Christer Einvik

1.6k total citations
29 papers, 1.3k citations indexed

About

Christer Einvik is a scholar working on Molecular Biology, Neurology and Cancer Research. According to data from OpenAlex, Christer Einvik has authored 29 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 12 papers in Neurology and 6 papers in Cancer Research. Recurrent topics in Christer Einvik's work include Neuroblastoma Research and Treatments (12 papers), RNA and protein synthesis mechanisms (10 papers) and Signaling Pathways in Disease (8 papers). Christer Einvik is often cited by papers focused on Neuroblastoma Research and Treatments (12 papers), RNA and protein synthesis mechanisms (10 papers) and Signaling Pathways in Disease (8 papers). Christer Einvik collaborates with scholars based in Norway, Denmark and Sweden. Christer Einvik's co-authors include Steinar Johansen, Jochen Buechner, Trond Flægstad, Bjørn Haug, Per Kogner, Cecilie Løkke, Henrik Nielsen, Baldur Sveinbjørnsson, John Inge Johnsen and Wayne A. Decatur and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Nature Communications.

In The Last Decade

Christer Einvik

29 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Christer Einvik Norway 21 1.1k 496 328 131 93 29 1.3k
Stephen M. Lewis Canada 21 1.5k 1.4× 451 0.9× 73 0.2× 109 0.8× 58 0.6× 44 1.7k
Margareta Wilhelm Sweden 19 1.1k 1.0× 340 0.7× 149 0.5× 703 5.4× 106 1.1× 30 1.4k
Gregory A. Horwitz United States 11 1.0k 1.0× 363 0.7× 142 0.4× 212 1.6× 277 3.0× 11 1.9k
Elizabeth T. Bartom United States 25 1.4k 1.3× 321 0.6× 66 0.2× 218 1.7× 165 1.8× 70 2.1k
Maximilian Kauer Austria 20 594 0.5× 251 0.5× 79 0.2× 237 1.8× 89 1.0× 35 1.1k
Beatrice Cardinali Italy 20 1.0k 0.9× 296 0.6× 58 0.2× 100 0.8× 89 1.0× 37 1.2k
Ryan D. Murphey United States 8 1.0k 1.0× 207 0.4× 41 0.1× 257 2.0× 127 1.4× 8 1.5k
Roland Meier Switzerland 15 506 0.5× 182 0.4× 166 0.5× 293 2.2× 62 0.7× 26 871
Takeshi Chujo Japan 15 1.8k 1.7× 658 1.3× 54 0.2× 51 0.4× 111 1.2× 27 2.0k
David P. Astling United States 15 591 0.5× 97 0.2× 49 0.1× 224 1.7× 154 1.7× 24 900

Countries citing papers authored by Christer Einvik

Since Specialization
Citations

This map shows the geographic impact of Christer Einvik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christer Einvik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christer Einvik more than expected).

Fields of papers citing papers by Christer Einvik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christer Einvik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christer Einvik. The network helps show where Christer Einvik may publish in the future.

Co-authorship network of co-authors of Christer Einvik

This figure shows the co-authorship network connecting the top 25 collaborators of Christer Einvik. A scholar is included among the top collaborators of Christer Einvik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christer Einvik. Christer Einvik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Løkke, Cecilie, et al.. (2023). Hsa-miR-323a-3p functions as a tumor suppressor and targets STAT3 in neuroblastoma cells. Frontiers in Pediatrics. 11. 1098999–1098999. 5 indexed citations
2.
Gallo-Oller, Gabriel, Cecilie Løkke, Per Kogner, et al.. (2018). Inhibitors of ribosome biogenesis repress the growth of MYCN-amplified neuroblastoma. Oncogene. 38(15). 2800–2813. 44 indexed citations
3.
Einvik, Christer, et al.. (2018). Hsa‑miR‑376c‑3p targets Cyclin D1 and induces G1‑cell cycle arrest in neuroblastoma cells. Oncology Letters. 16(5). 6786–6794. 20 indexed citations
4.
Wickström, Malin, Cecilia Dyberg, Jelena Milosevic, et al.. (2015). Wnt/β-catenin pathway regulates MGMT gene expression in cancer and inhibition of Wnt signalling prevents chemoresistance. Nature Communications. 6(1). 8904–8904. 193 indexed citations
5.
Buechner, Jochen & Christer Einvik. (2012). N-myc and Noncoding RNAs in Neuroblastoma. Molecular Cancer Research. 10(10). 1243–1253. 57 indexed citations
6.
Haug, Bjørn, Jochen Buechner, Dirk Geerts, et al.. (2011). MYCN-regulated miRNA-92 inhibits secretion of the tumor suppressor DICKKOPF-3 (DKK3) in neuroblastoma. Carcinogenesis. 32(7). 1005–1012. 57 indexed citations
8.
Buechner, Jochen, et al.. (2011). Tumour-suppressor microRNAs let-7 and mir-101 target the proto-oncogene MYCN and inhibit cell proliferation in MYCN-amplified neuroblastoma. British Journal of Cancer. 105(2). 296–303. 166 indexed citations
10.
Buechner, Jochen, et al.. (2010). Inhibition of Gene Function in Mammalian Cells Using Short-Hairpin RNA (shRNA). Methods in molecular biology. 703. 189–204. 4 indexed citations
11.
Nielsen, Henrik, et al.. (2009). A conformational switch in the DiGIR1 ribozyme involved in release and folding of the downstream I-DirI mRNA. RNA. 15(5). 958–967. 7 indexed citations
12.
Beckert, Bertrand, Henrik Nielsen, Christer Einvik, et al.. (2008). Molecular modelling of the GIR1 branching ribozyme gives new insight into evolution of structurally related ribozymes. The EMBO Journal. 27(4). 667–678. 16 indexed citations
13.
Henriksen, James R., et al.. (2007). Comparison of RNAi efficiency mediated by tetracycline-responsive H1 and U6 promoter variants in mammalian cell lines. Nucleic Acids Research. 35(9). e67–e67. 30 indexed citations
14.
Johansen, Steinar, et al.. (2006). RNA reprogramming of α‐mannosidase mRNA sequences in vitro by myxomycete group IC1 and IE ribozymes. FEBS Journal. 273(12). 2789–2800. 10 indexed citations
15.
Einvik, Christer, et al.. (2004). Twelve Group I Introns in the Same Pre-rRNA Transcript of the Myxomycete Fuligo septica: RNA Processing and Evolution. Molecular Biology and Evolution. 21(7). 1283–1293. 31 indexed citations
16.
Einvik, Christer, et al.. (2004). Optimization and Application of the Group I Ribozyme <I>Trans</I>-Splicing Reaction. Humana Press eBooks. 252. 359–372. 13 indexed citations
17.
Nielsen, Henrik, et al.. (2003). The ability to form full-length intron RNA circles is a general property of nuclear group I introns. RNA. 9(12). 1464–1475. 54 indexed citations
18.
Johansen, Steinar, Christer Einvik, & Henrik Nielsen. (2002). DiGIR1 and NaGIR1: naturally occurring group I-like ribozymes with unique core organization and evolved biological role. Biochimie. 84(9). 905–912. 20 indexed citations
19.
Einvik, Christer, Henrik Nielsen, Éric Westhof, François Michel, & Steinar Johansen. (1998). Group I-like ribozymes with a novel core organization perform obligate sequential hydrolytic cleavages at two processing sites. RNA. 4(5). 530–541. 34 indexed citations
20.
Einvik, Christer, et al.. (1998). Group I twintrons: Genetic elements in myxomycete and schizopyrenid amoeboflagellate ribosomal DNAs. Journal of Biotechnology. 64(1). 63–74. 39 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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