cell Biology and Cancer Research. According to data from OpenAlex, Christoph Block has authored 30 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 6 papers in Cell Biology and 5 papers in Cancer Research. Recurrent topics in Christoph Block's work include Protein Kinase Regulation and GTPase Signaling (13 papers), Melanoma and MAPK Pathways (7 papers) and Cell death mechanisms and regulation (6 papers). Christoph Block is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (13 papers), Melanoma and MAPK Pathways (7 papers) and Cell death mechanisms and regulation (6 papers). Christoph Block collaborates with scholars based in Germany, United States and China. Christoph Block's co-authors include Christian Herrmann, Alfred Wittinghofer, Ralf Janknecht, Nicolas Nassar, Detmar Beyersmann, Anant N. Malviya, Gudrun Horn, Christoph K. Weber, Thomas Linnemann and Andrew J. Dent and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Journal of Molecular Biology.

In The Last Decade

Christoph Block

30 papers receiving 1.2k citations

Peers

Christoph Block

ONCOL

Gongqin Sun United States

Julie E. Scheffler United States

Surinder K. Chander United Kingdom

Eva Estébanez‐Perpiñá Spain

Keizo Koya United States

Mokdad Mezna United Kingdom

Přemysl Poňka Czechia

Rebecca Del Conte Italy

C. Matthew Bradbury United States

Letizia Barbieri Italy

Gongqin Sun United States

Christoph Block

1.1k ×1.2

126 ×0.7

273 ×1.5

ONCOL

72 ×0.6

94 ×0.9

Citations per year, relative to Christoph Block Christoph Block (= 1×) peers Gongqin Sun

Countries citing papers authored by Christoph Block

Since Specialization

Citations

This map shows the geographic impact of Christoph Block's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christoph Block with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christoph Block more than expected).

Fields of papers citing papers by Christoph Block

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christoph Block. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christoph Block. The network helps show where Christoph Block may publish in the future.

Co-authorship network of co-authors of Christoph Block

This figure shows the co-authorship network connecting the top 25 collaborators of Christoph Block. A scholar is included among the top collaborators of Christoph Block based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christoph Block. Christoph Block is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wu, Jason, Fanyan Meng, Lun Dong, et al.. (2022). DDR2 Coordinates EMT and Metabolic Reprogramming as a Shared Effector of FOXQ1 and SNAI1. Cancer Research Communications. 2(11). 1388–1403. 7 indexed citations

Wu, Ling, Christoph Block, Mu Zhang, et al.. (2022). FOXQ1 recruits the MLL complex to activate transcription of EMT and promote breast cancer metastasis. Nature Communications. 13(1). 6548–6548. 24 indexed citations

Block, Christoph, et al.. (2019). A stroma-corrected ZEB1 transcriptional signature is inversely associated with antitumor immune activity in breast cancer. Scientific Reports. 9(1). 17807–17807. 18 indexed citations

Wu, Ling, Fanyan Meng, Lun Dong, et al.. (2019). Disulfiram and BKM120 in Combination with Chemotherapy Impede Tumor Progression and Delay Tumor Recurrence in Tumor Initiating Cell-Rich TNBC. Scientific Reports. 9(1). 236–236. 38 indexed citations

Dong, Lun, Fanyan Meng, Ling Wu, et al.. (2017). Cooperative oncogenic effect and cell signaling crosstalk of co-occurring HER2 and mutant PIK3CA in mammary epithelial cells. International Journal of Oncology. 51(4). 1320–1330. 4 indexed citations

Schmitz, Hans‐Peter, Johannes Jöckel, Christoph Block, & Jürgen J. Heinisch. (2001). Domain shuffling as a tool for investigation of protein function: substitution of the cysteine-rich region of Raf kinase and PKC η for that of yeast Pkc1p. Journal of Molecular Biology. 311(1). 1–7. 25 indexed citations

Weber, Christoph K., et al.. (2000). Mitogenic signaling of Ras is regulated by differential interaction with Raf isozymes. Oncogene. 19(2). 169–176. 55 indexed citations

Ahmadian, Mohammad Reza, et al.. (2000). Oncogenic insertional mutations in the P-loop of Ras are overactive in MAP kinase signaling. Oncogene. 19(47). 5367–5376. 10 indexed citations

Linnemann, Thomas, Matthias Geyer, Christoph Block, et al.. (1999). Thermodynamic and Kinetic Characterization of the Interaction between the Ras Binding Domain of AF6 and Members of the Ras Subfamily. Journal of Biological Chemistry. 274(19). 13556–13562. 121 indexed citations

10.

Herrmann, Christian, Christoph Block, Christoph Geisen, et al.. (1998). Sulindac sulfide inhibits Ras signaling. Oncogene. 17(14). 1769–1776. 109 indexed citations

11.

Daub, M., Johannes Jöckel, Thomas Quack, et al.. (1998). The RafC1 Cysteine-Rich Domain Contains Multiple Distinct Regulatory Epitopes Which Control Ras-Dependent Raf Activation. Molecular and Cellular Biology. 18(11). 6698–6710. 52 indexed citations

12.

Bähler, Martin, et al.. (1997). Ras‐binding domains: predicting function versus folding. FEBS Letters. 414(3). 599–602. 26 indexed citations

13.

Becker, Jörg D., et al.. (1997). Discrimination of Amino Acids Mediating Ras Binding from Noninteracting Residues Affecting Raf Activation by Double Mutant Analysis. Journal of Biological Chemistry. 272(47). 29927–29933. 31 indexed citations

14.

Nassar, Nicolas, Gudrun Horn, Christian Herrmann, et al.. (1996). Ras/Rap effector specificity determined by charge reversal. Nature Structural & Molecular Biology. 3(8). 723–729. 179 indexed citations

15.

Block, Christoph, Ralf Janknecht, Christian Herrmann, Nicolas Nassar, & Alfred Wittinghofer. (1996). Quantitative structure-activity analysis correlating Ras/Raf interaction in vitro to Raf activation in vivo. Nature Structural Biology. 3(3). 244–251. 124 indexed citations

16.

Block, Christoph & Alfred Wittinghofer. (1995). Switching to Rac and Rho. Structure. 3(12). 1281–1284. 6 indexed citations

17.

Beyersmann, Detmar, Christoph Block, & Anant N. Malviya. (1994). Effects of cadmium on nuclear protein kinase C.. Environmental Health Perspectives. 102(suppl 3). 177–180. 36 indexed citations

18.

Dent, Andrew J., Detmar Beyersmann, Christoph Block, & S. Samar Hasnain. (1990). Two different zinc sites in bovine 5-aminolevulinate dehydratase distinguished by extended x-ray absorption fine structure. Biochemistry. 29(34). 7822–7828. 72 indexed citations

19.

Block, Christoph, et al.. (1990). Probing of Active Site Residues of the Zinc Enzyme 5-Aminolevulinate Dehydratase by Spin and Fluorescence Labels. Biological Chemistry Hoppe-Seyler. 371(2). 1145–1152. 5 indexed citations

20.

Mannens, Geert, et al.. (1987). Immobilization of acetate kinase and phosphotransacetylase on derivatized glass beads. Enzyme and Microbial Technology. 9(5). 285–290. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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