Cheikh I. Seye
- Physiology top 0.1%
- Molecular Biology top 10%
- Physiology top 5%
- Cellular and Molecular Neuroscience top 5%
- Neurology top 5%
- Co-authors
- Gary A. WeismanLaurie ErbZhongji LiaoFernando A. GonzálezJean M. CamdenQiongman KongGrace Y. SunOlga J. Baker
- Topics
- Adenosine and Purinergic Signaling (35 papers)Cardiovascular Issues in Pregnancy (9 papers)Receptor Mechanisms and Signaling (8 papers)
- Partner nations
- United StatesPuerto RicoFrance
In The Last Decade
Cheikh I. Seye
45 papers receiving 2.1k citations
Peers
Comparison fields: 5 of 96
- Physiology 1.4k
- Molecular Biology 730
- Physiology 430
- Cellular and Molecular Neuroscience 316
- Neurology 295
Countries citing papers authored by Cheikh I. Seye
This map shows the geographic impact of Cheikh I. Seye's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cheikh I. Seye with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cheikh I. Seye more than expected).
Fields of papers citing papers by Cheikh I. Seye
This network shows the impact of papers produced by Cheikh I. Seye. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cheikh I. Seye. The network helps show where Cheikh I. Seye may publish in the future.
Co-authorship network of co-authors of Cheikh I. Seye
This figure shows the co-authorship network connecting the top 25 collaborators of Cheikh I. Seye. A scholar is included among the top collaborators of Cheikh I. Seye based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cheikh I. Seye. Cheikh I. Seye is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 1 | |
| 3 | Endothelial Cell-Specific Deletion of P2Y2 Receptor Promotes Plaque Stability in Atherosclerosis-Susceptible ApoE-Null Mice | 3 |
| 4 | The P2Y2 nucleotide receptor is an inhibitor of vascular calcification | 1 |
| 5 | 21 | |
| 6 | 12 | |
| 7 | 14 | |
| 8 | 30 | |
| 9 | 48 | |
| 10 | 16 | |
| 11 | 9 | |
| 12 | 48 | |
| 13 | 93 | |
| 14 | 17 | |
| 15 | 13 | |
| 16 | 78 | |
| 17 | 114 | |
| 18 | 49 | |
| 19 | 99 | |
| 20 | 4 |
About Cheikh I. Seye
Cheikh I. Seye is a scholar working on Physiology, Neurology and Cardiology and Cardiovascular Medicine, having authored 46 papers that have together received 2.2k indexed citations. Recurring topics across this work include Adenosine and Purinergic Signaling (35 papers), Cardiovascular Issues in Pregnancy (9 papers) and Receptor Mechanisms and Signaling (8 papers). The work is most often cited by research in Physiology (1.4k citations), Neurology (295 citations) and Endocrine and Autonomic Systems (192 citations). Cheikh I. Seye has collaborated with scholars based in United States, Puerto Rico and France. Frequent co-authors include Gary A. Weisman, Laurie Erb, Zhongji Liao, Fernando A. González, Jean M. Camden, Qiongman Kong, Grace Y. Sun, Olga J. Baker, Nataliya Chorna and Fernando A. González. Their work appears in journals such as Journal of Biological Chemistry, Circulation and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.