Christine Petzel

448 total citations
10 papers, 375 citations indexed

About

Christine Petzel is a scholar working on Molecular Biology, Immunology and Physiology. According to data from OpenAlex, Christine Petzel has authored 10 papers receiving a total of 375 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Immunology and 2 papers in Physiology. Recurrent topics in Christine Petzel's work include Heme Oxygenase-1 and Carbon Monoxide (4 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (4 papers) and Immune Response and Inflammation (3 papers). Christine Petzel is often cited by papers focused on Heme Oxygenase-1 and Carbon Monoxide (4 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (4 papers) and Immune Response and Inflammation (3 papers). Christine Petzel collaborates with scholars based in Germany and Italy. Christine Petzel's co-authors include Helmut Schweikl, Karl‐Anton Hiller, Stephanie Krifka, Carola Bolay, Wolfgang Buchalla, Gianrico Spagnuolo, Gottfried Schmalz, Marialucia Gallorini, Amelia Cataldi and Franz‐Xaver Reichl and has published in prestigious journals such as Biomaterials and Dental Materials.

In The Last Decade

Christine Petzel

10 papers receiving 372 citations

Peers

Countries citing papers authored by Christine Petzel

Since Specialization

Citations

This map shows the geographic impact of Christine Petzel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christine Petzel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christine Petzel more than expected).

Fields of papers citing papers by Christine Petzel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christine Petzel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christine Petzel. The network helps show where Christine Petzel may publish in the future.

Co-authorship network of co-authors of Christine Petzel

This figure shows the co-authorship network connecting the top 25 collaborators of Christine Petzel. A scholar is included among the top collaborators of Christine Petzel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christine Petzel. Christine Petzel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	HEMA-induced oxidative stress inhibits NF-κB nuclear translocation and TNF release from LTA- and LPS-stimulated immunocompetent cells 2020 ·Dental Materials ·Helmut Schweikl, (unknown), Marialucia Gallorini, Christine Petzel, Carola Bolay, (unknown), Karl‐Anton Hiller, Wolfgang Buchalla	15
2	Functions of transcription factors NF-κB and Nrf2 in the inhibition of LPS-stimulated cytokine release by the resin monomer HEMA 2018 ·Dental Materials ·Helmut Schweikl, Marialucia Gallorini, (unknown), (unknown), Christine Petzel, Carola Bolay, Karl‐Anton Hiller, Amelia Cataldi, Wolfgang Buchalla	19
3	Flavin-containing enzymes as a source of reactive oxygen species in HEMA-induced apoptosis 2017 ·Dental Materials ·Helmut Schweikl, Marialucia Gallorini, (unknown), Christine Petzel, Carola Bolay, Karl‐Anton Hiller, Amelia Cataldi, Stephanie Krifka, Wolfgang Buchalla	9
4	Critical role of superoxide anions and hydroxyl radicals in HEMA-induced apoptosis 2016 ·Dental Materials ·Helmut Schweikl, (unknown), Christine Petzel, Carola Bolay, Karl‐Anton Hiller, Wolfgang Buchalla	13
5	Interaction between LPS and a dental resin monomer on cell viability in mouse macrophages 2016 ·Dental Materials ·Helmut Schweikl, Matthias Widbiller, Stephanie Krifka, (unknown), Christine Petzel, Carola Bolay, Karl‐Anton Hiller, Wolfgang Buchalla	12
6	Activation of the Nrf2-regulated antioxidant cell response inhibits HEMA-induced oxidative stress and supports cell viability 2015 ·Biomaterials ·Marialucia Gallorini, Christine Petzel, Carola Bolay, Karl‐Anton Hiller, Amelia Cataldi, Wolfgang Buchalla, Stephanie Krifka, Helmut Schweikl	92
7	2-Hydroxyethyl methacrylate-induced apoptosis through the ATM- and p53-dependent intrinsic mitochondrial pathway 2014 ·Biomaterials ·Helmut Schweikl, Christine Petzel, Carola Bolay, Karl‐Anton Hiller, Wolfgang Buchalla, Stephanie Krifka	54
8	Function of MAPK and downstream transcription factors in monomer-induced apoptosis 2011 ·Biomaterials ·Stephanie Krifka, Karl‐Anton Hiller, Carola Bolay, Christine Petzel, Gianrico Spagnuolo, Franz‐Xaver Reichl, Gottfried Schmalz, Helmut Schweikl	46
9	Resin monomer-induced differential activation of MAP kinases and apoptosis in mouse macrophages and human pulp cells 2010 ·Biomaterials ·Stephanie Krifka, Christine Petzel, Karl‐Anton Hiller, (unknown), (unknown), Gianrico Spagnuolo, Franz‐Xaver Reichl, Gottfried Schmalz, Helmut Schweikl	64
10	Activation of stress-regulated transcription factors by triethylene glycol dimethacrylate monomer 2010 ·Biomaterials ·Stephanie Krifka, Christine Petzel, Carola Bolay, Karl‐Anton Hiller, Gianrico Spagnuolo, Gottfried Schmalz, Helmut Schweikl	51

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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